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injury相关的网络例句

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APHs, TF and GnH were tested in 93 patients with TBI,who were admitted to Zhejiang Provin-cial People's Hopital from March 2006 to June 2007. Patients with primary injury in the hypothalamic and pituitary regions, as detected by CT and/or MR/examination, as well as those with tumors or immune diseases in the CNS,endocrine or urinogenital systems, were excluded. The clinical data were analyzed according to Glasgow coma scores, type and degree of injury, and whether there was any secondary cerebral injury.

随机对浙江省人民医院2006年3月至2007年6月经系列头颅CT扫描和/或MRI检查,明确无下丘脑、垂体等部位的原发损伤,且既往无中枢神经系统、内分泌和泌尿生殖系统肿瘤及免疫性疾患史的93例门急诊和住院TBI患者的APHs、TF和GnH进行测定,并按患者的GCS评分、TBI类型和损伤程度以及有无继发性脑损害等因素进行分组研究,以同期20例健康体检者作为对照组。

Methods Both in vitro and in vivo models of ventilator-induced lung injury were used. Alveolar epithelial cells were stretched in vitro to mimic the lung injury in VILI. Rats were ventilated at large tidal volume to produce ventilator-induced lung injury in vivo. A total of 23 inflammatory cytokines were screened with micro gene array in stretched alveolar epithelial cells.

方法通过呼吸机诱导肺损伤的体内及体外模型,将分离的肺泡上皮细胞于体外拉长,利用基因微阵列技术分析并筛选出上调的炎性细胞因子,之后给予大鼠呼吸机机械通气,留取血清标本,测量这些细胞因子在血清中是否上调。

Compare the lung injury extent and the important physiological parameters to determine the optimal condition for rabbit ventilator induced lung injury, Choosing another 24 new Zealand rabbit, randomly divided into 4 groups:(1)conventional ventilation+PEEP group;(2)ventilator induced lung injury group: employ the experimental condition determined by the first part experiment;(3)VILI+HO-1 inducer hemin group:pre-treated with HO-1 inducer, the ventilation method is the same with VILI group;(4)VILI+HO-1 inhibitor Zn-PP group(Zn-PP group):pre-treated with HO-1 inhibitor, the ventilation condition is the same with VILI group.

第二部分24只家兔随机分为4组:(1)常规潮气量+PEEP组;(2)呼吸机所致肺损伤组:使用实验第一部分确定的最适条件;(3)呼吸机所致肺损伤+HO-1诱导剂干预组:预先给予氯铁血红素,通气条件同VILI组;(4)呼吸机所致肺损伤+HO-1阻断剂干预组:预先给予锌原卟啉,通气条件同VILI组。

Though the primary stimulator of 1H is not clear, it is related to physical, cell and body fluid IH is a chronic process involving multiple faction injury and blood flowing abnormal are revamped as the main reason, the proliferation level depend on the length and width of the injure of vessel large amount research illustrate injury cause many growth factor infolded in IH, for example: template derived growth factor, fibroblast growth factor, transmission growth factor , endothelin (ET-1) et al, some early responsive gene such as c-fos、c-jun c-myc also take part in SMC proliferation after vessel injury.

虽然IH的确切起始刺激因子并未阐明,但已知与物理、细胞和体液因子有关。IH是一个多因素参与的慢性过程,损伤和血液动力学异常一真被认为是IH的主要因素。内膜增殖的程度有赖于血管损伤的长度和深度。近年来大量的实验研究结果表明,损伤等刺激引起的众多的生长因子样物质在IH中起关键作用。如血小板源性生长因子、碱性纤维母细胞生长因子、转化生长因子β、内皮素-1(ET-1)等诸如C-fos、C-jun、C-myc等早期应答基因也参与了血管损伤后SMC的增殖。

For example, MI behaved most sensitive to heart state when spontaneous respiration inhibited with respiration machine; FD could manifest heart healthy level, which reduced sharply sooner after acute injury but remained almost steady afterwards; the parameters of Poincare dissert-dot plot and HRV could accurately distinguish the state change before and after ligaturing; the HF-ECG excelled in not only revealing injury degree but also locating injury position.

