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In this experiment, we induced experimental anisometropia and analyzed the effect of blur and aniseikonia on stereopsis.

阐明屈光参差影响立体视的机制,为临床矫正屈光参差及治疗屈光参差性弱视提供依据。

Objective To investigate the difference between the actual figure and theoretic figure of aniseikonia in anisometropic amblyopia patients; examine the stereoacuity of anisometropic amblyopia patients and ametropic amblyopia patients, and analysis the related factors, to give guideline for the treatment of anisometropic amblyopia patients.

目的 研究屈光参差性弱视患者的不等像值与理论值(0.25D的屈光参差引起0.5%的不等像)之间的差异及其规律;检查屈光参差性弱视及屈光不正性弱视患者的立体视锐度,分析其影响因素,为矫正屈光参差、治疗屈光参差性弱视提供依据。

There is no significant difference between the effect of experimental anisometropia on stereopsis and experimental anisometropia in different diopter when aniseikonia has been removed. It shows that there are no improvements after removing aniseikonia.

实验性屈光参差和去除不等像视影响的视网膜像模糊在各参差度比较均无显著差异,说明去除不等像视因素的影响并没有提高立体视锐度,可见实验性屈光参差对立体视影响的主要因素是由于屈光参差导致的视网膜像模糊。

Compared with previous reports of similarly aged Singapore Chinese adults, the prevalence of myopia, astigmatism, and anisometropia was lower, whereas the prevalence of hyperopia was similar.

与前期报道的相似年龄的新加坡中国成年人相比,马来人的近视、散光和屈光参差的患病率要低,但远视的患病率相似。

Methods We studied a series of 31 eyes of 24 patients who underwent PRK for the correction of anisometropia after cataract surgery.

目的观察准分子激光屈光性角膜切削术矫治白内障摘除术后屈光参差的疗效。

ABSTRACT Objective The purpose of this paper is to investigate the ef fects of ametropia and anisometropia on visual development. Methods Animal models were made by binocular and monocular chronic atropinization.

用幼猫双眼或单眼慢性阿托品化的方法,造成屈光不正或屈光参差的弱视动物模型,在整个视觉发育期,连续检测其条栅视力和对比敏感度函数,并与正常幼猫相比较。

Methods Fourty subjects, 20 anisometrope and 20 emmetrope, the AMP accommodation lag and NRA/PRA were measured after the distance refractive correction.

选取40位受试者,正视者右眼20只,屈光参差者20位,其中屈光参差者高度数眼20只,屈光参差者低度数眼20只,在屈光完全矫正的基础上用主观的测量方法测量其调节幅度、正负相对调节和调节滞后量。

The accommodation lag of emmetrope was (0.2375±0.2218) D, The accommodation lag of high myope eye of anisometrope was 1.2875±0.4389 D, The accommodation lag of low myope eye of anisometrope was 0.2250±

正视眼的主观调节滞后量为(0.2375±0.2218)D,屈光参差的高度数眼滞后量为(l.2875±0.4389)D,而屈光参差的低度数眼的调节滞后量为(0.2250±0.2913)D。

The accommodative function (accommodative lag and amplitude) of the dominant and non-dominant eye has significant differences in the adolescent hyperopic anisometrope.

儿童和青少年远视性屈光参差的主导眼和非主导眼的调节幅度和调节滞后不同;其主导眼的调节功能与同龄儿童无明显差别,非主导眼的调节功能存在缺陷。

Methods Animal models were made by binocular and monocular chronic atropinization.

用幼猫双眼或单眼慢性阿托品化的方法,造成屈光不正或屈光参差的弱视动物模型,在整个视觉发育期,连续检测其条栅视力和对比敏感度函数,并与正常幼猫相比较。

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