尿原的

We coupled the IgG of monoclonal antibody N34 specific to UK and the Fab of monoclonal antibody SZ-51 specific to the membrane protein GMP-140 of activated platelet. The bispecific antibody (N34 SZ-51) is able to bind UK and plasma-clot simutaneously. It enhanced plasma clot lysis 5 fold by HUK whereas it enhanced plasma clot lysis 38 times by LUK. UK in association with the bispecific antibody didn't decrease the concentration of fibrinogen in the assay supernatants.

利用抗体介导使纤溶酶原激活剂定向到血栓表面，可大大提高其纤溶作用，我们用双功能团试剂将抗尿激酶的单克隆抗体N34的IgG和抗活化血小板膜蛋白GMP-l40的单抗SZ-5l的Fab片段通过二硫键共价偶联，合成的双专一性抗体（N34-SZ-51）既具有血栓亲和性也具有尿激酶亲和性，从而增加了尿激酶对血栓的专一性，使高分子量尿激酶溶栓率提高了5倍，使低分子量尿激酶溶栓率提高了38倍之多，且不影响血浆中纤维蛋白原的含量。

Carboxypeptidase inhibitor from potato , a special inhibitor of TAFIa, was used to test effect of heparin and hirudin on activation of TAFI.

对尿激酶诱导的纤溶，水蛭素比肝素有更好的间接促纤溶作用，其原因主要是水蛭素能有效抑制再次血凝的发生，减少了尿激酶和纤溶酶原的短暂性耗竭。

Three methods and 3 calculating formula were used to choose for concrete operation according to the idea at urine iodine assayed with cerous sulfate reducing reaction by which WHO and ICCIDD draw up the preventive and curative criteria of iodine deficiency.

本文根据&WHO与ICCIDD制定的碘缺乏病防治标准与方法&中硫酸铈还原反应测定尿碘的意图，采用3种方法和3种计算公式对具体操作进行选择，并对原标准对照计算法的不合理以及计算误差对评价尿碘水平的效能进行了探讨。结果表明：（1）方法Ⅰ存在仪器误差，准确度不佳。方法Ⅱ线性关系和准确度较理想，但操作不便计算麻烦。方法Ⅲ线性关系和准确度与方法Ⅱ无差异，操作和计算简便、节约试剂。

Diabetes insipidus is a clinical syndrome characterized by excretion of large volumes of dilute urine due to deficiency and reduction of AVP secretion (central diabetes insipidus, CDI) or renal insensitive to AVP (nephrogenic diabetes insipidus, NDI).

尿崩症是由于抗利尿激素的产生不足或作用障碍使肾脏不能将原尿浓缩而排出大量低渗尿的一组临床综合征。

Results (1) the laminin synthesis was significantly improved by PBMC from nontreatment group (P＜0.05);(2) the 3 H-prolin incorporation was significantl decrease in the GEC treated by PBMC supernatant from nontreatment group(P＜0.05);(3) the supernatant of PBMC from nontreatment group stimulated the collagen Ⅲ synthesis(P＜0.05), while, there was no significant difference of collagen Ⅳ synthesis between 4 groups;(4) the activity of PBMC supernatant have no close relation with urinary protein excretion, pathological type and clinical type.

结果 (1)肾病极期未经激素治疗组PBMC上清明显促进了GEC合成层粘连蛋白；(2)未治PBMC上清抑制了GEC合成总胶原；(3)未治组PBMC上清促进了Ⅲ型前胶原的合成，而对Ⅳ型胶原的合成无明显影响；(4)肾病患儿PBMC的上述作用与是否足量激素治疗有关，而与尿蛋白能否阴转、肾组织病理类型、肾病临床类型等无直线相关关系。

Copeptin, the C-terminal part of the vasopressin prohormone, is secreted stoichiometrically with vasopressin.

Copeptin作为抗利尿激素原的C末端部分，是抗利尿激素系统定量分泌的。

Copeptin, the C-terminal part of the asopressin prohormone, is secreted stoichiometrically with asopressin.

Copeptin作为抗利尿激素原的C末端部分，是抗利尿激素系统定量分泌的。

The present invention also provides the affinity medium and the method for decontaminating urine prokinase and/or urokinase.

本发明还提供了新的用于纯化尿激酶原和／或尿激酶的亲和介质和方法。

Breast cancer commonly metastasizes to bones, producing both osteolytic and osteoblastic deposits. Different markers for quantitative determination of bone turnover have been developed to evaluate bone metastases of breast cancer. The urinary deoxypyridionline, a crosslink product of collagen molecules found in bone and excreted in urine during bone degradation, and bone specific alkaline phosphatase, an isoenzyme localized in the membrane of osteoblasts and released in circulation during bone formation, were recently described as a group of markers of bone turnover in metastatic cancer.

乳癌患者常见骨髓转移造成溶骨性及成骨性沈积物，而利用不同标记来定量骨质转换形态，可以评估乳癌骨骼转移的变化，而尿液中Deoxypyridinoline是骨质内胶原分子在骨骼破坏分解时至尿中的产物，骨特殊碱性磷酸酵素是一种同形酵素，位於成骨细胞的外膜且在骨骼形成时释放至血液中，利用此两种生化标记，可以来预测乳癌转移至骨骼的转换情形。

Prourokinase is not only a single-chain precursor of urokinase , but also one of the two human plasminogen activators.

尿激酶原是人机体内仅有的两种内源性纤溶酶原激活剂之一，为双链尿激酶的酶原前体。

What are your goals and strategies for growth?

你的成长目标和策略是什么？

And unto the angel of the church in Sardis write; These things saith he that hath the seven irits of God, and the seven star I know thy works, that thou hast a name that thou livest, and art dead.

3：1 你要写信给撒狄教会的使者，说，那有神的七灵和七星的，说，我知道你的行为，按名你是活的，其实是死的。