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心脏舒张期

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During diastole, the atria and ventricles of your heart relax and begin to fill with blood.

心脏舒张期,心房和心室放松,血液开始流入。

Intensity of radio signal also had this variation between in diastole and in systole .

离体心脏的心肌射频信号强度收缩期和舒张期之间也存在这种差异。

The last stage of diastole in the heart, occurring just before contraction and during which little additional blood enters the ventricle.

心舒张期心脏的各腔正常有节律的发生膨胀和扩张,在此期间为血液所充满

Primary atrial tumor has obvious characteristic on RT-3DE: All the atrial tumor display the same size of stereoimages as the tumor themselves. There is a clear boundary between the stereoimage of the myxoma and the atrial wall and there exists a short pedicle connecting the atrial myxoma to the atrial wall or the interatrial septum. The body of the atrial myxoma moves to and fro through the atrium and the atrioventricular valve with the contraction and relaxation of the heart with a fixative pedicle. Atrial myxoma leads to relative atrioventricular valve incompetence during systolic phase and relative atrioventricular valve stenosis during diastolic phase, but doesn't result in pathological changes of atrioventricular valve which restore to normal after exsection of the atrial myxoma. Malignant atrial tumor closely and extensively contact with atrial wall without obvious boundary, pedicle and movement with the contraction and relaxation of the heart.

所有心房肿瘤RT-3DE均实时显示与肿瘤大小相同的立体形态图像;心房黏液瘤立体形态图像与心房壁界限明显,都通过一短蒂与房间隔或心房壁相连,瘤体以蒂为固定点随心脏收缩、舒张在心房与房室瓣口之间往返运动;心房黏液瘤收缩期瘤体导致房室瓣相对关闭不全,舒张期瘤体导致房室瓣相对狭窄,但瘤体不引起房室瓣器质性病变,瘤体摘除后房室瓣功能即恢复正常;恶性心房肺瘤立体形态图像与心房壁关系密切,界限不明显,接触范围广泛,无蒂,瘤体不随心脏收缩、舒张运动。

The human heart is the center of the cardiovascular system. Integrated with mechanical, electrical, neural and biochemical properties, the heart is difficult to model, and there is no ideal integral model now. The developed electrical models haven't considered the mechanical properties and blood flow process in the heart; while the mechanical ones have normally deal with the heart in the diastole period, and the electrical activity was simplified too much although some mechanical models have investigated the heart during systole.

心脏是循环系统的核心,它集机、电、神经和生化控制于一体,建模难度很大,目前尚无较理想的统一模型;心脏电生理模型未与心脏的机械动力过程和血液流动过程联系起来;而心脏力学模型大都只研究舒张期的力学特性,即使研究收缩期心脏的力学问题,也往往对心电兴奋做了许多简化假设。

Methods:to analyze electrocardiogram,echocardiogram,left ventriculography,nuclear magnetic resonance results of 13 cases with ahcm.results:12 cases had apical wall thickening in echocardiogram, 3 cases had a "spade-like" configuration on left ventriculogram, 7cases were misdiagnosed as coronary heart disease initially.conclusion:ahcm is easy to be misdiagnosed as coronary heart disease, echocardiogram plays important role in the diagnosis of ahcm, in patients with suspicion of ahcm and inadequate echo images, the use of contrast echocardiography or mri should be considered.

对13例ahcm的心电图、超声心动图、左室造影及磁共振结果进行分析。结果:13例ahcm中心电图有特征性改变12例,超声心动图提示心尖区增厚12例,左室造影提示舒张期呈"黑桃"形改变3例,初诊误诊为冠心病7例。结论:ahcm易被误诊为冠心病,超声心动图是诊断该病的重要手段,必要时可做心脏超声造影术或磁共振检查。

Lactated Ringer's or 7.5% NaCl+6% dextran 70 was given for resuscitation 6h postburn.The volumes and rates of fluid infusion were controlled basically on the urinary output of 1.0ml*kg-1*h-1 and cardiac output of 70%~80% of preburn values.The volume load,+dp/dtmax,-dp/dtmax,CI,DO2 and VO2 were obtained to evaluate the effect of HSD resuscitation. Results The resuscitated volume of HSD was 30.56% less during first 24h postburn and 59.50% less at 4h after resuscitation than LR's.

采用犬35%TBSA Ⅲ度烧伤模型,伤后6 h分别用乳酸林格液及HSD进行复苏,并以每h尿量为1.0ml/kg及心输出量为伤前值的70%~80%来调整输液速度及输液量,观察HSD在复苏中容量负荷、左心室等容收缩期最大压力变化速率及左心室舒张期压力下降最大变化速率、心脏指数、氧供给(DO2)及氧消耗(VO2)等的变化。

The volumes and rates of fluid infusion were controlled basically on the urinary output of 1.0ml*kg-1*h-1 and cardiac output of 70%~80% of preburn values.The volume load,+dp/dtmax,-dp/dtmax,CI,DO2 and VO2 were obtained to evaluate the effect of HSD resuscitation. Results The resuscitated volume of HSD was 30.56% less during first 24h postburn and 59.50% less at 4h after resuscitation than LR's.

采用犬35%TBSA Ⅲ度烧伤模型,伤后6 h分别用乳酸林格液及HSD进行复苏,并以每h尿量为1.0ml/kg及心输出量为伤前值的70%~80%来调整输液速度及输液量,观察HSD在复苏中容量负荷、左心室等容收缩期最大压力变化速率及左心室舒张期压力下降最大变化速率、心脏指数、氧供给(DO2)及氧消耗(VO2)等的变化。

Of preburn values.The volume load,+dp/dtmax,-dp/dtmax,CI,DO2 and VO2 were obtained to evaluate the effect of HSD resuscitation.Results The resuscitated volume of HSD was 30.56%less during first 24h postburn and 59.50%less at 4h after resuscitation than LRs.

80%来调整输液速度及输液量,观察HSD在复苏中容量负荷、左心室等容收缩期最大压力变化速率及左心室舒张期压力下降最大变化速率、心脏指数、氧供给(DO2)及氧消耗(VO2)等的变化。

Methods:to analyze electrocardiogram,echocardiogram,left ventriculography,nuclear magnetic resonance results of 13 cases with ahcm.results:12 cases had apical wall thickening in echocardiogram, 3 cases had a "spade-like" configuration on left ventriculogram, 7cases were misdiagnosed as coronary heart disease initially.conclusion:ahcm is easy to be misdiagnosed as coronary heart disease, echocardiogram plays important role in the diagnosis of ahcm, in patients with suspicion of ahcm and inadequate echo images, the use of contrast echocardiography or mri should be considered.

对13例ahcm的心电图、超声心动图、左室造影及磁共振结果进行分析。结果:13例ahcm中心电图有特征性改变12例,超声心动图提示心尖区增厚12例,左室造影提示舒张期呈&黑桃&形改变3例,初诊误诊为冠心病7例。结论:ahcm易被误诊为冠心病,超声心动图是诊断该病的重要手段,必要时可做心脏超声造影术或磁共振检查。

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