损伤

The next was intestinal tract injuries （ incidence rate was 26.32%）,which mainly showed subserous bleeding, rupture of sermuscular layer and subserosal hematoma.

结论水下冲击伤具有死亡率高，肺损伤发生率高、肠道损伤发生率较高、肝脾等实质脏器损伤轻微，胆囊、膀胱等含液脏器和体表很少发生损伤等特点。

Results Followed-up for 25~37 months (average 27.5 months), the fisrt signs of reinnerveation of suprascapular nerve appeared at 5~12 months,average 9.94±3.26 months,that of axillary nerve appeared at 9~15months, average12.60±3.08 months;The results in patients with suprascapular nerve as recipient(upper partial 57.08±21.16°, total 40.71±14.27°) were significantly better than that in patients with axillary nerve as recipient(upper partial 31.25±20.97°, total 18.75±7.50°). And the muscular function in upper, middle and lower portions of trapezius are affected due to nerve transfer.

结果 随访25~37个月(平均27.5个月)，来院随访18例；副神经移位修复肩胛上神经术后功能恢复征象最早出现在5~12个月，平均9.94±3.26个月，修复腋神经最早出现在9~15个月，平均12.60±3.08个月；修复肩胛上神经肩外展恢复结果(上臂丛神经损伤57.08±21.16°，全臂丛损伤40.71±14.27°)优于修复腋神经的恢复结果(上臂丛神经损伤31.25±20.97°，全臂丛损伤18.75±7.50°)，P.05；副神经移位后斜方肌各部功能丧失明显。

In the case of granite, observation results indicate that the extending routes of active cracks are mainly intercrystalline, transcrystalline, inter-transcrystalline, and intracrystalline. When the strain rate is low, the long intercrystalline cracks are prevailing, and with increasing of the strain rate, the short transcrystalline and intracrystalline cracks gradually increase. The statistic results show that the active crack density increases with increasing of the strain rate, and the mean length of active cracks diminishes with increasing of the strain rate, that is, the induced damage in comminuted product increases with increasing of the strain rate. For the first time this dissertation integrates macroscopic results of comminution with active crack growth under impact loading. It elucidates why the dynamic comminution strength of mineral beds increases with increasing of the strain rate through the active crack extending, and analyzes the influence of structure and constitution of minerals on their damage under impact stress. It is authenticated that both the size distribution and the damage population of comminuted product can be characterized by the fractal.

为分析料层颗粒在冲击下的细观损伤效果，采用德国生产的ASM68K半自动图象分析仪对粉碎生成物中的损伤情形进行了观测，以花岗岩为例，观测结果表明花岗岩颗粒在冲击应力下的活化裂纹扩展路径主要表现为沿晶、穿晶、沿穿晶和晶内等形式，在低应变速率下，活化裂纹以沿晶裂纹较多，随着应变速率的提高，其它裂纹形式大量繁衍，统计结果表明活化裂纹密度随着应变速率的增大而增大，活化裂纹的平均长度随着应变速率的增大而减小，综合表现为颗粒的细观损伤程度随着应变速率的增大而增大；首次将应力作用下的活化裂纹演化特征与粉碎的宏观效果相联系，并就料层动态粉碎强度随应变速率增大而提高的现象从裂纹演化特征的角度进行了解释；分析了矿岩构造结构特性对损伤产生和分布的影响。

Results Serum EDLS increased within 24 h after brain trauma of different degree in all patients with brain injuries of various intensities.The group of slight injury was the most typal,EDLS demonstrated no change or decrease in a few patients with the injury intensity growing;In most cases EDLS decreased after brain operation or accompaning the wounded recovery; Contents of EDLS at 24 h among different pathological types of brain injuries showed no difference, but in the successful recovery patients with severe or ultra-severe brain injuries, EDLS contents at 72 h posttrauma were lower than in the patients of death or disability. The above change characteristics of EDLS related to injury locations and the degree of brain injury.

结果 不同程度脑外伤后24小时内血清EDLS均升高，以轻伤组最为典型；随着损伤程度的加剧，少数病例EDLS不变甚至下降；术后或随伤情的恢复，EDLS多同步下降；不同病理类型的脑损伤24小时内EDLS无明显差异，在成功康复的重型、特重型脑损伤病人，伤后72小时EDLS含量低于死亡或伤残组；EDLS 的上述变化特点与损伤部位和程度不同有关。

NAC also blocked the up-regulation of TGF-β in in vivo model of VILI. Conclusion TGF-β1 and TGF-β2 were up-regulated in VILI. Oxidant injury mediated up-regulation of TGF-β in VILI. NAC, which attenuated oxidant injury and blocked TGF-β up-regulation in VILI, could be a future therapeutic strategy in VILI.

结论TGF-β1和TGF-β2在通气机所致肺损伤中的表达增加并在血清中上调；TGF-β上调的机制与氧自由基损伤有关但与肿瘤坏死因子无关；N-乙酰半胱氨酸可对抗氧自由基损伤，能阻断TGF-β的上调，可能成为呼吸机相关肺损伤的一种新治疗途径。

(1) Oxidative/antioxidative reactions, inflammatory reactions, apoptosis in lung are involved in the pathogenesis of HILI;(2) ABX, rHuEPO have protective effects on HILI respectively;(3) Hyperoxia can increase VILI in rats, but has little effects with low tidal volume mechanical ventilation.

