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induced bundle相关的网络例句

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与 induced bundle 相关的网络例句 [注:此内容来源于网络,仅供参考]

Here we present evidence that NMDA receptors not only contribute to normal synaptic responses induced by stimulation of local layer V or white matters, but also contribute to generation of action potentials induced by a depolarizing step applied to the soma.

我们的研究表明,NMDA受体不仅参与刺激前扣带皮层的第五层细胞或刺激白质诱导的突触反应,而且参与在胞体施加去极化跃阶电流诱导的动作电位的发放。

After dialyzing for 10~20 min, SNP at a concentration of 1 μmol/L increased the induced responses to innocuous mechanical stimulation and decreased those to noxoius mechanical stimulation. The induced responses to both innocuous and noxious mechanical stimulations were all decreased after dialyzing the same dose of SNP for 20~30 min. The decrease was shown within 7~15 min dialysis when SNP was used at a concentration of 20 μmol/L.

脊髓深层神经元透析1 μmol/L SNP, 10~20 min后,非伤害性机械刺激诱发的反应增强,伤害性机械刺激诱发的反应减弱;透析20~30 min后,伤害性和非伤害性机械刺激诱发的反应均减弱;透析20 μmol/L SNP 7~15 min内伤害性和非伤害性机械刺激诱发的反应均降低。

Results 甀ntrahepatic vascular diseases, liver tissues diseases and the expressions of VEGF were different in different time of experiment induced by CC14. In earlier periods (2 to Swk) of rats? liver tissues induced by CC14, the inflammatory was significant. Fatting degeneration was seen in hepatocytes around central veins and part of small vesells.

早、中期(2~5wk),肝组织炎症反应显著,中央静脉及部分小血管周围肝细胞脂肪变,VEGF阳性肝细胞主要分布在脂肪变区域;晚期(8~12wk)则肝组织炎症反应轻微,但肝细胞病变显著,血管破坏、血管及成肌纤维细胞增生更趋明显,并见有局部纤维瘢痕形成,假小叶内肝组织肝窦扩张或毛细血管化,其VEGF示阳性。

Flow cytometry analysis showed that the 1% and 2% of lactic acid bacteria fermentation filtrates induced the apoptosis of Hela cells at 48 h and 72 h, while the 5% of the filtrate induced the apoptosis at 24 h, 48 h and 72 h, respectively.

流式细胞仪分析,1%、2%的乳酸杆菌DM9811发酵滤液在48、72h可诱导Hela细胞凋亡;5%的乳酸杆菌发酵滤液在24、48和72h均可诱导Hela细胞凋亡。

Firstly,the property of induced traffic and the role of induced economy in volume forecast are elaborated.After clarifying some inexact concept,the paper recommendes a serial of practicable forecasting methods.According to difference of forecasting method,the concept of released volume is separated from the indued traffic and the basic analysis method is introduced.

阐述了诱增交通量的特性、诱增经济在交通量预测中的作用,在澄清了实际应用中的一些模糊概念后推荐了一套实用的预测方法;根据性质及预测方法的不同,从传统的诱增交通量中分离出释放交通量的概念并对其分析手段进行了初步的探讨。

The results showed as:(1)Gegen decreases fasting insulin and insulin resistance index and increases insulin sensitive index of rats of insulin resistance induced by dexamethasone significantly;(2)Gegen can improve glucose tolerance of rats of insulin resistance but not to alloxan indued diabetes mice;(3)Gegen increases glucose intake of 3T3-L1 adipocytes and improves the insulin resistance of 3T3-L1 adipocytes induced by dexamethasone significantly;(4)Gegen can activate peroxisome proliferator- activated receptor gamma, make it bind to PPAR response elements;(5)Gegen can up-regulate mRNA expression of PPAR v ,aP2 and adiponectin.

