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dose rate相关的网络例句

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Illustrative results are presented for the radiation dose and dose profile with activity distributions: uniform, determined by steady-state diffusion into the target volume, or determined by steady-state diffusion with a constant rate of loss of activity from the target.

计算程序用Visual Basic V6.0编写,在微机上运行。为了验证计算结果,将均匀分布条件下的计算结果分别与文献报导的结果比较,发现它们是一致的。

The dose-response curve of histamine was shifted parallely to the right. pA2 was 9.34±0.61. The guinea-pigs were effectively reduced the rate of histamine induced death, decreased reaction extent of shock (P<0.01) and prolonged latency period of shock (P<0.05) by orally administered cetirizine ranged from 0.1, 0.2 and 0.4 mg.kg-1. The capillary permeability to intracutaneous injection of histamine was potently inhibited by orally administered cetirizine ranged from 0.0625 to 0.25 mg.kg-1 in mice, blue area obviously decreased (P<0.01), and dose-response curve existed obviously. In which the effect of these dosage groups were superior to the group of chlorphenamine.

结果:西替利嗪3×10-8~3×10-7mol.L-1可剂量依赖性地对抗组胺引起的肠肌收缩,使组胺的量效曲线平行右移,pA2为9.34 ± s 0.61,西替利嗪0.1,0.2及0.4 mg.kg-1口服给药时,能明显减轻豚鼠静脉注射组胺所致休克反应的严重程度(P<0.01),并可延长豚鼠惊厥反应的潜伏期及降低死亡率(P<0.05),西替利嗪0.0625~0.25 mg.kg-1口服给药可显著对抗组胺引起的小鼠皮肤血管通透性的增高,使蓝染面积显著缩小(P<0.01),且存在明显的量效关系,3个剂量组的作用均优于氯苯那敏组。

NGF had obvious cytotoxicity on these cell lines ,which shows to be dose-dependent, especially for MGC-803 and BEL-7404 ,their IC_(50) value at the 24~ hours are 49.9mg/L and 55.46mg/L ; Main features of apoptosis under fluorescent microscope include reduction in volume,nuclear chromatin condensation, intensification under jacinth fluorescence; The early apoptosis rate of pristine cells which detected by FCM increases with NGF dose increasing.

2NGF对4种人肿瘤细胞均有细胞毒性作用,且呈剂量依赖关系,尤其是对MGC-803和BEL-7404的抑制作用更强,作用24h的IC50值分别为49.9mg/L和55.46mg/L,荧光显微镜下可观察到MGC-803凋亡细胞的主要特点为:细胞体积缩小,染色质固缩,橘红色荧光增强;流式细胞仪检测到MGC-803细胞的早期凋亡率随NGF浓度的增高而增加,各浓度组与对照组的早期细胞凋亡率比较有明显差异(P<0.01)。

The typical apoptotic morphological features appeared in MUTZ-1 cells treated with 4mmol/L VPA for 72hours. Pyknosis of cells and nuclei, disintegration of nuclear chromatin and apoptotic body could be observed by light microscopy. Aggregation and margination of nuclear chromatin, concentration of plasm, increment of density and chromatin mass of irregular size could be observed by transmission electronmicroscope. The flow cytometric analysis indicated that the VPA could induce cell apoptosis, apoptosis rate increased in dose-dependent manner, ratio of cells at G0/G1 phase increased and ratio of cells at S phase decreased in dose-dependent manner, the cells were arrested at G0/G1 phase.

结果显示:VPA对MUTZ-1细胞的生长抑制作用呈现时间和剂量依赖性;经4mmol/LVPA处理MUTZ-1细胞72小时后,细胞呈现典型的凋亡形态特征,光学显微镜下可见凋亡细胞胞体固缩、核固缩、核碎裂及凋亡小体;透射电子显微镜下可见凋亡细胞核染色质边集、胞浆浓缩、密度增加,胞装内大小不规则的染色质团块;流式细胞术结果表明,细胞凋亡率随着VPA浓度的增加而逐步增高,G0/G1期细胞比例随着VPA浓度的增加而逐渐增多,S期细胞比例逐渐减低,细胞被阻滞在G0/G1期。

Results1. Content of arsenic in serum raises with dose adding, time of pouring medicine and time of blood sampling prolonging. Thin layer chromatography indicates that there is toadfish in medicine serum of Liu-Shen-Wan.2. The suppressive rate of medicine serum of Liu-Shen-Wan on HL-60 cell raises with adding of dose and time of pouring medicine and prolonging of blood sampling.3. Gimsa dying assay, in situ end-labeling method and flow cell machine all exhibits: HI-60 cells dealed with Liu-Shen-Wan medicine-serum present typical apoptosis morphology.

