查询词典 cell cleavage

Due to the complexity of the cell jitter, the NonSynchronous Tining Recovery methods are currently not mature With the emphasis being given to the Class A CBR traffic, this paper analyzes the performance of the queueing delay and cell jitter at the source node and intermediate nodes, and discusses the Source Timing Recovery at the destination node in ATM networks Firstly, this paper presents a description of the cell jitter of CBR traffic, and gives the definitions of two kinds of cell jitter regarding the Source Timing Recovery for CBR traffic Then, by using exact mathematical models and analysis methods, this paper analyzes the impact of the factors, such as the capacity of the queueing buffer, the randomness, the deterministic nature and the correlation in cell arrivals of the background traffic sources, on the queueing delay and cell jitter performance of the CBR traffic through Statistical Multiplexitng To obtain an insight into the power spectral distribution and look for better schemes for the depression and filtering of the cell jitter, within the analyses we succeed deriving the power spectrum of the cell jitter for CBR traffic Hence, not only the power spectral distribution of the cell jitter can in the frequency domain be qualitatively understood, but also can the rms (root-meansquare) value of the cell jitter be quantitatively obtained so as to more accurately measure the amplitude of the jitter In the end-to-end performance analysis of the queueing delay and cell jitter, we propose a kind of quasi-periodic cell stream model to characterize the jittered CBR traffic, and present an initial queueing analysis of the CBR traffic following such a model at a generic intermediate node Additionally, we briefly discuss the buildout/playout and Source Timing Recovery functions of the destination node Finally, regarding the Source Timing Recovery of CBR traffic, this paper systematically discusses several important principles of the cell jitter filtering and depression reported in the literature, introduces several implementation schemes of the Source Timing Recovery e.

由于信元抖动的复杂性，非同步定时恢复方法目前还很不成熟。本文针对A类CBR业务流在ATM网络源节点和中间节点的排队时延和信元抖动性能，以及在目的节点的源定时恢复问题作了较为全面的研究。首先，文中描述了CBR业务流的信元抖动，并具体地给出了两种与CBR业务源定时恢复有关的信元抖动的定义。然后，采用了精确的数学模型和分析方法，有针对性地分析了业务背景中信元到达的纯随机性、确定性和相关性以及排队缓存器容量等因素对CBR业务流经过统计复用后的排队时延和信元抖动性能的影响。为了了解信元抖动的功率频谱分布和寻求更好的抑制和滤除抖动的方法，在性能分析中，我们成功地完成了CBR业务流信元抖动的功率谱分析，使得不但可以从频域定性地认识信元抖动的能量分布特性，而且还可以定量地求出信元抖动的均方根值(rms：root-mean-square)，以更为准确地衡量抖动的大小。在CBR业务流的多节点端-端排队时延和信元抖动性能分析中，我们提出了一种准周期性(quasi-periodic)信元流模型来描述感染了信元抖动的CBR业务流，并基于这一模型进行了CBR业务流中间节点的初步排队分析。

Three are three major actions that Mn effects fracture behavior in the alloy at room temperature, as follow: One is that Mn fines grains, antiphase domains and weakens dislocation clog, which retain formation of cleavage crackle. The second is that Mn decreases APB energy and promotes slop and cross slip, in particular, a large number of cross slip make deformed APBs transform from line type into zigzag type and the APBs interact and cross each other, resulting in propagation of cleavage cracks. The last is that Mn raises the relative proportion of cleavage strength to grain-boundary strength and then restrains formation and propagation of cleavage crack.

Mn对合金室温断裂行为的影响主要反映在三个方面，一是Mn的存在细化晶粒和反相畴，使得位错塞积程度减小，从而阻碍解理裂纹源的生成；二是Mn的存在提高解理强度与晶界强度的相对比值，从而抑制了解理裂纹的形成和扩展；三是Mn降低反相畴界能量、促进超位错的滑移、交滑移，特别是断裂前位错大量的交滑移使形变反相畴由直线型变为折线"Z"字型，反相畴界间的相互作用、相互交错阻碍了解理裂纹的扩展。

In our system, we were unable to detect the cleavage product of overexpressed slit2. Nevertheless, it is interesting to investigate whether the cleavage of slit2 is important for its responsible function in CL1-5 or not. Our result showed that a non-cleavable slit2 showed similar growth inhibitory ability as the cleavable slit2. Therefore, the growth inhibitory ability of slit2 does not require cleavage at the reported cleavage site.

