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cerebral cortex相关的网络例句

查询词典 cerebral cortex

与 cerebral cortex 相关的网络例句 [注:此内容来源于网络,仅供参考]

Expression of nNOS mRNA, eNOS mRNA and iNOS mRNA in the E 20 cerebral cortex: There were no obvious changes of expression of nNOS mRNA in ETU-injected and control groups. Expression of eNOS mRNA increased significantly in cerebral cortex of the encephalocoele group, And there were no obvious changes of expression of eNOS mRNA in cerebral cortex of the control group and the other ETU-injection groups.

六、不同组E20胎鼠大脑皮质中nNOS mRNA、eNOS mRNA和iNOS mRNA的表达 1、实时定量PCR检测发现对照组、给药无畸形组、脊柱裂组和脑膨出组E20胎鼠大脑皮质中nNOS mRNA表达无明显改变; 2、给药无畸形组、脊柱裂组和对照组胎鼠大脑皮质中eNOSmRNA表达无明显改变,而脑膨出组胎鼠大脑皮质中eNOS mRNA表达明显增多; 3、在各组未检测到iNOSmRNA表达。

RESULTS: Compared with patients in the non-AD group, those in the AD group showed decreased cerebral glucose metabolism mainly in the temporal and frontal lobes and the limbic system in the right cerebral hemisphere and the inferior frontal gyrus and precentral gyrus in the left cerebral hemisphere, and increased cerebral glucose metabolism mainly in the temporal and occipital lobes in the right cerebral hemisphere and the parietal and occipital lobes and limbic system in the left cerebral hemisphere.

结果: 抑郁焦虑组与非抑郁焦虑组比较,脑葡萄糖代谢下降的脑区集中于右侧大脑半球,主要位于颞叶、额叶及边缘系统,左侧大脑半球较少,主要位于额下回和中央前回;脑葡萄糖代谢升高的脑区集中于右侧大脑半球的颞叶、枕叶和左侧大脑半球的顶叶,枕叶及边缘系统。

ACA = anterior communicating artery; BA = basilar artery; LACA = left anterior cerebral artery; LCCA = left common carotid artery; LECA = left external carotid artery; LICA = left internal carotid artery; LMCA = left middle cerebral artery; LPCA = left posterior cerebral artery; LSA = left subclaian artery; LA = left ertebral artery; RACA = right anterior cerebral artery; RCCA = right common carotid artery; RECA = right external carotid artery; RICA = right internal carotid artery; RMCA = right middle cerebral artery; RPCA = right posterior cerebral artery; RSA = right subclaian artery; RA = right ertebral artery.

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There were no statistically differences between the 8th~ 14thday, the 15th - 21th day post-BC and the control group. During the rt~24tkday post-BC , pycnosis degeneration or necrosis neurons of the locus cerebral cortex, dorsal hippocampus, dentate fornix were significantly increased, then decreased gradually, but recovered to normal by the 24thday after BC, and especially in parietal cortex and piriform cortex the necrosis neurons were significantly increased than temporal cortex , and there were no statistically difference berween the left and the right side. Pycnosis degeneration or necrosis neurons in the brainstem reticular formation were markedly increased in the 4thday after BC, and there were no statistically difference among the other groups and the control group.

与对照组相比,BC后在大脑皮质、背侧海马和齿状回部位,固缩变性和不完全坏死细胞数先显著增加(P<0.05),然后逐渐减少,至24d基本恢复正常;顶叶、梨状皮质比颞叶皮质变性坏死细胞多,差异有高度显著性(P<0.01);大脑左侧比右侧变性坏死细胞稍少但差异无显著性;在BC后4d,脑桥核、斜方体核平面的脑干网状结构中固缩变性和不完全坏死细胞数明显增多(P<0.05),其它组间无显著性差异。

In particular, the researchers found that regions in the cerebral cortex, including the somatosensory cortex, the insular cortex and the cingulate cortex, all behaved similarly for both toothaches.

研究人员发现,大脑皮层内的区域——包括体感觉皮层、岛叶皮层和扣带皮层——对两种牙痛作出类似反应。

In particular, the researchers found that regions in the cerebral cortex, including the somatosensory cortex, the insular cortex and the cingulate cortex, all behaved similarly for both toothaches.

