形成血管

Wenxin capsule can promote endolethial cells releasing CO and synthesing 〓, enhance activity of HO, induce angiectasis through activating guanylic cyclase and adenglate cyclate of intracellar of vessel smooth muscle cell to enhance content of intracellar cGMP and AMP. 3. Wenxin capsule can inhibit releasing of platelet activiting factor 〓 which inhibit platelet aggregation and have antithrombosis function. 4. Wenxin capsule can promote activity of t-PA, reduce activity of PAI-1, strengthen fibrinolytic function, therefore inhibit fibrin disposition. 5. Wenxin capsule can inhibit synthesis of smooth muscle cellular DNA. Its function don't kill it directly but prevent transformation from period Go to peried S. 6. Wenxin capsule is an effective prescription of anti-coronary atherosclerosis atherosis. The results of image pattern analysis tastifies that area of lipid plaque in treating group reduce 54% and 58% in contrast with that in control group. The results of light, electro- microscopy show that structure of endolethial cell and smooth muscle cell in treating group is more integral than that in control group.

其作用机制之一是由于该药具有良好的调脂降脂作用，其不仅能明显降低血清LDL-c及升高HDL-c含量，且能有效阻止TC沉积于血管壁，从而保护内皮细胞结构完整；此外，温心胶囊尚具有明显的抗氧化作用，该药不仅能明显降低血浆LPO含量，且能提高SOD活性，同时其对血红素在细胞内代谢的调节作用也参与了抗氧化过程。2 温心胶囊能明显促进内皮细胞释放CO及合成〓，提高HO活性，通过激活血管平滑肌细胞内鸟苷酸环化酶及腺苷酸环化酶引起细胞内cGMP、AMP浓度增加而引起血管舒张。3 温心胶囊能明显抑制血小板激活因子〓的释放，从而抑制血小板聚集，抗血栓形成。4 温心胶囊能明显提高t-PA活性，降低PAI-1活性，增强纤溶功能，从而抑制纤维蛋白沉积。5 温心胶囊能明显抑制平滑肌细胞DNA合成，其抑制作用不是直接杀伤抑制，而是阻止细胞由Go期向S期转化。6 温心胶囊是抗冠状动脉粥样硬化的有效制剂，图象分析结果证实，该药治疗组脂斑面积比对照组缩小了54％和58％，光电镜结果显示内皮细胞及平滑肌细胞结构较为完整。

Retinal neovascularization is a complicated pathophysiological process as a result of imbalance between angiogenic and antiangiogenic factors.

视网膜新生血管形成是促血管生成因子和血管生成抑制因子之间平衡失调的一个复杂病理生理过程。

At 4 weeks following implantation, cells on the surface of coralline hydroxyapatite were found and connective tissues were seen in the material pores in the experimental group. Cells on the coralline hydroxyapatite were observed only in the control group. At 8 weeks, new bone formation was detected on the surface of coralline hydroxyapatite; bony tissue deposition and a few chondroid tissues were found in the pores or surrounding the pores in the experimental group. A few fibrous connective tissues were observed in the control group. At 12 weeks, abundant mature woven bone was detected on the surface of coralline hydroxyapatite; medullary cavity-like structure and vessels were found in some regions in the experimental group. No new bone or bony tissues were found in the control group.

植入材料后4周，实验组可见珊瑚羟基磷灰石表面有细胞生长，孔隙内有结缔组织长入；对照组仅见珊瑚羟基磷灰石表面有细胞生长。8周时珊瑚羟基磷灰石表面有新生骨形成，孔隙内和孔隙边缘可见骨样组织沉积和少量软骨样组织形成；对照组仅见少量纤维结缔组织长入。12周时珊瑚羟基磷灰石材料表面有较多成熟编织骨形成，部分区域可见髓腔样结构形成，并有血管长入；对照组仍未见新骨及骨样组织形成。

The expression of ILK in the preretinal membranes in PDR indicate that ILK involves in the pathological process of neovascularization in PDR. Intravitreal injection of Avastin can decrease the numbers of endothelial cells in the proliferative fibrovascular membranes, and it also can down-regulate the expression of VEGF and HIF-1αprotein, but it can not affect the expression of ILK protein.

结论1。人PDR增殖膜中的新生血管有ILK表达，提示ILK参与了视网膜新生血管的形成过程；玻璃体内注射Avastin治疗PDR，能够减少视网膜新生血管内皮细胞数量，下调HIF-1α、VEGF在增殖膜的表达，但不能下调ILK的表达。

The middle gluteal muscle was multi-vascularied, and there were a lot of vascular networks in it.

