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induces相关的网络例句

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与 induces 相关的网络例句 [注:此内容来源于网络,仅供参考]

The cyclic fluctuation of the growth velocity adjacent to the absolute stability velocity of plane interface growth induces the banding structures.

无偏析结构中溶质分布在测量误差范围内完全均匀,表明该实验已达到高速绝对稳定平界面生长。

Kanzawa pointed, the abdominal vertebra BMD of PTH gene individuals in BB genetype is obviously higher than those of Bb and bb genetype,so he considered that the BstBI polymorphism of PTH gene has dependability with BMD while the DraⅡpolymorphism of PTH gene has no dependablity with BMD.3 The polymorphism of estrogen receptor gene and postmenopausal osteoporosisPMOP is a metabolic bone disease existing in post-menopause femmes, as a result of these femmes\' hypofunction of ovary,ESG level drawdown and the disequilibrium of bone couple procedure,the bone resorption of osteoclast exceeds the bone formation of osteoblastic,osteopenia and bone fragility accrescence have developed,then induces fracture and so on.

Kanzawa等研究发现,BB型PTH基因人的腰椎BMD较Bb、bb型显著为高,认为PTH基因BstBⅠ多态性与BMD有相关性;但PTH基因DraⅡ多态性则与BMD无相关性。三雌激素受体基因多态性与绝经后骨质疏松绝经后骨质疏松是指绝经后妇女由于体内卵巢功能低下,雌激素水平低落,骨偶联过程失衡,破骨细胞的骨吸收大于成骨细胞的骨形成作用所导致的骨量减少,骨脆性增加,进而易于发生骨折的代谢性骨疾病。

It is concluded that acrolein induces adult mice cardiomyocytes apoptosis, and it may be due to the increased intracellular oxygen free radicals and calcium concentration.

丙烯醛可引起成年小鼠心肌细胞的凋亡,其作用可能是由于细胞内氧自由及钙浓度的增加。

Experiments show that Ang Ⅱ activates JNK mediated by AT〓, and JNK increases transcription activity of c-Jun by phosphorylating c-Jun amido-terminal, and induces expression of c-Fos gene. c-Jun and c-Fos form transcriptional factor activated protein-1 complex. AP-1 may stimulates genes which correlate with cardiovascular remodeling expression, such as transforming growth factor, collagen type Ⅰ and Ⅲ, skeletal muscle α-actin,β-MHC, atrial natriuretic factor.

实验表明,在心肌和血管平滑肌细胞Ang Ⅱ通过AT〓介导激活JNK;JNK通过磷酸化c-Jun氨基末端增加c-Jun的转录活性并诱导c-Fos基因表达,c-Jun和c-Fos蛋白形成转录因子复合物激活蛋白-1(AP-1),并刺激各种心血管重塑相关基因如转化生长因子-β1(TGF-β1)、Ⅰ,Ⅲ型胶原、骨骼肌α-actin、β-MHC(β-myosin heavy chain,β-肌球蛋白重链)、心钠素的表达。

1The axial evolution law on the structure and the aerodynamic characteristics of the asymmetric flows around slender bodies of revolution at incidences is summarized:whether the flow asymmetry is powerless or powerful,the flow always appears in the structure that lee-side vortices rise,and shed vortices form,and new lee-side vortices come into being alternately on opposite sides of a slender body of revolution axially from nose apex downstream to afterbody,and always induces the sectional side force of waving quasi-sinusoidally downstream axially.

1总结出细长旋成体有攻角非对称绕流流场结构和气动力特性的轴向演化规律:无论非对称性强弱,沿轴向从头部尖顶往后体,非对称绕流总是呈现为左、右两侧背涡交替从物面向上飘起、形成脱落涡、再衍生出新生背涡的流场结构,并诱导出沿轴向呈类似正弦曲线形式振荡分布的截面侧向力。

In the view of the description of Secularity and Afterworld in the Quran and the Sunnach, it analyses and induces the concrete contents and thinking.

第二,从伊斯兰"两世"思想的基础、体现及精神实质的角度进行分析、研究,并探讨伊斯兰哲学各个流派对"两世"思想的不同阐释。

A hypnotist is someone who induces the state of hypnosis, and a hypnotherapist is a person who induces hypnosis to treat physical or mental illnesses.