研究员发现特别是大脑皮层区域,包括躯体感觉皮质、岛叶皮质和扣带回皮质,对两种牙疼都表现出相似的反应。

Hypoxia-ischemia for 10 minutes group , 20 minutes group: our experiment showed that at 24 hours after cerebral hypoxia-ischemia, expression of MMP-9mRNA was maximally increased, individually, in the hypoxia-ischemia for 10 minutes group, MMP-9mRNA was greatly expressed within superolateral surface cortex of cerebrum, whereas in the hypoxia-ischemia for 20 minutes group, the labeling of hybridization for MMP-9mRNA was strongly appeared within the cortexof cerebrum both the cortex in the subsequence of medial surface at the periphery of the cerebral longigudinal fissure and the cortex in superolateral surface where also there showed a few stained fibers.

3缺氧缺血10分钟、20分钟组:缺氧缺血后24小时脑内MMP9 mRNA阳性表达至最高峰,缺氧缺血10分钟组大量表达在端脑背外侧面皮质内,缺氧缺血20分钟组在端脑内侧面后份大脑纵裂旁周围皮质及背外侧面皮质内呈强阳性表达,少数纤维呈亦阳性染色。

Toluidine blue staining procedure showed that the neural complex consisted of cerebral ganglion, neural gland and loose connective tissue; the cerebral ganglion was divided into three parts as cerebral cortex, medullary layer and transitional zone; the neural gland was composed of glandular lobules, closely apposing at ventral side of the cerebral ganglion; the neurons and nerve fibers were colored deeply, making a distinct contrast with the background, while neither the neurons nor the nerve fibers were distinctly colored by Golgi-argentraffin staining procedure.

甲苯胺蓝染色法显示,神经复合体由神经节、神经腺和疏松结缔组织组成;神经节分为皮质部、髓质部和过渡带;神经腺位于神经节背侧面,由腺小叶组成;神经细胞呈深蓝色,神经纤维呈浅蓝色,与背景反差较大。Golgi镀银法显示的结果不理想,神经细胞和神经纤维着色不明显。

Besides hemal sex migraine, cause the cause that have a headache a lot of, if abscess of all sorts of sexual disease, encephalitis, meningitis, head is affected inside skull; All sorts of cerebral blood-vessel pathological changes, if cerebral block, cerebral hemorrhage, arachnoid haemorrhage finishing speech, ventricle bleeds, Hypertensive Cerebral disease; A pathological change is taken inside skull, if colloid tumour mixes meningeal tumour, head,transfer tumor; All sorts of heads are traumatic if defeat of cerebral concussion, head cracks injury, putamen,issue haematoma; The waist is worn cause low skull pressure head painful; All sorts of neuralgic; The headache of disease of facial features division and infection, toxic, heatstroke and nerve function all can be caused mussily have a headache.

除了血管性偏头痛外,引起头痛的原因很多,如颅内感染性疾病、各种脑炎、脑膜炎、脑脓肿;各种脑血管病变,如脑梗塞、脑出血、蛛网膜下腔出血、脑室出血、高血压脑病等;颅内占位病变,如脑膜瘤、脑胶质瘤和转移肿瘤;各种脑外伤如脑震荡、脑挫裂伤、硬膜下血肿;腰穿引起低颅压头痛;各种神经痛;五官科疾病的头痛以及感染、中毒、中暑和神经功能紊乱均可引起头痛。

Although does not have the obvious clinical manifestation,but it is the symptom cerebral infarction\'s one kind of omen,therefore has the necessity very much to this sickness\'s research.3、The discussion diabetes mellitus merge cerebral infarction\'s CT characteristic,CT blocks the stove spot and the cerebral infarction whether to have the symptom to relate.4、Diabetes merge symptomless cerebral infarction not easily notes by the patient and doctor or neglects,continues to have the cerebral infarction again easily,no matter but is symptomless perhaps has the symptom, can cause patient\'s cognition function to receive the harm.

虽没有明显的临床表现,但它是症状性脑梗塞的一种前兆,因此对此病的研究很有必要。3、探讨糖尿病合并脑梗塞的CT特点,CT梗塞灶部位与脑梗塞是否出现症状的相关系。4、糖尿病合并无症状性脑梗塞不易被患者及医师注意到或忽略,易再继续发生脑梗塞,而不管是无症状性或是有症状性的,都能使患者的认知功能受到损害。

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