查询词典 proliferation

TSPG has the similar effect as HGFs, can directly or/and indirected effect and regulate proliferation and differentiation of CD34〓 HSC/HPC to bring into full play in"invigorating qi"of Panax ginseng. 2. During the process of the extensive proliferation, orderly differentiation, functional activation and gene transfer of hematopoietic stem/progenitor cells in ex vivo culture systems, TSPG may be used to regulate the proliferation and differentiation of hematopoietic cells to perform potential application in the domain of hematpoietic stem cell engineering.

我们的研究结果提示：1、TSPG具有类似造血生长因子的功效，能直接或间接影响和调控造血干/祖细胞的增殖与分化，促进血细胞的生成，从而发挥人参&补气生血&之功效。2、在进行对造血干/祖细胞的扩增与诱导分化，功能激活，基因转染等造血干细胞工程研究中可利用人参皂甙的药理作用来调控细胞的增殖分化，发挥其在造血干细胞工程领域的潜在应用价值。

The results show that the main CO proliferation forms in coal bed are the Knudsen proliferation and the transition proliferation.

结果表明，CO在煤层中的扩散主要为Knudsen扩散及过渡型扩散方式，过渡孔的增加有利于其扩散，而微孔的增加则不利于其扩散。

Results The latent phase of the primary culture cells was 5-9 days, followed by 11-16 days of logarithmical proliferation; the plateau began at day 18-20. The subcultures showed 12-24 hours of latent phase, followed by 7-10 days of logarithmical proliferation, then entered the plateau (11-13 days). The hMSC expressed positive CD44 unanimously, but don′t expressed CD45. Low concentration of bFGF (10 μg·L-1) could stimulate the proliferation of hMSC significantly (P.05); while 25-100 μg·L-1 of bFGF could inhibit the ALP activity.

结果：原代培养细胞生长潜伏期5~9 d，对数增殖期11~16 d，生长平台期18~20 d；传代培养细胞生长潜伏期12~24 h，对数增殖期为7~10 d，11~13 d进入细胞生长平台期。hMSC均一表达CD44，不表达CD45.bFGF在较低浓度（10 μg·L-1）时即有显著的促进hMSC增殖的作用（P.05）， bFGF在25~100 μg·L-1对hMSC的ALP活性有较显著的抑制作用。

The effect of F 1C on joint swelling and the immune respones of AA rats were evaluated by lymphocytotic reaction, flow cytometry, and ELISA. Results F 1C by ig administration to AA rats decreased the primary joint swelling of AA rats, stronger than TW and weaker than CsA, promoted the decreased splenocyte proliferation induced by Con A, and inhibited the augmented thymocyte proliferation induced by Con A in AA rats. The evaluation in vitro showed the F 1C inhibited the augmented thymocyte proliferation from AA rats and promoted the deficient IgG produced by splenocyte from AA rats.

结果 ig给予F1C(30mg／kg)对AA大鼠原发性足肿胀具有明显的治疗作用，该作用较TW强，较CsA弱；F-1C对AA大鼠低下的脾细胞增殖反应具有改善作用；对亢进的胸腺细胞增殖反应具有抑制作用。F-1C体外对AA大鼠低下的脾细胞产生IgG水平具有明显的促进作用，并能抑制亢进的胸腺细胞增殖反应。

Results When the culture medium was supplemented with 0 .05μg/ml sodium selenite,the proliferation of transformed cells was significant ly promoted and the transformation did not change remarkably,when supplemented w ith 0.25μg/ml,the proliferation was promoted but the transformation was inh ibited,and when with 0.75μg/ml and 1.50μg/ml,the proliferation was not pro moted and the transformation was strongly inhibited.Big dose of (1.50μg/ml) sodium selenite m ade the nuclei of the untransformed lymphocytes gather together like a few grape s doing,and even caused them to fuse.

结果 在培养物中添加0.05μg/ml亚硒酸钠能显著促进转化细胞增殖；添加0.25μg/ml可促进转化细胞增殖，但显著抑制细胞转化；添加0.75μg/ml和1.50μg/ml，不仅不能促进细胞增殖，而且强烈抑制细胞转化。1.50μg/ml剂量的亚硒酸钠使非转化淋巴细胞发生葡萄状聚集甚至核融合。

The article focuses on the aberrant expression and significances of proto-oncogenes, tumor suppressor genes and genes associated with the proliferation process of gastric cells such as telomerase, transcription factor and cyclooxygenase, etc. These genes, alone or synergically, participate in gastric cancer cell proliferation, resulting in an uncontrolled growth characterized by over-proliferation of decreased apoptosis of gastric cancer cells.