结论(1)氧化抗氧化失衡，炎症反应，肺内细胞凋亡参与了高氧所致成年大鼠肺损伤的过程；(2)ABX通过抗炎、抗氧化、抑制细胞凋亡作用对成年大鼠高氧肺损伤具有保护作用；rHuEPO通过抗炎、抗氧化、抑制细胞凋亡及一定程度的抗纤维化作用对成年大鼠高氧肺损伤具有保护作用；(3)高浓度氧能明显加重大鼠通气机所致肺损伤，而高浓度氧对小潮气量通气影响较小。

Several investigators have determined that placing the electrode parallel rather than perpendicular to the target nerve substantially increases the size of the lesion, thereby reducing the likelihood the treatment will miss or only partially coagulate the target nerve.234,235 After a literature review and cadaveric study, Lau et al.234 concluded the ideal electrode position is across the lateral neck of the superior articular process rather than the groove formed at the angle of the superior articular and transverse processes, as was used in most studies.159,160Other investigators have found the maximal lesion size to be reached within 60 s of lesion time,235–237 independent of whether the system is temperature or voltage controlled.238 Studies conducted in human myocardium have determined that irrigation fluid has either no effect or a slightly beneficial effect on lesion size.239 Hence, the use of LA to prevent procedure-related pain or steroid to reduce the incidence of neuritis184 should theoretically have no adverse effects on the efficacy of radiofrequency denervation.

一些研究者认为，将点击平行的置于目标神经比将其垂直置于目标神经增加了消融的范围，因而减少损伤可能使目标神经不被或部分被消融。Lau等回顾了文献中对尸体的研究，总结出理想的电极放置位置为上关节突颈部外侧而非常采用的上关节突与横突形成的沟。其他学者研究发现，不论系统是温度控制或电压控制，其最大的损伤范围都在60秒内达到。在心肌灌注的研究中，灌注液多损伤范围造成轻微或不造成损伤。因此，使用LA预防过程相关性疼痛或类固醇防止神经炎发生时，需理论上确保对射频消融效果无副作用。

Though the primary stimulator of 1H is not clear, it is related to physical, cell and body fluid IH is a chronic process involving multiple faction injury and blood flowing abnormal are revamped as the main reason, the proliferation level depend on the length and width of the injure of vessel large amount research illustrate injury cause many growth factor infolded in IH, for example: template derived growth factor, fibroblast growth factor, transmission growth factor , endothelin (ET-1) et al, some early responsive gene such as c-fos、c-jun c-myc also take part in SMC proliferation after vessel injury.

虽然IH的确切起始刺激因子并未阐明，但已知与物理、细胞和体液因子有关。IH是一个多因素参与的慢性过程，损伤和血液动力学异常一真被认为是IH的主要因素。内膜增殖的程度有赖于血管损伤的长度和深度。近年来大量的实验研究结果表明，损伤等刺激引起的众多的生长因子样物质在IH中起关键作用。如血小板源性生长因子、碱性纤维母细胞生长因子、转化生长因子β、内皮素-1（ET-1）等诸如C-fos、C-jun、C-myc等早期应答基因也参与了血管损伤后SMC的增殖。

Every group rats were divided into two parts with the same rats. At last the rats were redivided ,then them were killed and their brains were excluded after one hour.4 hours or one week.Results PO2 and cerebral blood flow of E-selectin 2mg/kg group were higher than that of saline and lower than that of l0mg/kggroup(p.01).The volume of cerebral infarct and blood speed of E-selectin 2mg/kg group was smaller than that of saline and larger than that of l0mg/kg group(p.01).At the same time PCh.speeds.flows and the volume of infarct were related with the time (p.01).Conclusion E-selectin can effectively increase PC2 and rCBF after ischemia and reperfusion .reducing infarct volumes and speeds of blood at the same time . Artificial synthetic E-selectin can effectively reduce the reperfusion injury following cerebral ischemia.

结果 生理盐水组，2mg／kg组，10mg／kg组PO_2值依次提高(p＜0.01)，脑皮质血流量依次提高(p＜0.01)；脑血流速度依次减低；脑梗死体积依次减少(p＜0.01)；同时在测定的时间段里PO_2和脑组织血流量值与时间成正相关(p＜0.01)；脑梗死体积和脑组织血流速度值与时间成正相关(p＜0.01)。E一选择素在脑缺血再灌注损伤中表达的实验研究中文摘要结论E一选择素能增加脑缺血再灌注损伤的脑组织氧饱和度，脑血流量，减低脑血流速度；减少脑损伤体积；从而保护了脑缺血再灌注损伤的脑组织。

The parameters and methods for evaluating bird impact damage of fan/compressor blade are discussed.

结合发动机设计准则与设计规范的要求，提出了2个用于评定叶片损伤能力的叶片变形损伤参数和塑性应变损伤参数；经对模型叶片在不同撞击位置进行的鸟撞击损伤评定，证明方法可

He and Nina moved to California and lived at 2005 Ivar Street, Apt.

他和Nina搬到加州，并在2005年伊瓦尔街，公寓生活。

Droperidol （ potently inhibits transfected HERGchannels and this is the probable mechanism for QT prolongation.

氟哌利多有效地抑制了转染的 HERG 钾通道，可能是 QT 间期延长的机制。