查询词典 difference group

To investigate the change of these indexes after treatment between traditional Chinese drug treatment group and control group, and the difference between control group and TCDT group.Methods:1. 40 cases with CHD were divided into TCDT group and control group randomly. The control group was treated by regular western medicine of Isosorbide Dinitrate and ASA while the TCDT group was treated by Guanxinshengmaiyin.

1、选择40例冠心病患者，随机分为两组，即中药治疗组与西药对照组，中药组用&冠心生脉饮&治疗，西药组常规给予消心痛和阿司匹林口服；2、检测两组病人治疗前后血清VEGF和NO的含量；3、选择16例健康体检者作为健康对照组。

Objective To examine the value of Edaravone in the treatment of vascular dementia through ethologic and pathologic changes induced by chronic cerebral ischemiaMethods Wistar rats were randomly divided into sham operated group,model group,edaravone group and duxil groupThe chronic cerebral hypoperfusion models were made by permanent bilateral occlusion of both common carotid arteriesThe memory ability and ultramicrostructure of brain tissue were observedResults The escape latencies in model group were more significantly prolonged than that in the other groups (P＜005)But there was no significant difference between edaravone and duxil groupAnd pathologic changes observed by electron microscope in nerve cell of edaravone group and duxil group and model group were fairly light than that in sham operated groupEdaravone group had increased mitochondrium and frontal membranous synaptic vesicle and more productive metabolic activityConclusions Edaravone can lessen injures of hippocampal neurons loss to improve the spatial learning and memory abilitiesAnd it is proved further that edaravone can treat VD

目的 观察依达拉奉对慢性缺血致血管性痴呆大鼠行为学及形态学的影响，探讨依达拉奉在治疗VD中的应用价值。方法实验分假手术组、模型组及都可喜对照组及依达拉奉治疗组，应用双侧颈总动脉结扎方法制备慢性脑缺血大鼠痴呆模型，观察各组大鼠的记忆功能及脑组织超微结构改变。结果缺血组水迷宫表现同对照组、都可喜组及依达拉奉组相比有显著差异(P＜005)，依达拉奉组与都可喜组相比未见显著差异。同时电镜观察结果也发现依达拉奉组及都可喜组与模型组及假手术组相比神经细胞病理改变较轻，且依达拉奉组线粒体及前膜突触小泡增多，代谢活动较都可喜组旺盛。结论依达拉奉可能通过减轻海马神经元的损伤来改善慢性脑缺血大鼠空间学习记忆障碍，证明依达拉奉对慢性缺血致VD大鼠具有治疗作用。

The treatment regimens of Trial patients who were randomized into control group (Trial-control group) were quite the same with the standard treatments of Nontrial group. There was no significant difference in median survival between Trial-control group and Nontrial group. The median survival of Trial group patients who were randomized into trial group was significantly longer than that of Nontrial group patients.

在Phase III的临床试验中，病患会被随机分派到试验组(Trial-trial)或是控制组(Trial-control)，控制组的治疗方式与非临床试验组类似，两组存活时间在统计上没有差异；使用新药或新疗程的Trial-trial组的存活时间比非试验组显著较长。2000-2003年间存活时间有较长的趋势，而医疗成本部分则是2004年比起2000年显著的升高。

Get the neonate Sprague-Dawley rats medulla spinalis, through the conventional primary cell culture procedure, we use the method of difference adherence time and get the motor neurons of anterior spinal cord. When the neuron is in maturity, we make the mechanical injury model in vitro. All the models were divided into four groups: group A is control group; group B is 100μM ATP group; group C is 100μM ATP+20μg/ml suramin group and group D is 100μM ATP+10μM ouabain+10μg/ml Thapsigargin group. Culture the four groups neurons, after one day, we count the motor neuron, observe the survival and activity of neurons through MTT shade selection experiment, use flow cytometry to analyze the percentage of apoptosis of motor neurons of anterior spinal cord and detect the expression of protein p-GSK-3β(ser9) through Western-Blot technology.

方法取新生大鼠脊髓，通过常规的原代细胞培养程序，采用差速贴壁法分离出大鼠脊髓前角运动神经元，培养成熟后制作神经元机械损伤体外模型，分为A组：对照组、B组：100μMATP组、C组：100gMATP+20μg/ml Suramin组和D组：100μM ATP+IOμM Ouabain+10μg/ml Thapsigargin组，对各组机械损伤的运动神经元进行培养，1天后分别进行运动神经元计数、MTT比色实验观察运动神经元的存活及活性、流式细胞仪分析机械损伤的脊髓前角运动神经元凋亡百分率和Werstern Blot技术检测p-GSK-3β(ser9)的表达。

Results 1 there were no significant differences of amplitude of accommodation and accommodative lag between the myopia group and emmetropia group. The differences of amplitude of accommodation between the myopia group and hyperopia group were significant (t=2.21, P=0.03.05; t=2.83, P=0.006.05). 2 The difference of accommodative lag between the dominant eye (0.73±0.31) D and non-dominant eyes (0.81±0.38) D in myopia group was signiflcant.3 The accommodative lag of dominant eyes was (0.68±0.36) D and it of non-dominant eyes was (0.75±0.34) D, the difference was significant (t=2.06, P=0.042.05, n=95).There was no significant difference between the amplitude of accommodation of dominant eye (12.9±3.09) D and non-dominant eyes (12.6±3.09) D.

