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liver相关的网络例句

查询词典 liver

与 liver 相关的网络例句 [注:此内容来源于网络,仅供参考]

Digestion Constipation, diarrhea, loss of appetite, gastroptosis, gastritis, gastric ulcer, duodenal ulcer, haemorrhoids, enteritis, hepatitis, liver malfunction, fatty liver, liver cirrhosis, hangover,(for diabetes, balsam pear juice and enzyme complex 1:1), stomatitis , periodontosis, underweight, obesity.

消化 便秘、腹泻、食欲不振、胃下垂、胃炎、胃溃疡、12指肠溃疡、痔疮、肠炎、肝炎、肝功能不良、脂肪肝、肝硬化、宿醉、(糖尿病、须搭配1:1苦瓜汁)口腔炎、牙周病、太瘦、肥胖。

Results 102 cases of liver and spleen rupture in patients with conservative treatment, Hemoperitoneum full absorption of 73 cases of liver and spleen injuries fully restored, and 29 cases of liver and spleen damage the basic recovery, residual 2~3 cm in diameter around a little low-density area or a computerized stove, follow-up 1~3 months, respectively, disappeared.

结果 102例肝脾破裂患者经保守治疗,腹腔积血全吸收,73例肝脾损伤完全恢复,29例肝脾损伤基本恢复,残留少许直径 2~3 cm左右低密度区或机化灶,随访分别于1~3个月基本消失。

This depends on the damage of liver and kidney functions ,the obese patients with inapparent damage of liver and renal function can take this product,they shoud strengthen the self-monitoring at the same time.the obese patients with serious damage of liver and renal function should not take this product.

根据肝、肾功能的损坏程度不同:肝、肾功能损坏不明显的肥胖者可以服用该产品,同时加强自我监控;肝、肾功能损坏严重的一般不主张服用该产品

2TGF-β1 and PDGF-B mRNA in liver tissue of liver-fibrotic rats can express increasedly, TGF-β1 and PDGF-B relate with the happening and developing of liver fibrosis closely.

肝纤维化大鼠肝组织TGF一p,和PDGF一B mRNA表达增加,TGF一pl和PDGF一B与肝纤维化的发生发展关系密切。

Results 20 cases of CT examination had made proper diagnoses,the CT imaging finding about the most of peripheral cholangiocarcinoma included:There was the atactic low density tumour in liver,its borderline was unclear,enhanced scan appearances were irregular peripheral enhancement,centripetal enhancement,and delaying enhancement;There was concretion and dilatation of bile buct in or around the focus;The local envelope of liver was lacunose and the local liver was atrophied,There was abroad putrescence and liquescence in the focus.

结果 20例CT做出了正确的诊断,部分周围型胆管细胞癌的CT表现肝内不规则低密度肿块边界欠清,增强扫描表现病灶边缘呈不规则环状强化,有向心性强化的趋势,出现延迟强化;病灶内或周围可见结石,病灶内及周围肝内胆管扩张;局部肝包膜回缩、肝叶萎缩,病灶内大片坏死、液化。

It was suggested that the pharmacodynamics and therapeutic function of Qizhi complex prescription to fatty liver were: 1. Adjust lipometabolism: reducing TC and TG; 2. Decreasing fatty degeneration of liver cell; 3. Promoting the convalescence of hepatocellular function; 4. Anti-lipid peroxide: increasing the activation of peroxidase, decreasing the content of products of lipid peroxide. 5. Restraining Kupffer cell function of fatty liver rat; 6. Improving fluidity of hepatocellular microsome and mitochondria.

芪枳合剂治疗脂肪肝的效应及药效学机制为: 1、调整脂质代谢:降低血清甘油三脂和胆固醇; 2、减轻肝细胞脂肪变性; 3、促进肝细胞功能恢复,有效治疗脂肪性肝炎; 4、抗脂质过氧化:提高抗氧化酶活性、降低脂质过氧化产物含量; 5、抑制脂肪肝大鼠Kupffer细胞功能; 6、改善肝细胞微粒体、线粒体膜流动性。

The level of HLD-C was higher.,the level of MDA in Tiaozhi Hugan decoction was lower significantly, the level of SOD was higher significantly(in all group P<0.01)Conclusion It suggests that the pharmacodynamics mechanisms of Tiaozhi Hugan Decoction to fatty liver induced by high fatty diet are lowering body weight and liver index, adjusting lipometabolism, improving liver function and having anti-lipid peroxidation effects.

调脂护肝方干预非酒精性脂肪肝的药效机制为:降低体重与肝指数,调整脂质代谢,改善肝功能,抗脂质过氧化。

Objective To analyse the correlation between changes of liver hemodynamies and liver fibrosis markers in patients with trichloroethylene medicamentous dermatitis and furthermore study the prognosis of these patients with liver disfunction.

目的 探讨三氯乙烯药疹样皮炎患者肝脏血流动力学参数改变与肝纤维化指标的关系,为判断三氯乙烯药疹样皮炎肝损害的程度知预后提供依据。

Xiao Chai Hu Tang ;; Nitrosamines ;; Liver Cirrhosis, Experimental ;; Liver Fibrosis ;; Muridae ;; Swine ;; Serum ;; Fibrosis ;; Cells ;; Liver

小柴胡汤;;亚硝胺类;;肝硬化,实验性;;肝纤维化;;鼠科;;猪;;血清;;纤维化;;细胞;;肝

Endurance training increased the activity of iNOS in liver , thereby in2 creasing the level of NO , but the high level of NO didn' t result in the oxidative injury of the liver. I t indicated that the protection or oxidative injuri of nitric oxide in nonalcohol fatty liver was closely related to the physiological state of life.

耐力运动引起非酒精性脂肪肝小鼠肝脏iNOS活性升高,导致 NO水平升高,而高水平的 NO并未造成肝脏的氧化损伤,说明 NO水平的升高并不一定造成氧化损伤,其是否具有氧化损伤作用与机体的生理状态有关。

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