钌红

Objective To observe the effect of ruthenium red on the body temperature of rats with lipopolysaccharide-induced fever and investigate the possible mechanism.

目的 探讨钌红在脂多糖致大鼠发热过程中对体温的影响及可能的机制。

The results obtained are as follows:(1) intrarenal artery injection of capsaicin (20, 40, and 60 nmol/kg) increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2) pretreatment with ruthenium red (40 mmol/kg), a capsaicin receptor antagonist, completely abolished the effect of capsaicin; and (3) pretreatment with a nitric oxide synthase inhibitor L-NAME ( N6-nitro-L-arginine methylester, 0.1 mmol/kg), significantly enhanced the ARNA response to capsaicin.

结果表明：(1)肾动脉内注射辣椒素20、40和60nmol/kg可呈剂量依赖性地兴奋肾传入纤维的活动，而动脉血压不变；(2)静脉内预先应用辣椒素受体阻断剂钌红(40 mmok/kg)，可完全阻断辣椒素对肾传入纤维的兴奋作用。(3)静脉内预先注射一氧化氮合酶抑制剂L-NAME(0.1 mmok/kg)，能延长并增强肾传入神经对辣椒素的反应。

METHOD Abdominal aortas of rabbits fed with ordinary and high fat diets were stenosed to60.7%.

饲以普通或高脂肪食的兔腹主动脉狭窄60.7％，依万斯蓝、钌红动脉染色。

The data suggest that ETX directly trigger the release of CGRP from capsaicin-sensitive sensory nerve innervating blood vessels.

结果提示：内毒素直接刺激血管周围对辣椒素敏感的感觉神经释放CGRP，这一作用依赖于细胞外钙，并且对钌红敏感的机制。

Pretreatment of MAB with capsaicin or ruthenium red for 30 min inhibited ETX- induced release of CGRP by 90% and 65%, respectively.

应用辣椒素10μmol/L和钌红10μmol/L预灌流30min，内毒素引起的CGRP释放分别被抑制90%和65%。

Pectins are demonstratet by staining with ruthenium red.

可以用钌红来鉴定果胶。

Caffeine (the agonist of RyR1) and ruthenium red (the antagonist of RyR1) had no effect on the E value either.

激动剂咖啡因，抑制剂钌红也不改变RyR1的E值。

But the levels of 〓 elevation were lower than that in normal Tyrode's solution (P＜0.01 respectively), indicating that Ca〓 influx did contribute to the elevation of 〓. Ryanodine remarkably blocked the 〓 elevation by Str 1, 100nmol/L and 10μmol/L in normal Tyrode's solution, while ryanodine 10μmol/L, ruthenium red 10μmol/L and caffeine 10 mmol/L completely blocked the effects of Str on 〓 elevation in Ca〓- free Tyrode's solution (P＜0.01 respectively).

去除细胞外液中的钙离子，Str 1，100nmol/L和10μmol/L仍能分别升高〓到13±5％，71±8％和145±14％，但幅度低于其在正常台氏液中的作用。10μmol/L的ryanodine，10μmol/L钌红或10mmol/L咖啡因均可完全阻断Str 1，100nmol/L和10μmol/L在无钙台氏液中的作用(P＜0.01)。

By pretreatment with ruthenium red (RR, 100 1 μmol/L), an antagonist of vanilloid receptor subtype 1 (VR1), the above effects of CAP on carotid baroreflex were abolished.

2用香草酸受体亚型(vanilloid receptor subtype 1， VR1)阻断剂钌红(ruthenium red， 100 μmol/L)预处理后，CAP的上述反射效应即被阻断。

These stains may fade in time, e.g. ruthenium red. Or the stain may damage the specimen if left, for example SCHULTZE ' S SOLUTION, which dissolves cell walls and causes swelling.

这种染料经过一段时间后会褪色，如钌红；也可能会因为长时间存在而损伤标本，如 Schultze 溶液可以溶解细胞壁使细胞膨胀至死。

Methods: Five patients with parkinsonism or dystonia were assigned to general anesthesia using an modified endotracheal tube.

本实验依照人体实验之相关规定进行，五位患有帕金森氏症或肌张力异常的病人接受神经立体定位手术。

If you can benefit from this book, it is our honour.

如果您能从本书获益，这将是我们的荣幸。