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Results The specificity and accuracy were high,when pannus enhancement and morning stiffness or pannus enhancement and RF positive existed simultaneity.

结果敏感性最高的MRI指标血管强化与晨僵或RF阳性同时存在时,特异性及准确性均较高。

Third: for understanding the effect of c-myc AS ODN on pannus formation in synovium, we investigated the effect of c-myc AS ODN on endothelial cell line—ECV304 cells proliferation.

了解c-myc AS ODN是否对滑膜中的血管形成具有抑制作用,观察c-myc AS ODN对血管内皮细胞系ECV304细胞增殖的影响。

Objective: RA is an autoimmune chronic systemic disease, characterized by inflammation, self-immune and the hyperplastic synovial pannus.

目的:类风湿关节炎(rheumatoid arthritis,RA)是一种慢性系统性疾病,其主要病理改变是滑膜炎和血管形成及其相关的血清组织中多种细胞因子、抗原抗体和粘附分子等的变化,而细胞因子是RA炎症和损伤的重要介质,对RA患者外周血及滑液中细胞因子改变已有大量研究,已证明多种细胞因子参与RA的病变过程。

Pannus is a form of superficial keratitis, or inflammation of the cornea, found most commonly in German Shepherd Dogs, Greyhounds, and Siberian Huskies.

角膜血管是一种浅表性角膜炎,常见于德国牧羊犬、意大利灵缇犬和哈士奇。

Results positive findings of MRI in shoulder joint as follow, synovitis and enhanced bilateral pannus (93.5%), bone erosion (77.9%), cartilage erosion (90.3%),change of synovium and bone marrow was taken as diagnostic standard, the sensitivity, specificity, accuracy, 69.0%、76.7%、72.3%.

分析病变早期E-MRI特征。结果肩RA MRI阳性表现,滑膜增生血管形成(93.5%),骨侵蚀(77.9%),软骨侵蚀(90.3%)等,以滑膜和骨髓改变诊断肩RA,其敏感性、特异性、正确率分别为: 69.0%、76.7%、72.3%。

Brane, such as synoviocytes, macrophages, osteoclasts and TNF-αvv acts on different kinds of cell in synovial memchondrocytes, which can produce metalloproteinase,collagenase, stromelysin and so on, further induce pannus formation, joint inflammation, bone erosion and cartilage degradation.

TNF-α可作用于RA关节组织中的滑膜细胞、巨噬细胞、软骨细胞和破骨细胞,使这些细胞活化,产生金属蛋白酶、胶原酶、基膜溶解酶等,导致局部炎症反应和血管形成,进一步引起软骨破坏和骨侵蚀。

Morning stiffness, symmetric arthritis, RF positivity, ESR, CRP and synovial hyperplasia, enhanced pannus, bone marrow edema and bone erosion have certain differentiating diagnosis value between early RA and non-RA.

以晨僵、对称性关节肿胀、ESR增快、RF阳性、CRP值升高、血管强化、滑膜增厚、骨侵蚀、骨髓水肿为指标时对于鉴别早期RA与非RA具有一定诊断价值。

Therefore, both of the photooxidative DNA damage and the genetic polymorphism of the repair enzyme hOGG1 play some role in the development of pterygium.

因此,日照所引起DNA的光氧化性损伤及患者本身修复DNA的能力是否完备,都可能在眼的致病机转中扮演重要的角色。

We evaluated the photooxidative DNA damage, indicated by the specific content of 8-hydroxy-2'- deoxygaunosine (8-OHdG), in human pterygium. In addition, we analyzed the relationship between the genetic polymorphism of the DNA repair enzyme hOGG1 and the pterygium risk in a Chinese population.

首先,我们尝试以8-氢氧-2'-去氧鸟粪核糖苷( 8-hydroxy-2'- deoxyguanosine, 8-OHdG )为指标,找出眼组织中去氧核糖核酸( deoxyribonucleic acid, DNA )所受到的光氧化性损伤之证据。

Recorded on the unreeling film inside her; a long unwinding silk cocoon.

在她取出了的眼上作记号;长长的解开的蚕茧。

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