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形成血管

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Endovascular restenosis is an abnormal repairing response of vessel against injury. Following stent implantation, vessel contractibility reconstitution, local inflammation and thrombogenesis, intima hyperplasia, mainly vascular smooth muscle hyperplasia, are found.

血管内再狭窄是血管对抗损伤的一种异常修复反应,表现为支架植入后血管的收缩性重构,局部的炎症与血栓形成,以及以血管平滑肌细胞增殖为主的内膜增生。

The embryonic vessels failed to invade into the labyrinthine layer of placenta, which impaired the embryonic-maternal vascular connection. These defects could not be rescued by wild-type tetraploid blastocysts, excluding the possibility that they were due to the extraembryonic tissues.

组织免疫组化分析结果显示:毛细血管腔异常扩张,排列紊乱,不能形成大血管及细致有序的血管网络;胚外中胚层来源的卵黄囊脏层扁平上皮细胞形态变圆,与内皮细胞连接受损;胚胎血管不能侵入胎盘迷路层,胚体与母体血管连接受损。

Research results indicate that varicosity can be treated by means of endovenous thrombus formation, blood vessel wall damnification and vessel enclosure induced by light absorption of haemoglobin irradiated by semiconductor laser.

研究结果表明:半导体激光进行腔内静脉照射,由于血红蛋白对光的吸收,可以引起静脉腔内血栓形成、血管壁损伤、血管闭合,实现对静脉曲张的治疗;激光照射导致静脉腔内血栓形成,与血栓性静脉炎的形成机制不同,并没有影响全身凝血功能。

Antibodies against col Ⅲ reacted strongly in the entire pterygial stroma. Conclusions Components of ECM are changed in the pterygium: the sub-epithelial connective tissues and vessels are more prominent in the pterygia than in normal conjunctiva tissues, and abnormal fibers are visible only in the deeper subepithelial connective tissues. This study documents the overexpression of LN and col Ⅲ. They may play roles in cellular adherency, migration and angiogenesis. Also this study documents the overexpression of MMP3 which may degrade ECM in the pathogenesis of the pterygium.

结论翼状胬肉组织中ECM的成分较正常球结膜发生改变,有大量的弹性纤维变性、胶原纤维增生及新生血管形成;col Ⅲ在翼状胬肉组织中表达增加,LN及胶原纤维可能对翼状胬肉中血管形成、细胞粘附和迁移及ECM重建起作用;MMP-3在翼状胬肉组织中表达增加,其对ECM成分的降解作用可能有助于翼状胬肉的形成和向角膜中央侵袭。

Cerebral thrombosis、cerebral embolism and encephalorrhagia Because the blood fat lead high, deposition on the cerebral wall, gradual The formation artery gruel kind harden spot piece, the spot piece increase to make blood vessel blood inside the wall bolt formation and cause blood vessel break Blood but formation of.

脑血栓、脑栓塞及脑出血是由于血脂过高,沉积在脑血管壁上,逐渐形成动脉粥样硬化斑块,斑块增多使血管壁内血栓形成,致使血管破裂出血而形成的。

Twelve hours latter,uterine vessels formation was revealed by tannin-Fe lavement solution.Results The uterine vessel formation of rats in the model group showed that varices,tetanic shape,the density of all levels vessel net increased obviously and its twist,serpentine shape decreased markedly,while that in the large,middle dosage Gongning groups displayed absolute contrarily and fit like a glove with the normal control group.The uterine vessel formation of rats in the small dosage Gongning group and indomethacin group also showed the same transformation as it in the model group.