结果表明:①葛根显著降低地塞米松诱导的胰岛素抵抗大鼠血清胰岛素水平和胰岛素抵抗指数,升高胰岛素敏感指数,改善大鼠的胰岛素抵抗;②葛根显著改善胰岛素抵抗大鼠的糖耐量,但对四氧嘧啶糖尿病小鼠的糖耐量没有明显的作用;③葛根显著增加胰岛素抵抗3T3-L1脂肪细胞对葡萄糖的摄取,改善地塞米松诱导的3T3-L1脂肪细胞的胰岛素抵抗;④葛根能促进HepG2肝癌细胞过氧化物酶体增殖物激活受体γ与靶基因的特异DNA序列结合,对PPARγ及其下游基因的转录具有激活作用;⑤葛根能够促进PPARγ及其下游基因脂肪酸结合蛋白(aP2)、脂联素mRNA的表达。

METHODS Acute incomplete ischemia was induced by ligaturing bilateral carotid arterise and cutting heads in mice, the survival time and asthmatic time were observed; the coagulation timewas measured by sheet glass method,and the blood viscosity was also assayed; the platelet aggregation induced by collagen were studied by turbdimetry in vitro.

采用结扎小鼠双侧颈总动脉造成急性不完全性脑缺血模型,观察平均存活时间;另取小鼠断颅后测定断颅喘息时间;采用玻片法,观察胡芦巴总皂苷对凝血时间的影响;采用比浊法;观察其对体外血小板聚集率的影响,并测定全血粘度。

It is found that substructures of stress-induced εM are γlamellas and stacking faults. A new growing mechanism of stress-induced εM were raised, that is ε phase nucleates at many sites on parallel γlamellas and stacking faults,εM unit-layers join each other in length and combine in width.

并据此就ε马氏体的形成长大机制提出新的观点:εM相形成时先在层错和γ夹层处多位置形核,然后以εM单元层长度方向彼此相接、宽度方向相互合并的方式长大。

Albicans strains with fluconazole. Method: A series of 16 strains C. albicans from the same body were induced to form pseudo-hyphae in RPMI 1640 medium and DMEM medium at 37 癈 for twenty-four hours respectively. The pseudo-hyphae and yeast were counted and the ratio between pseudo-hypha and total cells was calculated. C. albicans strains were induced to form pseudo-hyphae in RPMI 1640 medium for seven days by transferring twelve times. The genome DNAs of the hyphal-form of C. albicans was cleaved by two restriction enzymes EcoR I and Bgl II, the products were electrophored and photoed.

以同一母体来源的白念珠菌CA-1~A-17(共16株)作为研究对象,采用RPMI1640和DMEM培养基,37℃培养24h诱导其假菌丝形成,记录不同观察时间时的菌丝相和酵母相细胞数,计算菌丝相白念珠菌形成率;以RPMI1640培养基,37℃连续传代培养7天(转种12次)后,分离菌丝相白念珠菌并提取其基因组DNA;以EcoRⅠ和BglⅡ消化菌丝相基因组DNA,消化产物进行琼脂糖凝胶电泳并拍照;用随机引物(5'…TCACCACGGT…3')扩增菌丝相基因组DNA,采用变性聚丙烯酰胺凝胶电泳分离扩增产物并拍照。

Using this model three competence-deficient pneumococci, which was identified by PCR and transformation experiment, were constructed. It is found different strain has different bacterial optimal density for being induced into competence. The optimal density for strains 1, 2 and 22 was 0.1, 0.08and 0.07 respectively. The optimal condition for transformation is c plus y culture at pH8.0. In c plus y culture at pH6.8, which restrained pneumococci forming transformation naturally, pneumococci could be induced to form transformation by CSP.It is concluded that we could induce different S.pneumoniae by CSP into competence in their optimal density in laboratory. 2. It was discussed the relationship between pneumococcal transformation formation and its opportunistic pathogenesis. Comparing their virulence with that of parental strains by challenging mice intraperitoneally and adhering to ECV-304 cell respectively.

结果表明不同肺炎链球菌株的转化最适菌密度不同,1、2和22号菌株的最适转化密度分别为0.1、0.08和0.07;用于转化的CTM培养基最适为pH 8.0的C+Y培养基;在自然转化下不能诱导转化发生的pH 6.8的C+Y培养基中,使用CSP诱导,也能促使细菌发生转化,只是转化效率较低;结论:实验室条件下,不同细菌在一定菌密度时可利用CSP诱导肺炎链球菌成为感受态发生转化。2、为探讨肺炎链球菌转化对毒力表达的影响,本研究分别通过体外粘附人脐静脉血管内皮细胞ECV-304和体内小鼠毒力实验观察1、2和22号肺炎链球菌和其转化缺陷菌株1t、2t和22t毒力的差异。

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他的红酒和白酒的品质,根据酿造者的能力和葡萄的质量,各有差异。

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