查询词典 cholesterol

Low density lipoprotein. This is often known as bad cholesterol and is thought to promote arterial disease.

低密度脂蛋白，这是常常被称为坏cholesterol 和被认为是促进动脉疾病。

The normal control group, hyperlipidemia model group, high dose of perilla seed group (2.5 g/kg), low dose of perilla seed group (0.83 g/kg) were designed to measure the contents of total cholesterol, three acids glyceride, high density lipoprotein cholesterol, low density lipoprotein cholesterol, very low density lipoprotein cholesterol and malondialdehyde, superoxide dismutase activity.

方法］采用高脂饮食建立鹌鹑高脂血症模型，分别测定正常对照组、高脂模型组、紫苏籽高剂量组(2.5g/kg)、低剂量组(0.83g/kg)血清中总胆固醇、甘油三脂、高密度脂蛋白、低密度脂蛋白、极低密度脂蛋白、丙二醛含量及超氧化物歧化酶的活性。

Cholesterol gallstones formed in C57L mice and fatty livers developed in AKR mice. Conclusions Biliary cholesterol hypersecretion is the key pathophysiological defect of gallstone formation, lith genes have effects on biliary cholesterol hypersecretion and susceptibility to cholesterol gallstone formation in C57L mice. Lithogenic bile is formed at the canalicular membrane and precedes the development of cholesterol gallstones. It is most likely that cholesterol and bile acid hyposecretion make the AKR strain susceptible to the development of fatty livers and resistant to gallstone formation.

结论胆道胆固醇的高分泌是胆囊胆固醇结石形成的主要病理生理基础，结石基因决定了C57L鼠肝内胆汁中胆固醇的高分泌和胆囊胆固醇结石的易患性，成石胆汁形成在肝内胆管，先于胆囊结石的形成；肝内胆汁中胆固醇和胆酸的低分泌可能与AKR鼠脂肪肝的发生和胆囊结石的免患性有关。

Methods Forty-five female rabbits were randomly divided into 5 groups, there are 9 in each: group A: normal control; group B: sham operation + cholesterol feeding; group C: OVX + cholesterol feeding; group D: OVX + cholesterol feeding + estradiol benzoate; group E: OVX + cholesterol feeding + estradiol valerate.

将45只3月龄雌兔随机分为5组，每组9只。A组为正常对照，B组为假手术+胆固醇饮食，C组为OVX+胆固醇饮食，D组为OVX+胆固醇饮食+苯甲酸雌二醇，E组为OVX+胆固醇饮食+戊酸雌二醇。

Methods Rabbit model was adopted with gallstone induced by high cholesterol diet. Dynamic study on the serum lipoprotein cholesterol, hepatic total cholesterol, bile cholesterol and glycocholate, and low density lipoprotein recepter activity of hepatocytes had been conductd for the test and control groups after 1,2,3 and 4 weeks respectively.

采用高胆固醇膳食诱发兔胆囊结石模型，对进食高胆固醇膳食后1，2，3，4周实验组和对照组血清脂蛋白胆固醇、肝脏总胆固醇、胆汁中胆固醇和甘氨胆酸，肝细胞低密度脂蛋白受体活性变化进行了动态研究。

Methods: Foty healthy New Zealand white rabbits were randomly divided into four groops. Control groop was fed with normal diet for 10 weeks, model groop was fed with 1.5% cholesterol diet plus normal diet; APS groop was fed with 1.5% cholesterol plus 12.0mg/kg APS plus normal diet; Captopril groop was fed with 1.5% cholesterol plus 15.0mg/kg Captopril plus normal diet. After ten weeks' experiment, the concentration of each group's Triglyceride, Total Cholesterol, High Density Lipoprotein Cholesterol, C Reactive Protein, Nitric Oxide, Maleic Dialdehyde and Endothelin-1 were measured, so was the activity of superoxide dismutase.

