查询词典 cartilage

Results The pharynx, larynx, epiglottic cartilage, thyroid cartilage, cricoid cartilage, arytenoid cartilage, hyoid bone, vocal cord, artery, and vein were successfully restructured and displayed.

结果 成功重建并显示了咽、喉、会厌软骨、甲状软骨、环状软骨、杓状软骨、舌骨、声带、动脉、静脉、环杓肌、咽缩肌、甲状舌骨肌等结构，形成三维立体模型，使其可以任意角度旋转、切割、观察。

To sum up, we first conclude that we can isolate mesenchymal stem cells from human fetal articular cartilage and they can be induced to differentiae into several cells under appropriate induction conditions. Anymore, they have their original tissue speciality. Therefore, we first report that mesenchymal stem cells of articular cartilage origin are superior to mesenchymal stem cells of bone marrow origin as seeding cells of cartilage tissue engineering and we provide a kind of new seeding cells of cartilage tissue engineering for the future clinic application.

综上所述，我们首次报道不仅可从人胚胎的关节软骨中获得间充质干细胞，而且软骨来源的间充质干细胞除具有骨髓来源的间充质干细胞的多向分化能力外，还具有其来源的软骨组织特异性，那么，其作为软骨组织工程的种子细胞要优于骨髓来源的间充质干细胞，从而首次提出了一种新型的软骨组织工程的种子细胞，对于未来的软骨组织工程的临床应用提供理论依据。

By using low-frequency dynamic frequency density wave, the OA kneepad could regulate many aspects of pathological process of knee OA including: inhibition of biological effects of cytokine, MMP-3, HA, oxygen free radicals and NO; enhancing the biological activity of SOD; up-regulating the mRNA expression of Bcl-2 as well as down-regulating the mRNA expression of P53, thereby to inhibit the apoptosis of cartilage cells and delay the cartilage degeneration of the macro-morphology, cartilage cells and cartilage matrix.

OA护膝采用低频温热动态变频疏密波，针对膝OA病理过程中的多个环节进行调节，能有效抑制细胞因子、MMP－3、HA、氧自由基、NO的生物学效应，增强SOD的生物学活性，提高关节软骨细胞Bcl-2mRNA表达，减弱软骨细胞P53mRNA表达，从而抑制软骨细胞凋亡，延缓膝关节软骨宏观形态、软骨细胞及软骨基质的退变。

PartⅢThe study of macrography, histology, pathology and apoptosis After the impactive injury of acetabular cartilageObjective To discuss the change of short and long peoriod for the impactive injury of acetabular cartilage by using different energy,the pathological mechanism of chondral injury and the relation between chondrocyte apoptosis and cartilage degeneration. Methods fifty-four New Zealand white rabbits were divided into high energy and low energy groups randomly, of which the two groups which were made through the homemade impactive equipment by impacting the right acetabular cartilage of rabbit at 35cm and 15cm with 0.400kg weight vertically and simulating the mechanism of impactive injury of human acetabular cartilage.

方法新西兰大白兔54只，平均年龄6个月，体重2.5±0.5kg，模拟人髋臼软骨冲击伤机制，采用自制冲击装置，用质量为0.400kg的重锤，分别沿导向杆从高35cm和15cm处下滑垂直冲击兔右侧髋臼软骨，建立兔髋臼软骨冲击伤模型，随机分为高能组和低能组，另一组为对照组，每组18只。

After 24 weeks,the operation area of group A was more smooth,and the surrounding normal cartilage naturally straight flush,transparent form new cartilage,subchondral bone formation in good condition;Group B restoration surgery the basic integrity of the cartilage tissue, center is not yet fully integrated,there was slight depression;CollagenⅡimmunohistochemistry of cartilage that was new brown area.Group C has no formation of articular cartilage.The growth and the intergration of subchondral bone of group A and B were better.

术后24周取材，见A组山羊手术区关节表面较为光滑，与周边正常软骨自然连续平齐，透明的新生软骨组织形成，软骨下骨形成完好；B组山羊手术区修复的软骨组织基本完整，中心部位仍未完全融合，有微小凹陷；Ⅱ型胶原免疫组化示新生软骨组织呈棕黄色。C组术区关节凹陷，无关节软骨组织形成。A组和B组，软骨下骨的生长及与周围组织的结合均较好，无植入物脱落现象的发生。

After 12 weeks,the result showed the operation area of group A articular surface was smooth,with the surrounding normal cartilage naturally straight flush, transparent form new cartilage tissue;Group B restoration surgery the basic integrity of the cartilage tissue,but the center of many not fully integrated,there is slight depression,surface wear,good subchondral bone formation;control group, depression joint operation areas,non-articular cartilage formation,such as lack of bone joints.

