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Vascular endothelial growth factor is a major angiogenic factor involved in physiological as well as pathological angiogenesis.

生理和病理性血管生成中，血管内皮生长因子(vascularendothelialgrowthfactor，VEGF)是主要的血管生成因子。

Vascular endothelial growth factor is an angiogenic factor that promotes the growth of solid tumor by inducing angiogenesis.

实质固态的肿瘤细胞生长可藉由血管内皮生长因子激活血管的形成。

The extension of the beam propagation factor (M~2 factor) to the apertured case is discussed.

对有光阑情况时光束传输因子(M2因子)的推广作了讨论。

Objective To evaluate the therapeutic effect of vascular endothelial growth factor and tumor necrosis factor receptor on avascular necrosis of the femoral head in rabbits.

目的 探讨应用血管内皮细胞生长因子与肿瘤坏死因子受体联合治疗股骨头坏死的治疗效果。

Presence of a genetic factor like azoospermia factor microdeletions may lead to irreversible spermatogenic arrest in these cases.

遗传因子如无精子因子微缺失的存在可能在这些病例中导致不可逆的精子发生阻滞。

The ionomer membrane under the conditions of 20℃ and 0.05 MPa shows better permeaselectivities that the permeability coefficient of CO_2 and CO_2/ N_2 separation factor are 223 Barrer and 65.70, respectively, while CO_2 / CH_4 separation factor is only 14.70. An obvious increase in P_(CO_2) with decreasing pressure difference exhibits a characteristic of facilitated CO_2 transport, which is not disappear and almost unchange after two months. There is no this behavior for N_2 and CH_4. Hence, P_(CO_2) and α_(CO_2/N_2) and α_(CO_2/CH_4) can be enhanced simultaneously.

该离聚体膜显示出良好的CO_2渗透性能，在20℃和0.05MPa压差下，CO_2渗透系数P_(CO_2)和CO_2/N_2分离系数αCO_2/N_2分别高达223 Barrer和65.70.P_(CO_2)和αCO_2/N_2随CO_2压差的降低而明显增大，显示出促进输送特征，这一行为即使在膜放置2个月之后并不消失，渗透分离性能变化不大。N_2和CH_4无此性能，因此在气体压差较低时，P_(CO_2)和αCO_2/N_2、αCO_2/CH_4可同时提高。

The ionomer membrane under the conditions of 20℃ and 0.05 MPa shows better permeaselectivities that the permeability coefficient of CO_2 and CO_2/ N_2 separation factor are 223 Barrer and 65.70, respectively, while CO_2 / CH_4 separation factor is only 14.70. An obvious increase in P_(CO_2) with decreasing pressure difference exhibits a characteristic of facilitated CO_2 transport, which is not disappear and almost unchange after two months. There is no this behavior for N_2 and CH_4. Hence, P_(CO_2) and α_(CO_2/N_2) and α_(CO_2/CH_4) can be enhanced simultaneously.

该离聚体膜显示出良好的CO_2渗透性能，在20℃和0.05MPa压差下，CO_2渗透系数P_(CO_2)和CO_2/N_2分离系数αCO_2/N_2分别高达223 Barrer和65.70.P_(CO_2)和αCO_2/N_2随CO_2压差的降低而(来源：e2b2A2bBC论文网www.abclunwen.com)明显增大，显示出促进输送特征，这一行为即使在膜放置2个月之后并不消失，渗透分离性能变化不大。N_2和CH_4无此性能，因此在气体压差较低时，P_(CO_2)和αCO_2/N_2、αCO_2/CH_4可同时提高。

Objective 1. To investigate the expression of protein of nometastatic gene 23(nm23H1),vescular endothelial growth factor and protein of nuclear factor kappa B ( NF-kBP65) in retinoblastoma, blepharal basal cell carcinoma , blepharal squamous cell carcinoma and meibomian gland carcinoma. 2. To explore the relationship between expressions of these proteins and biological behavior of these ocular malignancies.

目的：1、检测肿瘤转移抑制基因蛋白nm23H1、血管生成因子VEGF以及细胞核因子NF-kBP65蛋白在视网膜母细胞瘤、眼睑基底细胞癌、眼睑鳞状细胞癌和睑板癌中的表达。2、探讨它们与眼部恶性肿瘤的生物学行为的关系。

Dialectic retinopathy,the most common microvascular complication of diabetes is the leading cause of blindness.Many drugs have been used to prevent the development of it.The primary drug treatment includes the intensive control of blood glucose,blood pressure and the reducing ofblood-fat. The directed against drug treatment includes advanced glycation end products inhibitor, vascular endothelial growth factor inhibitor,inhibitive factor promoter,supplement of inosital, aldose reductase , protein kinase C inhibitors, GH inhibitors,improvement of retinal microcirculation, DAG-PKC inhibitor, ginkgo blibe extract, antioxidant.

糖尿病视网膜病变是最常见的糖尿病微血管病变，目前有很多致力于延缓糖尿病视网膜病变的药物研究，包括严格控制血糖、血压，降低血脂的基础药物治疗，以及蛋白非酶糖基化终末产物抑制剂、血管内皮生长因子抑制剂和抑制因子促进剂、肌醇的补充及醛糖还原酶抑制剂的应用，及蛋白激酶C抑制剂、生长激素抑制素、改善视网膜微循环药物、DAG-PKC抑制剂、银杏叶提取物、抗氧化剂等针对性药物的应用。

The edge metal of round hole often splits if the burring factor is less than the limiting burring factor.

当孔缘处变形程序超过极限变图 1 圆孔翻边示意图形程度时，孔缘处就会发生破裂。

Don not attempt to do something which you can not to do.

不要企图做那些办不到的事情。

The expression of CTGF and TNF-αweredetected by immunochemistry and the number of Clara Cells was calculated.

光镜下观察肺组织的病理变化，采用免疫组化染色观察肺组织中结缔组织生长因子和肿瘤坏死因子-α的表达和Clara细胞的数量。