查询词典 deficiency statement

Deficiency could lead to sthenia syndrome,whereas sthenia syndrome also causes deficiency,generating a vicious circulation.Muddy stasis as the pathologic products,obstruct in renal collateral throughout the whole disease course.

其中瘀浊内停为本病的病理产物，瘀阻肾络可见于本病的全过程，从丹参用药占51.8%显而易见，且用药最多的前22种药物中，活血化瘀通络之药竞占11种。

Chinese medicine, when I look and I said I Qi-deficiency and blood deficiency, I had the summer of 2000 in B-and sex hormones 7, all normal, in February 2001 have they done that I B super-normal uterine size, accessories and there was no problem I did not take contraceptive measures, has not been pregnant, I am What is this disease?

以前我看中医时和我说我气虚血虚，我在2000年夏做过B超和性激素7项，一切正常，于2001年2月又做过B超说我子宫大小正常，附件也没有问题，我没采取过避孕措施，一直没有怀孕，我这是什么病？

Choose the person that feed content constitution deficiency of yin with irritability to answer to be given priority to in order to eat food of method of treating yin deficiency by reinforcing body fluid and nourishing the blood according to the constitution, wait like soup of lean lean, soft-shelled turtle, testacean food, lotus seed and tremella.

根据体质选择食物体质阴虚者应以进食滋阴食物为主，如瘦肉、甲鱼汤、贝壳类食物、莲子和银耳等。

Shortening of telomere may be one of pathomechanism of spleen Qi deficiency syndrome. The accumulation of free radical produced by the decrease of the antioxygen ability may be damage the telomere, and that may be one of the mechanisms of telomere shortening in liver of rat model with spleen Qi deficiency syndrome.

提示端粒长度缩短可能是脾气虚证的病理机制之一；脾气虚证状态下，机体的抗氧化能力下降、自由基蓄积损伤端粒可能是脾气虚证模型组大鼠肝组织端粒长度缩短的机制之一。

Results: The changes following hydrocortisone intervention of rats could be identified by PCA, and the kidney deficiency syndrome rats entered a state of "hyperfunction", involving a series of changes in metabolic pathway and metabolic network. The response integral area of lactic acid (δ 1.37) increased, indicating the accumulation of metabolites of lactic acid. The amount of dimethylamine (δ 2.72) also increased, indicating that the disorder of kidney function was related to kidney-deficiency syndrome. The response of creatinine (δ 3.87), asparaginic acid (δ 2.83), taurine (δ 3.44, 3.28), and hippurate (δ 7.84, 7.56, 7.64, 3.97) all decreased significantly, indicating that the hydrocortisone might cause adrenal cortex excretion injury. The response integral area of succinate (δ 2.41) and citric acid (δ 2.53, 2.68) decreased, which was usually caused by disorder of the mitochondrial function.

结果：PCA方法处理肾虚证和空白对照大鼠尿液数据显示，两组大鼠的数据可以在得分图实现分类，与对照组比较肾虚组大鼠由于氢化可的松诱导，大鼠机体进入一个过消耗后衰弱的状态，相关代谢发生显著变化，乳酸(δ 1.37)代谢发生堆积；二甲胺(δ 2.72)的含量增加，提示肾虚与肾功能异常是密切相关的；天冬氨酸(δ 2.83)，牛磺酸(δ 3.44， 3.25)，马尿酸(δ 7.84， 7.56， 7.64， 3.97)，肌氨酸(δ 3.87)等的相对积分面积明显下降，这预示糖皮质激素可能引起了肾上腺皮质分泌功能的损害；琥珀酸(δ 2.41)和柠檬酸(δ 2.53， 2.68)是三羧酸循环能量代谢及糖酵解的中间产物，其含量的降低，通常是由于线粒体功能紊乱所引起。

Results The long term results of the vascularized fibular graft varied in different types of bone defects. Nine patients with congenital pseudoarthrosis of tibia had the limb function restored in 65％ and one had nonunion of fibular graft, 3 fractures(7 times), and 5 angular deformities. Three patients with congenital radial deficiency had 62％ restoration of the limb function. All of them showed early closure of epiphyses of the grafted fibulae. Five cases with osteomyelitis and bone defects combined soft tissue deficiency had 94％ restoration of the limb function, and only one occurred fracture of fibular graft. One case with soft tissue defect and concomitant ulnar defect, ulnar nerve and median nerve injury, 67％ function of limb was recovered, and union of fibula graft was good. 100％ function of limb was recovered in 1 case with cavernoma of radius and without occurring valgus deformity of donor site.

