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血小板

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Results: The tumor cell-platelet adhesion was significant in SACC-LM than in SACC-83; aspirin, RD, magnesium acetylsalicylate inhibitet the adhesion of SACC-LM in the final concentration of 1, 5 ,25 μ g/ml respectively.

结果:SACC-LM细胞与SACC-83细胞相比和血小板的粘附增高,阿斯匹林、阿斯匹林镁、RD在1,5,25μg/ml时抑制血小板与SACC-LM细胞的粘附,RD能抑制SACC-LM实验性肺转移的形成。

Results: The tumor cell-platelet adhesion was significant in SACC-LM than in SACC-83; aspirin, RD, magnesium acetylsalicylate inhibitet the adhesion of SACC-LM in the final concentration of 1, 5 ,25 μ g/ml respectively. RD inhibited experimental metastasis of SACC.

结果:SACC-LM细胞与SACC-83细胞相比和血小板的粘附增高,阿斯匹林、阿斯匹林镁、RD在1,5,25μg/ml时抑制血小板与SACC-LM细胞的粘附,RD能抑制SACC-LM实验性肺转移的形成。

Methods: Tumor cell-platelet adhesion of highly metastatic SACC-LM, non-metastatic SACC-83 and effect of aspirin, arginine-aspartate , magnesium acetylsalicylate on adhesion were studied in vitro. Antimetastatic effect of aspirin, RD, magnesium acetysalicylate on experimental metastasis of SACC was observed in vivo.

体外实验观察高转移潜能的SACC-LM细胞、非高转移潜能的SACC-83细胞与血小板的粘附,阿斯匹林、阿斯匹林镁、精氨酸-天冬氨酸(arginina-aspartate,RD)对SACC-LM细胞与血小板粘附的影响;动物实验观察阿斯匹林、阿斯匹林镁、RD对SACC-LM细胞实验性肺转移的作用。

The ratio of eicosapentaenoic acid/arachidonic acid was 0.49-0.92 in fish oil group,but 0.02-0.03 in the other two groups.

第6周后,全血和富含血小板血浆中的血小板聚集性,鱼油组显著低于另2组(P.05)。

Platelet adhesiveness and aggregation are related to many blood factors, its' genetic variations may contribute to AR.

血小板粘附、聚集过程涉及多种血液因子,它们的基因多态性有可能影响阿司匹林的抗血小板作用。

Their platelet aggregation, adhesiveness, and the function of their thromboxane were detected and relatively analyzed.

结果 :PE患者血液中血小板粘附、聚集和血栓素测值明显增高,且血小板粘附、聚集和血栓素与 PE疾病呈正相关性。

The purpose of this study was to explore the clinical value of the platelet antibody screening and typing in platelets transfusion by using microcolumn gel immunoassay. The platelets antigen??antibody reactions including the antibody screen and blood crossmatch were detected by MGIA.

为了探讨微柱凝胶免疫分析技术在血小板输注中血小板抗体筛选和配型的临床应用价值,运用微柱凝胶免疫分析技术检测血小板抗原抗体反应,包括抗体筛检、交叉配血。

Results:The results of these 2 methods were(66±17)×109/L and(98 ±2 0)×109/L,their difference was significant(P<0.001).Their influential f actors were large platelets,platelet clumps,cryoglobulins and schistocytes besid es microcytes.

结果:仪器法结果为(66±17)×109/L,而显微镜计数法为(98±20)×10 9/L,两法结果差异有显著性(P<0.001);URI报警时除小红细胞干扰外,还有其他诸多因素;大血小板血小板凝集块、冷球蛋白、衰老破坏的红细胞碎片等,其中最常见的为大血小板

With the development of FCM technology in recent years, above characteristics mentioned, to aim at different antibody of markers on platelet surface, we can gauge function state of platelet.

血小板表面含有不同结构和功能的糖蛋白受体,并有相应配体,血小板活化状态不同,其受体表达也不同,一旦血小板处于活化状态,这些受体的表达也发生变化。

"Overall, the data are the data: they're probably right," Dr Ron Waksman commented, noting that a recent paper from his group showed similar findings. The problem is that most people will generalize from these conclusions after publication, and there is three-vessel disease and there is three-vessel disease. Not all three-vessel disease is the same.

也有人指出CABG和支架组是否完全再血管化还缺乏资料,也没有二联抗血小板治疗的资料,Joseph P Carrozza (Harvard Medical School, Boston, MA)在述评中指出Hannan等的研究要注意延长二联抗血小板治疗的问题,DES治疗的患者较短期的二联抗血小板治疗可能会导致更多的死亡。

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