本文着重阐述了与胃癌细胞增殖过程密切相关的端粒酶、核转录因子、环氧化酶等基因以及参与细胞周期调控的原癌基因、抑癌基因的异常表达及临床意义，这些基因或单独作用，或相互协同，参与胃癌细胞的增殖，使肿瘤细胞生长以增殖过多、凋亡过少的失控性生长为特征。

In this work, it was found that MT at the concentrations of 〓 and 〓 mol·〓 could enhance the proliferation in rats and mice thymic lympho- cytes and human PBL as well as enhancing the proliferation in AA rats, 11 months mice and Mor-dependent mice thymic lym- phocytes, and RA patients PBL, and what was more that MT at a lower concontrations had a marked up-regulation on the above decreased lymphocytes proliferation.

结果表明，依赖组小鼠痛阈下降，跳跃次数增加，肛温降低，伴有胸腺细胞增殖能力的低下，Mor依赖小鼠的脑和血浆甲硫氨酸脑啡肽亦明显降低。ig MT（30、90 mg·〓×7d）能明显提高依赖性小鼠的痛阈，减少跳跃次数，恢复其肛温，阻止Mor对淋巴细胞功能的抑制，并能维持脑、血浆Met-Enk接近正常对照水平。

Results The activation of NF-κB, proliferation response, and expression of IL-4 and I L-5 mRNA and protein in T lymphocytes stimulated by PMA were significantly hig her than those of their blank control (P.01), while those indexes of T l ymphocytes stimulated by PMA and PDTC simultaneously were significantly lower th an those stimulated by PMA alone (P.01). The apoptotic index of T lympho cytes stimulated with PMA were significantly lower than that of their blank cont rol (P.01), and the apoptotic index of asthmatic guinea pig T lymphocytes stimulated with PMA and PDTC simultaneously were significantly higher than that stimulated by PMA alone (P.01). The significant positive correlations w ere found between the activation of NF-κB and the proliferation (r=0.64, P.001), and the expression of IL-4 and IL-5 mRNA and protein of T lymph ocytes, respectively (r=0.55-0.68, P.001). There was also signific ant negative correlation between the activation of NF-κB and apoptosis of T l ymphocytes (r=0.62, P.001). Conclusions NF-κB may participate in the signal conduction of PKC regulated proliferation, apoptosis and expression of IL-4 and IL-5 of T lymphocytes in asthma.

结果加入PMA培养的哮喘组T淋巴细胞NF-κB的活化、细胞增殖反应、IL-4和IL-5的mRNA和蛋白质的表达均显著高于其空白对照(P.01)，而同时加入PMA和PDTC培养的哮喘组T 淋巴细胞以上指标均显著低于只加入PMA培养的哮喘组T淋巴细胞(P.01)；加入PMA 培养的哮喘组T淋巴细胞的凋亡指数显著低于其空白对照(P.01)，而同时加入PMA 和PDTC培养的哮喘组T淋巴细胞凋亡指数显著高于只加入PMA培养的哮喘组T淋巴细胞(P 。01)。T淋巴细胞NF-κB的活化与增殖反应呈显著正相关(r=0.64，P.00 1)，与IL-4和IL-5的mRNA和蛋白质的表达也均呈显著正相关(r=0.55-0.68，P 。001)，而与凋亡指数呈显著负相关(r=-0.62，P.001)。

Icariin, the main constituent of Epimedium sagittatum Maxim, and its metabolite icariside Ⅱ showed similar activity in cell proliferation. The total flavonoids from Viscum coloratum Nakai could stimulate cell proliferation and significantly promote cellular ALP activity. Daidzein and its derivatives exhibited no significant activity in cell proliferation.

结果发现：中药淫羊藿中的主要成分淫羊藿苷及其大鼠体内主要代谢物淫羊藿次苷Ⅱ具有类似的作用，可显著促进成骨样细胞的增殖；中药槲寄生总黄酮对成骨样细胞的增殖有一定的促进作用，并可显著提高细胞内ALP活性；大豆苷元及其衍生物对UMR106细胞的增殖作用不明显。

Results: 1 Tritiated thymidine incorporation assay showed that 1μM adenosine plus coformycin slightly inhibited CTLL-2 proliferation, while 5-20μM adenosine elicited marked inhibition on CTLL-2 proliferation responded to IL-2 a dose-dependent fashion. 2 Coformycin alone could also inhibit CTLL-2 proliferation at the concentration of 2.5μM. The underlying reason maybe due to the endogenous adenosine produced by CTLL-2 cells.

结果：11μM腺苷对CTLL-2增殖仅有轻微的抑制作用，而5、10、20μM腺苷的存在可显著抑制CTLL-2的增殖，并且呈剂量相关，P值均小于0.05.2CTLL-2经过2.5μM助间型霉素的单独处理也可引起轻度的增殖受抑现象，分析其可能原因是T细胞自身可以分泌腺苷，所以该抑制现象的出现与内源性腺苷有关。3本实验所用腺苷浓度对CTLL-2的活细胞百分率影响轻微。

This matter overthrew all basises I learned from Sunday school .

这件事把我从周日学校学到的所有道理都颠覆了。

It's one of the cruel ironies of aging: if you want to remember your favorite restaurants, the less you should eat at them.

这是一个老化的残酷讽刺：如果你想记住你最喜欢的餐馆，你应该更少吃到它们。