英语人>网络例句>NOS 相关的网络例句
NOS相关的网络例句
与 NOS 相关的网络例句 [注:此内容来源于网络,仅供参考]

By using β-NADPH-diaphorase histochemical staining technique,wefound for the first time that NOS activity was detected in the epithelium of airpathway and endothelium of vessels but was not detected in the small vessels(ID<100μm),and the NOS express in pulmonary vascular endothelium of preascitic andascitic broilers were elevated,especially in the endothelium and smooth muscle layer ofsmall vesseles with thicker walls and of muscularized nomuscular artery in asciticbroilers.

本研究从田间自然发生腹水症的肉鸡群中选取正常肉鸡、亚临床腹水鸡和腹水鸡为实验对象,通过比较这三种肉鸡血流动力学、心电图、动静脉血血气、肺外动脉舒张反应性、肺血管结构以及肺组织一氧化氮合酶的活性和分布等方面的不同,探讨了肺动脉舒张能力在肉鸡肺动脉高压形成中的作用和机制。

Results: The NOS positive cells are scattered in pterygopalatine ganglion, otic ganglion. The decreased density and the increased somatic size of the NOS positive cells of pterygopalatine ganglion, otic ganglion of rats was followed in rat's development and to the largest in adulthood.

结果:NOS神经细胞在翼腭神经节、耳神经节中总体呈均匀散在分布,从幼年到成年,密度呈逐渐下降趋势,而NOS神经元胞体大小逐渐增加,至成年时最大,直至老年无明显改变。

However, BH4 stopped subsequent remodeling when NOS activity was otherwise declining. A broad antioxidant, Tempol, also reduced oxidant stress yet did not recouple NOS or reverse worsened hypertrophy/fibrosis from sustained transverse aortic constriction.

然而,在NOS活性降低时BH4阻止心脏的继续重构,广义的抗氧化剂,Tempol也可以在保持主动脉缩窄的情况下,NOS没有恢复偶联或逆转心脏肥厚或纤维化,降低氧化应激。

The proportion of the number of double-labeled neurons with NOS/TMR was 55%(17/31)and 34%(17/49) in the whole population of the NOS positive neurons and retrogradely neurons,respectively.

计数显不:NOS/TMR双标记细胞分别占NOS阳性细胞总数的55%(17/31)和TMR逆行标记细胞总数的34%(17/49)。

Methods The rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

MethodsThe rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

方法采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

The inducible nitric oxide synthase is obviously expressed in speen of rats under the predator stress, suggesting important function for iNOS in speen physiology. However, the expression and subcellular localization for NOS in speen development and differentiation are largely unknown. In the present study, the changes in iNOS activity in spleen of rats under predator stress were studied to explore the role of iNOS in the decreased immunoactivity of the stressed rats. The changes in iNOS were observed with immunobistochemical method, and the contents of NOS and NO in spleen with biochemical method.

在捕食压力下,大鼠脾脏诱导型一氧化氮合酶明显表达,说明iNOS起到了重要的生理功能,然而,NOS的表达及在亚细胞水平的定位以及相应的差异变化等都很不明确,本试验采用捕食应激模型,利用免疫组织化学方法和生化测定,检测心理性应激对大鼠脾脏iNOS活性的影响及探讨这种影响与应激动物免疫力下降之间的关系。

Changes of the NOS activity and the expression of NOS protein in central nervous systems of rats,which were subacutely intoxicated with oral exposure to lead acetate for 30 days,were observed with improved NADPH techniques and immunohistochemistry to study neurotoxic mechanism of lead.

本文采用SD成年大白鼠饮用醋酸铅水染毒30天,然后用改进的NADPH测定法及免疫组化SP法观察脑组织中NOS酶活性及NOS酶蛋白亚型的变化。

The vasodilating mechanism of AE from CM ma he related to promoting the release of NO from endothelium of blood vessel and synthesis of NOS and NOS in aortic tissue.

桑白皮丙酮提取物的舒张血管作用机制可能与血管释放NO和促进cNOS、NOS合成有关。

We observed the correlation between the dynamic change of the serumconcentration of NOS and hemodynamics index,found the serum concentration ofNOS was depressed observably after lung contused and NOS was actived and arrivedpeak at 24h after injected LPS.L-arigin can improve hemodynamics index andthe lung wet/dry index in lung contusion phase; Aminoguanidine can improvehemodyamics index and the lung wet/dry index obviously in ARDSphase.

通过观察ARDS动物模型血清一氧化氮合酶浓度的动态变化以及与血气指标和血液动力学监测指标的关系,发现肺挫伤早期NOS的浓度明显降低,ARDS发生后NOS的浓度明显升高,并在24h达到其峰值,肺挫伤早期给予NOS促进剂、ARDS发生过程中给予NOS阻滞剂可明显改善血气及血液动力学指标、肺湿/干比。

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