将40只雄性新西兰兔随机分为4组，每组10只，对照组餵普通饲料；模型组：普通饲料加质量分数为1.5%的胆固醇餵养；黄芪多糖组：普通饲料加质量分数为1.5%的胆固醇餵养，同时腹腔注射APS(12.0mg/kg)；卡托普利组：普通饲料加质量分数为1.5%的胆固醇餵养，同时灌喂卡托普利(15.0mg/kg)；10周后，测定各组空腹血清或血浆三脂酰甘油、总胆固醇、高密度脂蛋白胆固醇、一氧化氮、内皮素-1(ET-1)、丙二醛、C反应蛋白含量以及超氧化物歧化酶活性，并计算主动脉内膜粥样斑块面积。

Methods Twelve rabbits fed with high-cholesterol diet for 8 weeks wererandomly divided into two groups:(1) high cholesterol group (n=6):maintained on high cholesterol diet for 6 weeks;(2) niacin group (n=6):the same cholesterol diet plus niacin 200mg·kg~(-1·d~(-1) for 6 weeks. Controlgroup (n=6) was fed with normal diet for 14 weeks. Subcutaneousadipose was collected from inguen for RNA analysis. Leptin and MCP-1levels in serum and adipocytes culture supernatant were measured byELISA.

选取12只健康雄性新西兰兔给予高胆固醇饲料培养8周，建立高胆固醇兔模型后，随机分为：(1)高胆固醇组：继续饲以高胆固醇饲料6周；(2)烟酸治疗组：在饲以高胆固醇饲料的基础上给予烟酸缓释剂0.2g.kg~(-1)。d~(-1)，共6周；另选普通饮食14周雄性新西兰兔(n=6)作为对照组。

Methods Rabbits were randomized to three groups. Complex model group were fed with feed containing 0.5% cholesterol for 3 weeks. At the fourth, eighth and twelfth weeks, rabbits were given injection of lipopolysaccharide (1 μg) at auricular artery, cervical part and fold inguen, respectively. Normal group were fed with normal diet, while High cholesterol group were fed with feed confaining 0.5% cholesterol only. After 16 weeks, vascular tissues were analyzed using morphometric and histological methods, and the sera were checked for cholesterol, LDL-C, C-reaction protein and tumor necrosis factor-α. Results The cholesterol and LDL-C of High cholesterol group were increased, compared to those of normal group.

给含0.5%胆固醇的饲料，3周后，分别在第4、8、12周采用耳动脉内、颈部、腹股沟处肌肉注射大肠埃希菌脂多糖，并设立正常组和单纯高脂组。16周后观察兔的一般状态，取血清检查血脂六项、C-反应蛋白和TNF-α，取耳动脉、颈动脉、主动脉弓、胸主动脉、腹主动脉、髂动脉、肝脏，放置4%多聚甲醛中过夜，常规行HE染色，检查血管病变和相关脏器病变情况。

Objective To survey the relationship between nutrimental condition and the levels of serum lipids and lipoprotein. Methods The levels of total cholesterol, triglycerides high density lipoprotein cholesterol, low density lipoprotein cholesterol, apolipoprotein A1 (apoA1) and apolipoprotein B100(apo B100) B100 in 514 middle school students aged from 12 to 15 years old in Haikou City were determined.

目的 探讨营养状况与血脂及载脂蛋白水平的关系方法检测了海口市514例12～15岁中学生的营养状况并分析其与血清总胆固醇(Total-Cholesterol，TC)、甘油三酯(Triglycerides，TG)、高密度脂蛋白胆固醇(High Density Lipoprotein Cholesterol，HDL-C)、低密度脂蛋白胆固醇(Low Density Lipoprotein LDL-C)、载脂蛋白(Apolipoprotein，apo)A1及B100的关系。

PlasmaLDL was decreased markedly in cholesterol gallstone than in control group (p0.05). In cholesterol gallstone group, plasma TG and TG/TC ratio were significantly positive correlativity. Conclusions: These results indicate that the changes of plasma lipids may affect the metabolism of cholesterol and bile acids and itmay tade an important role in the formation of cholesterol gallstone. TG/TC ratio may act as an indirect reflect of reference index.

本实验结果提示：血浆脂蛋白的变化，可能因影响体内的胆固醇及胆汁酸等代谢而与胆囊胆固醇结石的形成密切相关，TC、TG的测定及TG/TC比值可以作为间接反映胆囊胆固醇结石成核的参考指标。

On closer examination, though, this is not a vote for multilateralism but just the opposite.

仔细审视后我们发现，这并非是对多边主义投出的赞成票，而是恰好相反。

Uncovering their weak spots, so I can defeat them.

揭露出他们的弱点，这样我就可以打败他们了。