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speech defect相关的网络例句

查询词典 speech defect

与 speech defect 相关的网络例句 [注:此内容来源于网络,仅供参考]

Control group: inflammatory cell infiltration (1W); inflammatory cell disappearance, fibroblast and capillary tachyauxesis (2W); osteoblast appearance around bone defect (1M); small amount neoformative bone tissue around bone defect.

对照组:术后1周,骨缺损区周围有大量炎性细胞浸润;术后2周,炎性渗出吸收,成纤维细胞、毛细血管生长活跃;术后1个月,骨缺损区周围有成骨细胞;术后3个月,骨缺损边缘有少量新骨沉积。

Control group: inflammatory cell infiltration (1W); inflammatory cell disappearance, fibroblast and capillary tachyauxesis (2W); osteoblast appearance around bone defect (1M); small amount neoformative bone tissue around bone defect. Conclusion TGF-β1 can induce alveolar bone regeneration.

对照组:术后1周,骨缺损区周围有大量炎性细胞浸润;术后2周,炎性渗出吸收,成纤维细胞、毛细血管生长活跃;术后1个月,骨缺损区周围有成骨细胞;术后3个月,骨缺损边缘有少量新骨沉积。

Autogenous bone is an ideal material to repair bone defect, however, autogenous bone harvest increases patient trauma and pain. Moreover, its source is very limited, and it could not meet the requirement to repair large-segment transplantation. Metal prosthesis has problems such as sensitivity to defect, loosening, breakage, bad histocompatibility and nondegradation.

自体骨虽然是理想的骨缺损修复材料,但自体取骨增加了患者的创伤和痛苦,并且供骨的来源有限,难以满足大段骨移植的要求;金属假体对缺口敏感,存在松动、断裂、组织相容性差、不可降解等问题。

MRS2 encode a magnesium transporter protein in Saccharomyces cerevisiae and deletion of MRS2 gene results in decrease in mitochondrial magnesium concentration, Group II RNA splicing defect and growth defect on nonfermentable carbon source.

MRS2 基因编码线粒体镁离子转运蛋白,MRS2缺失会导致酵母线粒体镁离子浓度下降、线粒体内II型内含子剪接缺陷和非发酵碳源上的生长缺陷。

To obtain more information of magnesium homeostasis in mitochondria, mTn-lacZ/LEU2 transposon library was transformed into mrs2 deletion mutant to screen for suppressor genes of MRS2. YMR166C, a member of mitochondrial carrier family, was identified as a suppressor gene of MRS2. Deletion of YMR166C gene can rescue the defects of mrs2 deletion mutant such as the decrease in mitochondrial magnesium concentration, Group II RNA splicing defect and growth defect on nonfermentable carbon source. For the first time we demonstrated YMR166C is involved in mitochondrial magnesium homeostasis.

为了增进对线粒体镁离子代谢调控基因的了解,利用酿酒酵母mTn-lacZ/LEU2转座子文库筛选MRS2的抑制基因,发现线粒体载体家族成员YMR166C基因的缺失可以挽救MRS2基因缺失的突变体的生长缺陷、II型内含子剪接缺陷,并可以调节线粒体镁离子浓度,首次发现YMR166C是线粒体镁代谢相关基因。

All but 2 of 40 patients with perforated appendicitis had a clearly visualized focal defect in appendiceal wall enhancement, the researchers report, and only 2 of 62 patients with nonperforated appendicitis had false-positive findings for a focal appendiceal wall defect.

四十名阑尾穿孔患者中有两名其阑尾壁的局灶缺损可以被清晰增强显示,六十二名非阑尾穿孔患者中仅两名出现阑尾壁局灶缺损的假阳性发现。

Methods There were 9 cases of osteofibrous dysplasia.We erasioned proc tissue resulting in bone defect through operation,and filled the bone defect by nano-.hydroxyapatite compounding rhBMP-2 completely.We detected liver and kidney function,X-.ray examination to understand information of toxicity,immune reaction and bone reparation by post-.operation follow-.up.

选取9例骨纤维结构不良病例,手术刮除病变组织造成骨缺损,用纳米羟基磷灰石复合rhBMP-2完全填充骨缺损,术后随访分别做肝肾功能、免疫学指标检测和X线片检查,以了解材料对人体有无毒性、免疫反应和骨修复情况。

It is extremely necessary to find an ideal filling material for the treatment of benign bone defect, particularly those benign osteolytic defect in children with a comparatively large cavity.

为寻找良性骨缺损理想的充填材料,特别是修复儿童良性溶骨性较大的骨缺损,采用天然型无机骨充填良性溶骨性骨缺损5例,其中骨囊肿3例,纤维异样增殖症1例,非骨化性纤维瘤1例。

During the operation,a postauricular\|mastoid flap based on the edge of the helix defect was raised .As the frame,a piece of concha cartilage taken together with the flap or costal cartilage of 0.3~0.4 cm in width was fixed to the auricular defect.

应用陈氏对外伤性耳廓部分缺损修复术式,并改进设计以耳廓缺损前缘为蒂的耳后乳突区皮瓣,携带耳甲后软骨块,或以缺损缘一端为蒂,切取一条正常软骨宽0.3~0.4cm,固定支撑,皮瓣包卷覆盖塑形。

Total-energy calculations indicated that the pseudocubic and the defect cubic phases with their defect zinc-blende structures are energetically favorable relative to other Si3P4 phases.

总的能量计算表明,具有缺损闪锌矿结构的膺立方和缺损立方相相对于其它相能量最低,因而是最稳定的2个相。

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