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In fetal human kidneys, high level of FPC expression is present in ureteric bud and its expression continues throughout the process of renal tubuobranching.

在人类，FPC在胎肾即开始表达于输尿管芽，并持续表达于输尿管芽分支演变为集合管的整个过程。

To determine the effects of ovarian steroid hormones and some cytokines on the expression of RANTES in cultured cells METHODS AND RESULTS The study included the following two parts 1 The Protein Production of RANTES in Peritoneal Fluids and the Expression of RANTES mRNA in Eutopic and Ectopic Endometrium of Patients with Endometriosis Twenty-eight patients with endometriosis were selected as study group and twenty patients without endometriosis and maglinant disease were selected as control group . In our study, 10 samples of peritoneal fluid in Em and 10 samples of in En were taken, 13 specimens of ovarian endometrioma , 3 red peritoneal endometriotic lesions , 2 uterosacral ligament nodules , 17 matched eutopic endometrium and 12 specimens of endometrium in En were biopsied.

研究目的 1 从蛋白水平检测RANTES在子宫内膜异位症患者腹水中的表达，从核酸水平比较RANTES在内异症患者的异位病灶和在位内膜与非内异症患者子宫内膜之间表达的差异，探讨RANTES在内异症患者盆腔炎性反应中的作用；2 体外培养在位和异位的子宫内膜细胞，研究细胞因子TNFα、INFγ、雌孕激素以及PPARγ激动剂15-d-PGJ〓对RANTES表达的影响，探讨这些因素在内异症发病机理及内异症患者盆腔炎性反应中的作用。

Among them group A dendritic cell surface processes were less and cytoplasm vacuolated obviously. 2. Compared the expression of cell surface molecules of 5d, 7d ,10d,14d, we found the CD1a, CD83, CD86, HLA-DR and CD80 expression was significantly increased P.

口腔癌患者外周血单个核细胞诱导培养第5d、第7d、第10d、第14d时，细胞表面DC分子的表达情况：第7d时的DC与第5d相比，CD1a、CD83、CD86、CD80和HLA-DR表达率明显上升P 0。

ResultsThere was no expression of Caspase-3 in group A. The expression of Caspase-3 in group C was decreased compared with that in group B（P＜0.01. In groups B and C, expansion and vacuolization of mitochondria, swollen neurons and apoptotic bodies appeared, which suggested that WBH could induce neuron injury, and the neuron injury in group C was more severe than that in group B.

结果A组大鼠海马区神经元无Caspase-3表达，B、C两组大鼠海马区神经元有Caspase-3表达，B组较C组Caspase-3表达平均灰度值减小。B、C两组大鼠海马神经元超微结构发生改变、细胞器水肿，部分线粒体空化，内质网稀疏，突触前膜、后膜结构不完整，可见自噬体结构；B组大鼠的神经元超微结构损害较C组轻。

Due to begin to present differentiation,IEC-6 cells grew slowly.2.This study design and compose three pairs siRNAs to RPS20 of rats,and they interference the expression of RPS20 on the aspect of mRNA and protein successfully,and prove their validity.3.After interference,IEC-6 cells cytoplasm present vacuolization,and its migration,and proliferation were suppressed,but there was no apoptosis to happen.These results indicate that interfering expression of RPS20 may affect the function of digestion and absorption and gastrointestinal mucosa healing,which is in accordance with condition of low level on digestive and absorptive function and gastrointestinal mucosa healing.

本课题设计并合成了3对针对大鼠RPS20的siRNAs，而且都能够有效地抑制IEC-6细胞RPS20mRNA与蛋白的表达，表明了序列的有效性；3 IEC-6细胞胞浆空泡化，并且迁移、增殖能力受到抑制，但并没有导致细胞早期凋亡的发生，该结果提示干扰RPS20可能影响IEC-6细胞的消化吸收功能和胃肠粘膜损伤修复功能，与前期收集的脾气虚证患者消化吸收功能下降、黏膜损伤修复能力下降等表现相符合。

Nimodipine in addition to its cerebral vasodilating effect can protect transient focal cerebral ischernic injury through inhibiting iNOS expression in the infarcted area, and 〓 may be involved in the iNOS expression in the ischemic brain.

尼莫地平除了扩张脑血管外，还可通过抑制缺血脑组织的iNOS表达限制NO过量产生而发挥抗脑缺血作用。此外，〓可能介入缺血脑组织的iNOS表达。

These results suggest that nimodipine in addition to its cerebral vasodilating effect may protect brain from ischemic neuronal damage through modifying iNOS expression in the infarct area, and imply that 〓 may be involved in the iNOS expression in ischemic brain tissue which contributes to the recruitment of penumbra into infarction after transient focal cerebral ischemia.

上述结果表明，除了扩张脑血管外，尼莫地平可能还通过抑制iNOS在缺血性脑组织的表达阻止NO过量产生而起到脑缺血损伤的保护作用。鉴于尼莫地平是典型的钙拮抗剂，本结果还提示〓可能涉及缺血脑组织的iNOS表达，从而介入半暗区向梗死区转变的病理过程。

The expression of Bcl-2mRNA were significantly inhibited after Vasoligation (P＜0.05), Vasostomy made its expression increased to the level of before vasoligation

输精管阻断后Bcl-2mRNA的表达被显著抑制（P.05），复通后Bcl-2mRNA的表达恢复，与SOG组无显著差异。

Compared with SHRc , after 4 weeks, Pro-EGF mRNA expression level of SHRsv and SHRlv VSMC were significant lower.Conclusions: Valsartan decrease SBP of SHR, inhibit SHR VSMC proliferation, reverse vascular hypertrophy of thoracic aorta and mesenteric artery in SHR, improve the two arteries vasomotion function. Pro-EGF mRNA expression only in SHR VSMC, not in the VSMC of WKY. It indicated pro-EGF gene may play an important role in hypertension advance .

缬沙坦有效降压的同时，抑制SHR大鼠VSMC增殖，逆转其弹性血管和阻力血管的肥厚，明显改善其弹性血管和阻力血管的舒缩功能。pro-EGF mRNA只在SHR大鼠VSMC表达，而在WKY大鼠VSMC没有表达，提示pro-EGF基因可能在高血压的发生发展中起一定的作用，可能是高血压的一个病理机制。

Only weak expression of ICAM-1 was found on bronchial and alveolar epithelium, pulmonary arteriolar and venular endothelium in normal and sensitized rat lung. The expression of ICAM-1 in all tissues mentioned above were increased in asthmatic rats.

免疫组化结果显示，哮喘大鼠支气管上皮细胞、肺动静脉内皮细胞及肺泡上皮细胞ICAM-1表达均明显高于对照组。

Objective:To compare the response control and attention of Schizophrenic patients with that of the healthy controls by Integrated Visual and Auditorycontinuous performance test. To quantitate the impairment of cognitive function in patients, and to explore the relationship between cognitive function and the severity of the disorder.

目的：探讨精神分裂症病人在IVA持续操作测试中的反应控制能力、注意力等，并与健康人进行比较，从而量化精神分裂症病人的认知功能损害，并进一步探讨其认知功能与疾病严重程度的关系。

Main effective factors including subcooling degree, mechanical vibration, gas hydrate reformation, environment temperature, noncondensing gas and surfactant are analyzed.

指出过冷度、机械振动、重复生成水合物、环境温度、不凝性气体、添加剂是影响气体水合物生成的主要因素，还对R152a水合物的放冷进行了实验研究。