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AbstractThe endothelial corneal dystrophiesresult from primary endothelial dysfunction,which are genetically and phenotypically heterogeneous.

角膜内皮营养不良endothelial corneal dystrophy，ECD）是由原发性角膜内皮功能紊乱引起的一种角膜营养不良，其遗传方式和临床表型都具有异质性的特点。

Whereas, large dose of L-NAME raised the MABP significantly, constricted pial arteriole (90 minutes, 10%), decreased regional spinal cord blood flow (90 minutes, 22%), augmented the dysfunction of neural system.

表明一氧化氮在继发性脊髓损伤中，小剂量的一氧化氮可能起到保护作用，但过量时又可起到破坏作用。

This is to review the progress in research of fetal cardiac bypass and prevention of placental dysfunction.

随着诊断技术的不断提高，许多胎儿先天性心脏病在宫内即能通过超声心动图早期诊断[1]。

Pneumonedema, alveolar and dysfunction of gas exchange is closely related to the decrease of pulmonary surfactant.

肺水肿、肺萎陷以及换气功能障碍与肺泡表面活性物质(pulmonary surfactant， PS)减少有着密切关系。

Coagulation dysfunction does exist in neonates with pneumorrhagia caused by scleredema and asphyxia. Plasma TXB2 and FPA should be considered as the early objective monitoring indices in neonates who are highly suspected as having pneumorrhagia .

新生儿肺出血患儿存在血小板活化和异常凝血；窒息和硬肿症所致的新生儿肺出血都存在止血凝血功能障碍这一共同因素；TXB2和FPA可作为预测新生儿肺出血的早期客观监测指标。

Coagulation dysfunction does exist in neonates with pneumorrhagia caused by scleredema and asphyxia. Plasma TXB2 and FPA should be considered as the early objective monitoring indices in neonates who are highly suspected as having pneumorrhagia . Lung diseases Infant, newborn Thromboxane B2 Fibrinopeptides A Hemorhage

新生儿肺出血患儿存在血小板活化和异常凝血；窒息和硬肿症所致的新生儿肺出血都存在止血凝血功能障碍这一共同因素；TXB2和FPA可作为预测新生儿肺出血的早期客观监测指标。

ConclusionsCoagulation dysfunction does exist in neonates with pneumorrhagia caused by scleredema and asphyxia. Plasma TXB2 and FPA should be considered as the early objective monitoring indices in neonates who are highly suspected as having pneumorrhagia.Key wordsLung diseasesInfant, newbornThromboxane B2Fibrinopeptides AHemorhage

结论新生儿肺出血患儿存在血小板活化和异常凝血；窒息和硬肿症所致的新生儿肺出血都存在止血凝血功能障碍这一共同因素；TXB2和FPA可作为预测新生儿肺出血的早期客观监测指标。

Methods 19patients（68±9）years with postnecrotic left ventricular dysfunction（ejection fraction31.7%±5.4%）were studied to examine the benefit of adding60mg a day of trimetazidine for3months to the classical therapy.

19例（68±9）岁确诊为缺血性心肌病（左心室射血分数0%）的患者在常规治疗的基础上加用每日60mg曲美他嗪治疗3个月，随诊观察患者临床情况以及超声心动图指标的变化。

objective trimetazidine has shown anti-ischemic properties to improve exercise tolerance without haemodynamic effects and direct cyto protective effect on the myocardium.however,it is not clear whether trimetazidine has beneficially effect on severe ischemic cardiomyopathy.methods 19patients（68±9）years with postnecrotic left ventricular dysfunction（ejection fraction31.7%±5.4%）were studied to examine the benefit of adding60mg a day of trimetazidine for3months to the classical therapy.the assessment included clinical evaluation,electrocardiography and echocardiography.results after adding60mg a day of trimetazidine for3months,attack of angina was relieved and dyspnea was improved（p.05）.but heart rate,systolic blood pressure,rate pressure product,ejection fraction,left ventricular volume and global wall motion index were not changed.conclusion trimetazidine seems to improve clinical status while can not change oxygen supply-to-demand ratio without adverse effects in patients on severe ischemic carˉdiomyopathy.

目的 曲美他嗪是一种新型的抗心肌缺血的药物，能够改善患者的运动耐量和心肌的直接保护作用同时并没有血流动力学效应。但目前对于严重缺血性心肌病患者的临床治疗价值还需要进一步观察。方法 19例（68±9）岁确诊为缺血性心肌病（左心室射血分数0%）的患者在常规治疗的基础上加用每日60mg曲美他嗪治疗3个月，随诊观察患者临床情况以及超声心动图指标的变化。结果 19例患者均可耐受在常规治疗基础上加用每日60mg曲美他嗪的治疗，随诊发现患者心绞痛发作明显减少，运动耐量明显改善。同时患者心率、收缩压、心率血压乘积、左心室射血分数、左心室容量以及整体室壁运动指数无明显改变。结论缺血性心肌病的患者在常规治疗基础上加用曲美他嗪是安全的，同时其能明显改善患者的临床状态，而这种作用并不改变心肌氧的供求比值。

Conclusions— Moderate and severe prearrest left ventricular systolic dysfunction was associated with substantially lower rates of survival to hospital discharge compared with normal or nearly normal function.

结论——与正常或接近正常心功能相比，停跳前中和重度左室收缩功能障碍与显著低下的出院时生存率有关。

So it was last week in Selma , alabama.

上星期在亚拉巴马州塞尔马市发生的事件也是这样一个转折点。

The group also spearheads various social programs in China.

该组织还带头参与中国的各种社会活动。