术后12周取材，A组修复区可见部分软骨样组织，关节软骨无磨损，修复的软骨呈白色半透明外观，与周围正常关节软骨有连续性，可见一明显的凹陷，无明显软骨下骨外露；B组修复区也可见部分软骨样组织，关节软骨在修复区可见磨损，软骨呈白色不透明外观，软骨下骨形成较好；空白对照组术区关节凹陷，无关节软骨组织形成，关节下骨缺如。

Recently, bipolar radiofrequency energy has become popular as a method of performing chondroplasty because of accurately treatment to degenerative cartilage and minimal injury to normal cartilage. But the questions of the effects of bipolar RFE to degenerative cartilage, the reaction of chondrocyte and cartilage matrix, the change of the cartilage after the treatment, and the influence to the cartilage degenerative course, were still unknown.

但是双极射频在关节软骨退变治疗中究竟发挥何种作用，治疗区软骨细胞及软骨基质成分反应是怎样的，随着时间的推移软骨表面的变化如何，双极射频对软骨退变的病程有无影响，这些问题都不清楚。

The human cartilages are composed of chondrocyte and extracellular matrix,the form of chondrocytes are hypertrophy and the quantity are less;the ECM of cartilage are compised of type II collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury,the ommilayer injury ofcartilage can be recovered by marrow,but because of without stimulation mechanism,the new tissues are merely fibrocartilages,they can not be coincide with hyaline cartilage in menchanics;the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation,thus,the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.Ever since,people tried their best to find a way to reconstruct articular cartilage.

造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte. Ever since, people tried their best to find a way to reconstruct articular cartilage.

中文题名人骨髓基质干细胞成软骨诱导及多孔复合材料作为细胞载体的体外实验研究副题名外文题名 Cartilage induction of human mesenchymal stem cells and experiment on compound porous materials as cells' scaffold in vitro 论文作者刘晓岚导师周江南学科专业外科学研究领域\研究方向学位级别博士学位授予单位中南大学学位授予日期2003 论文页码总数68页关键词骨组织工程软骨细胞骨髓基质干细胞壳聚糖高分子外消旋聚乳酸馆藏号BSLW /2003 /R68 /10 造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

Results 1、 Generally, we can see the original blue and white, shiny, no cracks in the articular surface of the cartilage after the stress increases gradually yellow, surface roughness, cracks appear; when the pressure decreases, the yellowing, rough, the color of the fracture restore gradually and become shiny.2、the shiny smooth surface can be seen under a light microscope, formation, cell distribution, tidy, clear the level of cartilage at the articular surface stress increases, the surface roughness changes, defects, disordered cells, uneven dyeing ; when the articular surface of the pressure gradually decreased, the cartilage gradually repair and the surface of cells at the surface appear only disorder.3、immunohistochemical observation can be seen throughout the observation period, cartilage cells are type Ⅱ collagen expression and expression after 3 weeks gradually weakening, when the seventh week begin to strong gradually.4、 electron microscopy shows that when stress increases the articular surface, the cartilage cells became flat, the cytoplasm in the endoplasmic reticulum, Golgi apparatus decreased with collagen disorders; and when stress decreases the articular surface, cartilage cells gradually returned normal, cytoplasm in the endoplasmic reticulum, Golgi body gradually restore quantity; collagen fibers with a gradual rules.

结果：①大体观察可见到原本蓝白色、有光泽、无裂纹的软骨在关节面压力增大后，逐渐呈灰黄色，表面粗糙，出现裂隙；当压力逐渐减小后，变黄、粗糙、有裂隙的软骨颜色逐渐恢复，变得有光泽②光镜下可见表面光滑、平整，细胞分布均匀、整齐，层次清楚的软骨在关节面压力增大后，表面变粗糙、缺损，细胞排列紊乱、染色不均；当关节面压力逐渐减小后，软骨表面逐渐修复，细胞仅在表层排列紊乱③免疫组织化学观察可见整个观察期内软骨细胞胞浆内均有Ⅱ型胶原表达，术后3周内表达逐渐变弱，从第7周时开始逐渐变强。④电镜下可见当关节面压力增大后，软骨细胞逐渐变扁，胞质中内质网膜、高尔基体减少，胶原排列紊乱；当关节面压力减小，软骨细胞形态逐渐恢复正常，胞质中内质网膜、高尔基体数量逐渐恢复；胶原纤维排列逐渐有规则。

Graf, without question, is the greatest women's player in history.

格拉芙，现在，毫无疑问是女子网坛历史上最伟大的选手。

If not, you will have to look around at all your important data.

如果没有，您将需要检查所有的重要数据。