结果 模型组大鼠子宫各级血管扩张增粗,走行僵直,螺旋状和蜿蜒状走行消失或明显减少,血管形成的各级血管网密度增加,分布不均;消炎痛组、宫宁小剂量组子宫血管仍存在较明显的血管腔扩大、走行僵直和血管网密度增加,宫宁大、中剂量组子宫血管构筑的异常改变消失或明显改善。

To pay attention to carry out effective cardiopulmonary resuscitation, hemostasis by compression and deep vein intubation rapidly in the initial stage of emergency. The carotid shunt tube plays an important role in severe carotid injury and re-establishment process. We use external jugular vein to transplant and re-establish,which are rung with artificial blood vessels for the long distance carotid injury and achieved the best result. We expose as fully as possible for the hepatic venae and the posthepatic post caval injury,and adopt effective methods to stop bleeding and repair. The application of blood shunt pump could raise the success rate for those severe patients. The self-double vastransplantation with both ends being all trousers-style one opening solve some problems such as there are no suitable caliber self-transplantation blood vessels and the artificial blood vessels are easy to occur obliteration for long-term and so on for the great or middle blood vessels.We use improved Fogarty duct and anterograde intubation to eliminate thrombus could eliminate thrombus effectively and avoid accessory injury for the deep vein injury and extensive thrombosis.

抢救初期要注意进行有效的心肺复苏、压迫止血和快速进行深静脉插管;颈动脉转流管在严重颈动脉损伤重建过程中有重要作用;对长距离颈动脉损伤用颈外静脉移植外环包人造血管重建,可取得最佳效果;对肝静脉及肝后下腔静脉损伤,要充分暴露,采用有效方法止血和修复,对病情极危重的凶险性出血者,血液转流泵的应用可提高成功率;对大、中血管损伤,两端均为裤式单开口自体双血管移植,解决了无适宜口径自体移植血管和应用人造血管远期易闭塞等问题;对深静脉损伤并广泛血栓形成者,应用改进的Fogarty导管和顺行插管取栓法,可有效取栓并避免了副损伤。

To pay attention to carry out effective cardiopulmonary resus-citation,hemostasis by compression and deep vein intubation rapidly in the initial stage of emergency.The carotid shunt tube plays an important role in severe carotid injury and re-establishment process.We use external jugular vein to transplant and re-establish,which are rungwith artificial blood vessels for the long distance carotid injury and achieved the best result.We expose as fully as possible for the hepatic venae and the posthepatic post caval injury,and adopt effective methods to stop bleeding and repair.The application of blood shunt pump could raise the success rate for those severe patients.The self-double vastransplantation with both ends being all trousers-style one opening solve some problems such as there are no suitable caliber self-transplantation blood vessels and the artificial blood vessels are easy to occur obliteration for long-term and so on for the great or middle blood vessels.We use improved Fogarty duct and anterograde intubation to eliminate thrombus could eliminate thrombus effectively and avoid accessory injury for the deep vein injury and extensive thrombosis.

抢救初期要注意进行有效的心肺复苏、压迫止血和快速进行深静脉插管;颈动脉转流管在严重颈动脉损伤重建过程中有重要作用;对长距离颈动脉损伤用颈外静脉移植外环包人造血管重建,可取得最佳效果;对肝静脉及肝后下腔静脉损伤,要充分暴露,采用有效方法止血和修复,对病情极危重的凶险性出血者,血液转流泵的应用可提高成功率;对大、中血管损伤,两端均为裤式单开口自体双血管移植,解决了无适宜口径自体移植血管和应用人造血管远期易闭塞等问题;对深静脉损伤并广泛血栓形成者,应用改进的Fogarty导管和顺行插管取栓法,可有效取栓并避免了副损伤。

In nude mice, large and invasive tumors were found after injection of HuH7 cells subcutanesously. While mice injected with SeLT cells demonstrated highly vacuolated cells-filled tissue masses, severe necrosis, 4 inflammation as well as degenerated blood vessels.

利用皮下注射肝癌细胞株到裸鼠之后,实验发现HuH7 形成较大的肿瘤、有大量的血管形成且有转移的现象,而SeLT 虽然能够形成tissue mass 但其中大部分的细胞都有严重坏死及发炎的现象产生,伴随著新血管也呈现有萎缩的现象。

As angiotensin II has a potent vasoconstrictive action, while bradykinin is a vasodilator, the reduced formation of angiotensin II and the inhibition of bradykinin breakdown leads to vasodilation.

由于血管紧张素Ⅱ有一个强有力的血管收缩的行动,而缓激肽是一种血管扩张,减少对血管紧张素 II 和形成的抑制缓激肽破裂导致血管扩张。

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