结果应用带血管腓骨移植修复骨缺损的远期效果因缺损类型的不同而存在较大差异。9例先天性胫骨假关节患者，术后肢体功能恢复65％，其中移植腓骨骨不连1例、骨折3例(7次)、成角畸形5例；3例先天性桡骨缺如患者，术后肢体功能恢复62％，3例术后均出现移植腓骨骨骺线早闭，畸形复发；5例骨髓炎、骨外露、骨缺损并软组织缺损患者，术后肢体功能恢复94％，仅1例发生移植腓骨骨折；1例前臂软组织缺损并尺骨缺损，尺神经、正中神经损伤者，术后肢体功能恢复67％，骨愈合顺利；1例桡骨海绵状血管瘤者，术后肢体功能恢复100％，骨愈合顺利。

Two clinical phenotypes for citrin deficiency have been reported. One is adult-onset citrullinemia type II (CTLN2) and another is neonatal intrahepatic cholestasis caused by citrin deficiency.

Citrin缺陷病(citrin deficiency， CD)已报道两种临床表现型，一种是成人发病瓜氨酸血症Ⅱ型(CTLN2)，另一种是citrin缺陷导致的新生儿肝内胆汁淤积症。

A further study on 65cases of kidney-YIN deficiency syndrome under sub-health status has shown that themain clinical menifetation of kidney-YIN deficiency syndrome under sub-healthstatus are: waist and back being ache, tiredness lack of strength, feeling leg and kneeweak, dry throat, insomnia and dreamful sleep, losing hair, night sweat, constipateion, tinnitus, eye acerbity, feverish sensation in palms and soles, etc.

进一步对其中65例亚健康(来源：ABC论文3c3c3c网www.abclunwen.com)状态肾阴虚证的表现研究发现，较为常见的症状有腰背酸痛、疲倦乏力、头晕、记忆力差、怕热、耳鸣、腿膝酸软、咽干、失眠多梦、心慌、反应迟钝、手脚心热、容易出汗、少气懒言、入睡出汗、手足麻木等。

Manifetation of deficiency of liquid(mainly eye acerbity, dry throat, constipateion,etc); 3. Manifetation of hyperactivity of fire due to yin deficiency and heart soultroubled by heat(mainly, night sweat, feverish sensation in palms and soles, insomniaand dreamful sleep, ect); 4. Manifestations of tontue and pulse of kidney-YINdeficiency syndrome are mainly reddened tongue and deep slim pulse.

进一步应用因子分析等方法分析，得出亚健康状态肾阴虚证的主要证候特点有：①肾虚证表现(主要为腰背酸痛、腿膝酸软、疲倦乏力、耳鸣、脱发等)；②阴液亏虚表现(主要为眼涩、咽干、大便干结等)；③阴虚火旺，热扰心神等表现主要为盗汗、手心发热、失眠多梦等；④亚健康状态肾阴虚证的舌脉以舌红、脉沉细为主。

Improvement for the dysmnesia of memory due to chronic blood deficiency in brain of rats induced by ligating the carotid arteries of two sides were observed. Results FBD can improve the dysmnesia of mice induced by scopolamine, NaNO2, and 45% ethanol significantly, also dysmnesia due to chronic blood deficiency in brain of rats induced by ligating the carotid arteries.

结果 FBD对由东莨菪碱所致的小鼠记忆获得障碍、亚硝酸钠所致的小鼠记忆巩固不良和45%乙醇所致的小鼠记忆再现障碍均有明显的改善作用；并可改善大鼠双侧颈总动脉结扎所致的慢性脑部供血不足而产生的记忆障碍。

The second is to cut a mere 100 calories from your diet , which is the equivalent of a tablespoon of butter.

第二个就是从你的饮食中减少一百卡路里，只是相当于一大汤匙的黄油。

This is where the Rorschach test comes in.

现在，罗氏测试就出现了。