催眠师是指某个引起催眠状态的人;而催眠医疗师是指出于治疗身体或精神疾病而引导产生出催眠的人。

The major results of this proposal are as follows:(1) ABA induces a rapid, substantial accumulation of apoplastic H2O2 in mesophyll and bundle sheath cells of maize leaves, and the accumulation of apoplastic H2O2 is involved in the induction of the chloroplastic and cytosolic antioxidant enzymes.(2) ABA-induced H2O2 production activates a 46 kDa MAPK, which in turn induces the expression of antioxidant genes and up-regulates the activities of antioxidant enzymes. The activation of MAPK also enhances H2O2 production, forming a positive amplification loop.(3) Water stress also induces the activation of a 46 kDa MAPK, which is dependent on the accumulation of ABA and H2O2 production induced by water stress and involved in the up-regulation of the expression and the activities of antioxidant enzymes.(4) ABA-induced H2O2 production mediates NO generation, which in turn activates a 46 kDa MAPK and results in the up-regulation in the expression and the activities of antioxidant enzymes in ABA signaling. NO-independent signaling is also involved in ABA- and H2O2-induced antioxidant defense.

本项目的主要研究结果如下:(1)ABA诱导的H2O2产生主要出现在叶肉细胞与维管束鞘细胞的质外体中,质外体H2O2的积累能够上调叶绿体与细胞溶质中抗氧化酶的活性;(2)ABA诱导的H2O2产生活化一个46kDa的MAPK,由此而导致编码抗氧化防护酶基因的表达与酶活性的上调;而MAPK的活化也能增强H2O2的产生,从而形成一个正的反馈调节环;(3)水分胁迫也能诱导一46kDa MAPK活化,这一MAPK活化依赖于水分胁迫诱导的ABA积累以及H2O2的产生,同时参与水分胁迫诱导的抗氧化防护基因的表达与抗氧化酶活性的上调;(4)ABA诱导一个依赖于H2O2的NO产生,NO活化一个46kDa的MAPK,从而导致抗氧化防护基因的表达以及抗氧化酶活性的上调;同时一个不依赖于NO的信号转导途径也存在于ABA诱导的抗氧化防护过程中。

It was found from structure-electrochemical studies of hemoglobin that (1) electroreduction induces the T→R transition with chirality transition and umbrella conversed effect;(2) electric field induces the secondary structural transition between two α helices, which is smilar to the denaturalization of the protein;(3) The electrochemical reaction mechanism of EC including T→R transition and parameters have also been obtained.

在血红蛋白的结构-电化学研究中发现:(1)电化学还原诱导血红蛋白的T→R构型转化,手性转化的伞翻转效应;(2)电场诱导血红蛋白的两种α螺旋二级结构间的转化,属于电场诱导血红蛋白的部分变性;(3)获得血红蛋白电化学还原的包括R→T转换的EC机理和参数

1、Cur inhibits K562 cells growth and induces cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as p-Erk1/2、c-myc which are relevant with cell growth and apoptosis; 2、Cur synergizes STI571 to inhibit K562 cell growth and induce cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as Hsp90、PKC which are relevant with cell growth and apoptosis; 3、Cur reverses the resistance of K562/G01 cells to STI571, and synergizes STI571 to inhibit K562/G01 cell growth and induce cell apoptosis; 4、Cur inhibits human originated CML CD34~+ cell growth、induces cell apoptosis, and enhances STI571 to down-regulate the expression of p210~, finally inhibit cell growth and induce cell apoptosis.

从以上实验结果我们得出如下结论: 1、Cur抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游p-Erk1/2、c-myc等信号分子有关; 2、Cur协同STI571抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制Hsp90和下游PKC等信号分子有关; 3、Cur可逆转K562/G01细胞对STI571的耐药性,并与STI571协同抑制K562/G01细胞增殖和诱导凋亡,其抑制K562/G01细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游Procaspase-3和NF-κB等信号分子有关; 4、Cur可抑制来源于CML患者骨髓的CD34~+细胞的增殖并诱导其凋亡,还可协同STI571下调CML CD34~+细胞p210~表达,进而协同抑制细胞增殖、诱导细胞凋亡。

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