结果显示，在CL1-5细胞中，我们没有侦测到Slit2被切割的情形，并且当不能被切割的Slit2蛋白表现在CL1-5时，其抑制生长的能力与可被切割的Slit2相同，因此这个报导中的切割点与Slit2抑制生长的功能无关，而单独过度表现Slit2-C terminal蛋白片段就具有抑制细胞生长的能力。

1、Cur inhibits K562 cells growth and induces cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as p-Erk1/2、c-myc which are relevant with cell growth and apoptosis; 2、Cur synergizes STI571 to inhibit K562 cell growth and induce cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as Hsp90、PKC which are relevant with cell growth and apoptosis; 3、Cur reverses the resistance of K562/G01 cells to STI571, and synergizes STI571 to inhibit K562/G01 cell growth and induce cell apoptosis; 4、Cur inhibits human originated CML CD34~+ cell growth、induces cell apoptosis, and enhances STI571 to down-regulate the expression of p210~, finally inhibit cell growth and induce cell apoptosis.

从以上实验结果我们得出如下结论： 1、Cur抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游p-Erk1/2、c-myc等信号分子有关； 2、Cur协同STI571抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制Hsp90和下游PKC等信号分子有关； 3、Cur可逆转K562/G01细胞对STI571的耐药性，并与STI571协同抑制K562/G01细胞增殖和诱导凋亡，其抑制K562/G01细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游Procaspase-3和NF-κB等信号分子有关； 4、Cur可抑制来源于CML患者骨髓的CD34~+细胞的增殖并诱导其凋亡，还可协同STI571下调CML CD34~+细胞p210~表达，进而协同抑制细胞增殖、诱导细胞凋亡。

Higher Ca distributed in bulliformcell than in mesophyll cell and bundle sheath cell of dune reed, higher Mg distributedin mesophyll cell and higher K, Na and Cl distributed in sheath cell HigherNa and Mg distributed in mesophyll than in bulliform cell and bundle sheathcell of light salt meadow reed, and higher K, Ca and Cl distributed in itsbundle sheath cell. Higher Na and Mg distributed in bulliform cell than in mesophyllcell and bundle sheath cell of heavy salt meadow reed, higher K, Ca and Cl distributedin its mesophyll cell. This paper discussed the distribution conditions of theabove five ions in leaf cell of the four reed ecotypes and the meaning ofphysiological adaptation to habitat in detail.

沙丘芦苇的泡状细胞内Ca分布较叶肉细胞和鞘细胞高，叶细胞内Mg分布较高，在鞘细胞内K，Na和Cl布较高；轻度盐化草甸芦苇叶肉细胞内分布了较多的Na和Mg，在鞘细胞内K，Ca和C1分布较叶肉细胞和泡状细胞高；而重度盐化草甸芦苇泡状细胞内分布了较多的Na和Mg，叶肉细胞分布了较多的K，Ca和Cl；详细讨论了以上五种离子在不同生态型芦苇叶片内不同细胞类型的分布状况与环境适应的意义。

In the gravid uterus, The CKs immunolabelling were detected in glandular cell, luminal epithelial cell, traphoblast cell, endoblastic cell and allantoic cell; Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus but in endoblastic cell and some luminal epithelial cell.

对分离得到的子宫内膜基质细胞和子宫内膜腺上皮细胞进行免疫组织化学标记的结果显示在体外子宫内膜基质细胞表达泛角蛋白，子宫内膜腺上皮细胞表达波形蛋白，并且这一特性不因为传代而发生丢失。

2B8a was weakly reactive to neutrophils (23.72%) and negative for T cells, NK, DC, RBC and Plt. The antibody reacted to all 3 marrow CD34+ cells with an average positive rate of 39.33% while it was negative for G-CSF-mobilized CD34+ peripheral blood stem/progenitor cells (PBSC, 1.25%). Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明： 2B8a抗原在外周血B细胞上表达（3/3例，平均阳性细胞数为26.29 ％），而在T淋巴细胞和NK细胞上不表达（0/3例）；在粒细胞和单核细胞上阳性表达均为2/3例，平均阳性细胞数分别是23.72 ％和59.84 ％；在DC细胞、红细胞和血小板上均不表达（0/3例）。2B8a抗原在骨髓CD34＋细胞上的阳性表达是3/3例，平均阳性细胞数39.33 ％，而在G-CSF动员的外周血CD34+细胞上的阳性表达仅1/3例，平均阳性细胞数为1.25 ％。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 ％、98.61 ％、94.93 ％和5.68 ％；在T系细胞系Molt-3上的平均阳性细胞数为31.40 ％，而在Molt-4、JM和CCRF-CEM 细胞上不表达；在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 ％、33.40 ％、29.70 ％、28.19 ％、16.23 ％和8.02 ％；在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达，而在羊膜细胞系FL细胞上呈一定的阳性表达，平均阳性细胞数为45.03％。