结果:1 血清砷含量随着给药剂量、给药天数增加和采血时间的延长而升高;薄层色谱表明六神丸含药血清中有蟾酥成分。2 六神丸含药血清对白血病细胞的抑制率,随给药剂量、天数增加和采血时间的延长而升高3 吉姆萨染色、流式细胞仪检测、原位末端标记检测均证明六神丸含药血清能诱导HL-60白血病细胞凋亡。

The local injection of trypsin can be used in clinical practice as a new and effective therapy for snakebite.Our experiments indicate that when a lethal dose of crotalinae snake venom was injected into mice subcutaneously, survival rate increased significantly if a dose of trypin had been injected locally and promptly.

本文提供了一种局部注射胰蛋白酶治疗蛇伤的方法,在给小白鼠皮下注射致死剂量的竹叶青蛇毒后,立即注射胰蛋白酶可提高存活率68%,注射蝮蛇毒后,立即注射胰蛋白酶可提高存活率67%,注射尖吻蝮蛇毒后立即注射胰蛋白酶可提高存活率57%。

ResultsCompared with model group, the clearance rate K of charcoal particles, phagocytic index a, thymus and spleen index and ear swelling degree of 80 mg/kg.bw Fortunella crassifolia flavonoid dose group were significantly improved(P.05), and a very significant effect (P.01) was made to the HC50 value at this dose.

结果与模型对照组相比,金橘黄酮80 mg/kg·bw 剂量时,小鼠碳粒廓清指数K、吞噬指数a、胸腺和脾脏指数、耳肿胀度有显著提高(P0.05),血清溶血值(HC50)有极显著提高(P.01);金橘黄酮160 mg/kg·bw 剂量对全部试验参数的影响具有极显著性(P.01)。

Results: Compared with the model group, helianthin resid rate of the high dose and low dose group of JG, and motilin and CCK was higher. There is statistical signficance.

结果 加味柴平颗粒剂高、低剂量组与模型组相比较,甲基橙残留率降低,而胃动素以及胆囊收缩素升高,差异有统计学意义。

Methods Seventy-five SD rats were randomized into five groups: normal, model, iodine glycerin (20 g/L), high-dose SP (200 g/L) and low-dose SP (100 g/L). The experimental periodontitis rat models were established by ligating molars with steel wire combined with intramuscularly injection of prednisolone (1.25mg, 8 times). Except that the model and normal groups were treated by applying saline, the other groups were treated by applying the corresponding drugs 0.5 mL to the peridentium, tid for 10 days. After treatment, debris index, probe depth and bleeding rate during probing were investigated and the pathological changes of periodontal tissues were also observed.

方法]SD大鼠随机分为正常组,模型组,碘甘油组(20g/L),舒口散高、低剂量组(200、100 g/L);采用钢丝结扎牙齿加肌注波尼松龙(l.25 mg/次,共8次)的方法复制大鼠实验性牙周炎模型;药物采用口腔局部涂搽,每次0.5mL,每日3次,10 d为1疗程,模型组与正常组给予等容积生理盐水;检测各组软垢指数、探诊深度、探诊出血阳性例数,并取牙周组织进行病理学检查。

Serum containing SSTG was obtained at different time after perorally administrated with different dose of SSTG.The cardiomyocytes were deprived of oxygen and glucose to mimic hypoxia reoxygenation injury and were treated by serum containing SSTG when reoxygenation.LDH content in supernatant was detected after reoxygenation.LDH release suppression rate was used to study the time-effect and dose-effect of serum containing SSTG.

以中药有效组分配伍方剂双参通冠方不同灌胃剂量、药后不同取血时间所得的药物血清为受试药物;培养心肌细胞,进行缺氧复氧实验,复氧同时给予不同双参通冠方药物血清处理,实验结束后取培养上清检测LDH值,以LDH释放抑制率为指标,观察含药血清药理作用强度与体内给药的量效、时效关系。

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