结果 近视患儿的主导眼和非主导眼的调节幅度和调节滞后与正视儿童均差异无统计学意义；而其主导眼和非主导眼的调节幅度比远视患儿明显更大(t=2.21， P=0.03.05； t=2.83，P=0.006.05)；两组的调节滞后差异无统计学意义。50例近视患儿主导眼和非主导眼的调节滞后值分别为(0.73±0.31)D和(0.81±0.38)D，主导眼和非主导眼间差异有统计学意义(t=2.14，P＝0.038.05)；调节幅度分别为(13.39±3.51)D和(13.26±0.60)D，差异无统计学意义。95例观察对象的主导眼的调节滞后度为(0.68±0.36)D，非主导眼调节滞后度为(0.75±0.34)D，主导眼和非主导眼间的差异有统计学意义(t=2.06， P=0.042.05)；主导眼调节幅度(12.9±3.09)D，非主导眼为(12.6±3.09)D，差异无统计学意义(t=1.49， P=0.14)。

Result: The nm-HAP-sol present the dispersed and uniformity needle. A431cells inhibition ratio was maximizing at the 5 day in the 400mg/L nm-HAP-sol group, the 200mg/L nm-HAP-sol group and the 100mg/L nm-HAP-sol group, inhibition ratio of the 400mg/L nm-HAP-sol group, the positive control group and the 50mg/L nm-HAP-sol group was respectively (77.89±6.29)%,(77.46±8.26)% and (1.23±0.15)%. It was detected that the number of A431 cells were persistently increasing in the 50mg/L nm-HAP-sol groups while decreasing in the 400mg/L nm-HAP-sol group, the positive control group and abidingly increasing for 4 and 3 days then decreasing in the 100mg/L and 200mg/L nm-HAP-sol group by inverted microscope.

结果：超声乳化法配制的HAP纳米溶胶呈均匀分散的针状；400mg/L、200mg/L和100mg/L溶胶组对A431细胞抑制率在第5天达到各组最大值，此时400mg/L溶胶组与阳性对照组的抑制率分别为（77.89±6.29）%和（77.46±8.26）%， 50mg/L溶胶组抑制率为（1.23±0.15）%；倒置显微镜下见50mg/L溶胶组细胞数目持续增加，聚集成簇，细胞呈现多角形或梭形，胞浆透亮，而100mg/L 和200mg/L溶胶组的细胞数目分别增加至第4天和第3天开始下降，400mg/L溶胶组和阳性对照组细胞数目持续减少，细胞体积缩小，漂浮细胞增多。

There are the normal group,the AS group,the pure swimming group,the swimming plus AS group,the simvastatin group(simvastatin plus AS of overstrain),the large dose Tongxinluo group,the small dose Tongxinluo group,the bulbus allii macrostemonis four drugs group and the ginseng group.

目的对络气虚滞型动脉粥样硬化模型中9个编号组即正常组、动脉粥样硬化组、单纯游泳组、游泳加动脉粥样硬化组、辛伐他汀组(即辛伐他汀+过劳加动脉粥样硬化，下同)、通心络大剂量组、通心络小剂量组、薤白四味组、人参组进行综合评价。

12H treatment group compared with 12h model group: the level of MCP-1 of the Gardenoside group decreased and MIP-1 is no different with the model group; the level of the MCP-1, MIP-1 of the Baicalin group are no different with the model group; the level of MCP-1, MIP-1 of the Concha Margatitifera Usta decreased than the model; the level of MCP-1 of the Cholic Acid group is no different and the level of MIP-1 decreased than the model; the level of MCP-1 of the Hefang Group decreased and the level of MIP-1 of it decreased than the model group.

合方治疗脑缺血24小时组缺血脑组织nNos光密度值较缺血24小时模型组明显降低（p＜0.01）。细胞的死亡分为坏死和凋亡两种方式，凋亡是细胞为避免对机体造成更大损伤而采取的一种程序性的死亡方式。珍珠母、胆酸、栀子苷、黄芩苷、合方治疗组较脑缺血24小时模型组可明显降低P53阳性表达，考虑药物抑制细胞凋亡可以起到对缺血脑组织的保护作用。

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中，脑缺血能够诱导P53蛋白表达，P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程，药物通过抑制细胞凋亡，从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高（p＜0.05）。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。

12H treatment group compared with 12h model group: the density of light of nNos of the Gardenoside group decreased and P53 decreased with the model group; the density of light of the nNos of the Baicalin group decreased and which of the P53 is no different with the model group, the density of nNos of the Concha Margatitifera Usta decreased and which of P53 decreased than the model; the density of of nNos of the Cholic Acid group is no different and the density of P53 decreased than the model; the density of the light of nNos of the Hefang Group is no different and which of P53 decreased than the model group.

珍珠母治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。栀子苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。黄芩苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。合方治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。

"The operators will suffer a temporary decrease in the number of consumers and short message services as people may not like having to provide their ID cards for a phone number," Xu Junqi, vice-dean with the Policy-Making Institute of the Telecommunications Research Centre under MII, said yesterday.

信息产业部下属的通信决策研究院副主任徐俊其昨天说：&实行手机实名制后，人们可能不太情愿用身份证注册手机号，所以通信运营商会暂时面临用户减少和短信用户减少的问题。&

I don't believe in make-believe!

我不相信这些虚构的故事。