例如,交互信息对自主呼吸被接呼吸机抑制后的状态敏感,分维具有表征心脏健康状况潜力的特性,在急性损伤后明显减少,但不随损伤的时间延续而变化;心率变异性参数和散点图可以灵敏地区分出急性损伤前和损伤后的状态;而高频心电在表征心脏损伤和定位方面独具特色。

Animals in injury group were subjected to a m oderate lateral hydraulic percussion brain injury (0 .2 MPa),and then were subdi vided into 5,15,30,60,120,240,480 and 720 minutes groups according to the tim e elapsed after injury .

损伤组 动物均给以0 。2 MPa液压冲击脑损伤,按冲击后处死时间不同再分为5、15、30、60、120、 240、480和720分钟组。

Results:Four cases occured in 24h postoperatively,3 cases among them for injury of bile duct outside liver,the other one for the ductus cysticus stump's clip exfoliation.8 cases occured after LC in 48 hours,because of leakage of cholangiole,injury of aberrant duct or heat injury of common bile duct.

结果:12例胆漏中4例在术后24h内发觉,其中3例为肝外胆道损伤,1例为胆囊管残端钛夹零落;8例在术后48h以后发觉,其原因为胆囊床毛细胆管渗漏、迷走胆管损伤和胆分管热损伤。

Much progress has been made in recent years through studying septic shock,however brain injury is rarely involved.This study takes the models of endotoxin-shock rats as research object to simulate septic shock.The study aims are to explore anomalous signal transduction in brain injury caused by septic shock and the effect of Sini Decoction in the process;define the molecular and cellular mechanisms of reviving yang for resuscitation method used to prevent and treat brain injury in septic shock and establish the scientific foundation for modernizing traditional Chinese medicine.

研究目的:1。明确内毒素休克后脑损伤组织中炎症介质水平,中性粒细胞和脑血管内皮细胞等细胞表面黏附分子表达情况,脑血管平滑肌细胞、神经胶质细胞和神经细胞对细胞外炎症介质刺激产生的功能代谢变化以及结构改变等三者之间的规律性关系。2。

Media revealed that Bryant's injury is not a simple stamp injury to his right index finger joints appear avulsion fracture, injury, while the index finger ligaments Department also showed that there are fragments of bone, as Bryant insisted on re - debut game, the Lakers medical staff in the game space has only adopted a conservative treatment, using bandages will be fixed Bryant's finger.

赛后据美国媒体透露,科比的伤势并不是简单的戳伤,他的右手食指关节处出现撕脱骨折型损伤,而食指关节韧带处骨头也显示有碎片,由于科比执意要重新登场比赛,湖人队医在比赛间隙只是采取了一个保守性的治疗方法,用绑带将科比的手指进行固定。

Gelatin microsphere is firstly made by encapsulating bFGF into gelatin by special manufacture process, then this microsphere was combinated into PLGA conduit or PLGA film.when used, we need only pack the injury peripheral nerve, injury spinal cord, wounded pars encephalic with the PLGA conduit. The conduit or film can slowly release buff which would be absorbed by around tissues.bFGF was already proved to be nutritious to the regeneration and alive of neuron and protect injury neuron.bFGF can also promote morphogenesia and division of nerve cell which would raise the survival rate of neuron, guide the generation of axon etc.bFGF is one of the important factors of sustaining the normal survival of cholinergic nerve.

使用时,只需将此材料贴敷外周损伤神经伤患处,脊髓伤口,或在脑部开颅手术后填充于术后创面,可有效地在伤口局部缓慢地释放并由机体组织吸收bFGF等神经营养因子。bFGF等已被证明具有神经营养作用,可促进神经元的再生与存活,保护受损的神经元。bFGF等对神经细胞兼有促进形态发生与分裂作用,具有提高神经元的成活率、诱导轴突向外生长等功能,是维持前脑基底部胆碱能神经元正常生存的重要因素。

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