纤维化

Fibrosis is the main cause of ligamentum flaum hypertrophy, and fibrosis is caused by the accumulation of mechanical stress with the aging process, especially along the dorsal aspect of the ligamentum flaum.

纤维化是黄韧带肥厚的主要原因，造成纤维化的主要因素是随着年龄的增长机械应力的积累，特别是在着黄韧带的背侧面。

E, the perchloric acid soluble protein, the phosphatidylinositol transferase, the phosphoglycerate kinase and the endoplasmic reticulum-60 protease;（4） The expressions of the above-mentioned four proteins in the FHD group were nearly the same as those of normal level.

在肝纤维化过程中伴有蛋白质合成与分解异常和糖酵解活动增强；中药调节物质代谢相关蛋白质的表达是抗肝纤维化的途径之一。

MMP-3 proenzyme protein expression in the different stages of liver fibrosis was measured by half quantitative analysis of Western blot.

建立四氯化碳中毒性大鼠肝纤维化模型，采用半定量Western免疫印迹方法，对肝纤维化不同阶段肝内MMP - 3的酶原蛋白的表达水平进行了检测。

Objective:To investigate prophylactic and therapeutic effect and mechanism of Telmisartan on hepatic fibrosis induced by CCl4 in experimental rats.

目的：观察替米沙坦对CCl4诱导的大鼠实验性肝纤维化的防治效果，并探讨其抗肝纤维化的作用机制。

Abstract］ Objective To study the effect of zinc protoporphyrin,an inhibitor of heme oxygenase-1（HO-1）,on pathological and pathophysiologic changes of rat pulmonary fibrosis induced by bleomycin,and to investigate the role of Znpp in the pathogenesis of pulmonary fibrosis.

目的 观察血红素加氧酶-1（HO-1）抑制剂锌原卟啉对博莱霉素致大鼠肺纤维化模型病理和病理生理改变的影响，探讨Znpp在BLM诱导肺纤维化发病中的作用。

Results Beginning from alveolitis, PF in different diseases has similar pathological changes in a progressive and reparative process.

结果不同病种的肺间质纤维化性改变都是从肺泡炎开始，在发展和修复过程中导致的肺纤维化倾向有共同之处。

Here we show that senescent cells accumulate in murine livers treated to produce fibrosis, a precursor pathology to cirrhosis.

在这里我们展示在鼠类的肝脏中堆积衰老细胞用于使肝脏纤维化，肝脏纤维化是肝硬化的前驱病理改变。

Liver fibrosis/physiopathol; liver fibrosis/tcm ther;liver/pathol;achillea alpina/pharmacol;plant extracts/pharmacol;plant extracts/admin;rats

肝纤维化/病理生理学；肝纤维化/中医药疗法；肝/病理学；蓍草/药理学；植物提取物/药理学；植物提取物/投药和剂量；大鼠

Th2 domination and the lack of IFN-γ may be one of the characters of fibrosing alveolitis. It can also promote the development of fibrosis.

Th2优势的反应及IFN-γ的相对缺乏，可能是致纤维化性肺泡炎的特点之一，起到了促进纤维化形成的作用。

Results: In experimental group, dynamic changes from alveolitis to pulmonary fibrosis could be observed in the slices by pathological method.

结果：实验组肺组织病理切片按时间顺序呈现由肺泡炎至纤维化动态改变，实验组肺泡炎和纤维化程度均高于对照组（均P.01）。

What are your goals and strategies for growth?

你的成长目标和策略是什么？

And unto the angel of the church in Sardis write; These things saith he that hath the seven irits of God, and the seven star I know thy works, that thou hast a name that thou livest, and art dead.

3：1 你要写信给撒狄教会的使者，说，那有神的七灵和七星的，说，我知道你的行为，按名你是活的，其实是死的。