Cell line analysis showed that the antibody notably reacted to three out of 4 cell lines (Raji, SMS-SB, Nalm-6 and Nall-1) with the positive rates of 98.78%, 98.61%, 94.93% respectively and weakly to one of them with 5.68% in B lineage cell lines and monoblastic cell line (U937, 67.78%) while it was only weakly positive or negative for other myeloid leukemia cell lines including Meg01 (33.40%), HL-60 (29.70%),K562 (28.19%), KG1a (16.23 %) and HEL92.1.7 (8.02%). Among 4 T lineage leukemia,5 neuroblastoma and 1 colon cancer cell lines tested, only Molt-3 was found weakly positive (31.40%) for 2B8a, while the remaining 3 T cell lines (Molt4, JM and CCRF-CEM), 5 neuroblastoma cell lines (LA-N1, KCNR, BE, SK-N-SH, SK-N-AS) and the colon cancer cell line (HR8348) tested were negative.

结果表明： 2B8a抗原在外周血B细胞上表达（3/3例，平均阳性细胞数为26.29 ％），而在T淋巴细胞和NK细胞上不表达（0/3例）；在粒细胞和单核细胞上阳性表达均为2/3例，平均阳性细胞数分别是23.72 ％和59.84 ％；在DC细胞、红细胞和血小板上均不表达（0/3例）。2B8a抗原在骨髓CD34＋细胞上的阳性表达是3/3例，平均阳性细胞数39.33 ％，而在G-CSF动员的外周血CD34 细胞上的阳性表达仅1/3例，平均阳性细胞数为1.25 ％。2B8a抗原在B系细胞系Raji、SMS-SB、Nalm-6和Nall-1上的平均阳性细胞数分别为98.78 ％、98.61 ％、94.93 ％和5.68 ％；在T系细胞系Molt-3上的平均阳性细胞数为31.40 ％，而在Molt-4、JM和CCRF-CEM 细胞上不表达；在髓系细胞系U937、Meg-01、HL-60、K562、KG1a和HEL92.1.7上的平均阳性细胞数分别为67.78 ％、33.40 ％、29.70 ％、28.19 ％、16.23 ％和8.02 ％；在神经母细胞瘤细胞系SK-N-SH、KCNR、BE、LAN-1和SK-N-AS细胞以及结肠癌细胞系HR8348细胞上均不表达，而在羊膜细胞系FL细胞上呈一定的阳性表达，平均阳性细胞数为45.03％。

A high Ca zone was first observed in the animal pole right before the first cleavage, then it extended along the cleavage furrow and the Ca(superscript 2+) signal remained high in this region throughout the first cleavage.

在第一次卵裂过程中，斑马鱼胚胎的动物极顶端首先出现高钙斑，然后在分裂沟部位出现高浓度的钙信号，这一信号在卵裂过程中持续存在。

A high Ca2+ zone was first observed in the animal pole right before the first cleavage, then it extended along the cleavage furrow and the Ca2+ signal remained high in this region throughout the first cleavage.

在第一次卵裂过程中，斑马鱼胚胎的动物极顶端首先出现高钙斑，然后在分裂沟部位出现高浓度的钙信号，这一信号在卵裂过程中持续存在。

I was just taking her hand, to help her out of a car, and I knew it.

我只是握着她的手，扶她下车，但我知道这一切。

China and South Korea have excoriated Japan over its approval of new school books which they say whitewash the atrocities committed during Japanese occupation, and anti-Japanese protests have been held in Chinese cities. The lingering bitterness over Japan's past imperialism still threatens to mar relations between the big East Asian powers

中国和韩国强烈谴责日本通过新的中学历史教科书，他们认为这一教科书掩盖了日本在其占领期间所犯下的残酷暴行，反日抗议活动在中国数座城市发生，历史上日本帝国主义制造的痛楚无法被忘却，这有可能破坏东亚强国间的关系。