臀中肌由多条血管供血，诸血管肌支在肌肉内形成丰富的血管吻合网。

Topography map of the hip bone displays the depth of all parts of the hip bone and the projection of important blood vessel and nerve onto the hip bone imaginarily and directly.

将坐标系、等厚线和重要血管神经的投影图同时绘制在髋骨上，形成3张地形图，1张为髋骨内侧面地形图，1张为没有血管和神经结构的髋骨外侧面地形图，1张为有血管和神经结构的髋骨外侧面地形图。

VEGF and the classification that its accept put oneself in another's position and action mechanism VEGF say blood-vessel is connected again appear factor (Vascular Permeabilityfactor, VPF) or blood-vessel are opsonic , it is right blood-vessel forms those who have main effect to grow factor.

VEGF及其受体的分类和功能机制VEGF又称血管通透因子(vascular permeabilityfactor，VPF)或血管调理素，是对血管形成具有重要功能的生长因子。

Special staining methods, such as Masson and the Van Gieson staining were used to study the distribution of collogen fibers and elastic fibers. ResultsBy HE staining, the subepithelial connective tissues and vessels in the pterygium were more prominent than normal conjunctival tissues. An amorphous subepithelial superficial hyalinized zone and coarse eosinophilic granular materials were observed in the pterygia, but they were not found in normal conjunctival specimens. Coarse fibers were visible only in the deeper subepithelial connective tissues of pterygial samples. With Masson′s staining, the dense staining of collagen fibers was also more prominent in the pterygium than in the subepithelial connective tissues of normal conjunctiva. Abnormal collagen fibers were visible in the deeper sub-epithelial connective tissues of pterygial samples. With Van Gieson staining, abnormal collagen fibers were visible in the deeper subepithelial connective tissues. Dark coarse elastic fibers were found in the abnormal fibers only in the subepithelial deep connective tissues of pinguecula in the pterygia but not in the conjunctiva. With immunohistochemistry staining, MMP-3 was strong in the pterygial epithelium, moderate in fibroblast and absent from pterygial vascular walls. LN was strongly expressed in the blood vessel wall, moderately in the epithelial basement membrane and absent from the entire stroma.

结果HE染色：翼状胬肉组织上皮下基质中存在结缔组织的增生和血管形成；基质浅层存在一无定形物质透明区及粗糙的颗粒样嗜酸性物质，在翼状胬肉体部深层基质中存在粗糙的纤维组织；正常球结膜组织细胞排列整齐；基质为疏松结缔组织，胶原纤维平行排列，其间可见成纤维细胞，散在少量中性粒细胞、毛细血管；Masson染色：翼状胬肉浅层基质中存在致密的胶原纤维染色，深层基质中的胶原纤维存在变性样改变；VG染色：翼状胬肉组织深层基质中存在大量变性的胶原纤维，其间夹杂黑色的弹性纤维；免疫组化染色法：MMP-3在翼状胬肉上皮细胞中呈强表达，成纤维细胞中呈中等强度表达，血管内皮细胞中未见表达；LN在血管壁中呈强表达，在上皮细胞基底膜中呈中等强度表达，在整个基质中未见明显表达；col Ⅲ在整个翼状胬肉基质中呈强表达。

This plaque will cause an obstruction that interferes with the man''''s ability to get an rection.

由于血管壁上形成血栓，高血压导致通往前列腺的血管血流量减少，血栓会造成血管堵塞，影响男性的勃起能力。

Results 甀ntrahepatic vascular diseases, liver tissues diseases and the expressions of VEGF were different in different time of experiment induced by CC14. In earlier periods (2 to Swk) of rats? liver tissues induced by CC14, the inflammatory was significant. Fatting degeneration was seen in hepatocytes around central veins and part of small vesells.

早、中期（2～5wk），肝组织炎症反应显著，中央静脉及部分小血管周围肝细胞脂肪变，VEGF阳性肝细胞主要分布在脂肪变区域；晚期（8～12wk）则肝组织炎症反应轻微，但肝细胞病变显著，血管破坏、血管及成肌纤维细胞增生更趋明显，并见有局部纤维瘢痕形成，假小叶内肝组织肝窦扩张或毛细血管化，其VEGF示阳性。

He and Nina moved to California and lived at 2005 Ivar Street, Apt.

他和Nina搬到加州，并在2005年伊瓦尔街，公寓生活。

Droperidol （ potently inhibits transfected HERGchannels and this is the probable mechanism for QT prolongation.

氟哌利多有效地抑制了转染的 HERG 钾通道，可能是 QT 间期延长的机制。