平滑肌

词组短语

smooth muscle

与平滑肌相关的网络例句 [注：此内容来源于网络，仅供参考]

We studied thickness and area of airway smooth muscle and airway wall, standardized by perimeter of basement, and examined Brdu positive cell nucleus of airway smooth muscle, expression of phenotype mark protein such as sm α-actin, sm-MHC and calponin of airway smooth muscle by immunohistochemistry.

设立正常组、模型组、中药组、西药组和中西结合组，采用免疫组化方法分析激发二周、四周哮喘大鼠中、小气道壁厚度及面积、气道平滑肌厚度及面积、气道平滑肌细胞Brdu掺入、气道平滑肌表型标志蛋白(sm α-actin、sm-MHC、calponin)表达情况。

Result: At two weeks after OVA challenge, in model group airway wall thickness and positive cell nucleus of airway smooth muscle begin to grow up. At four weeks after challenge, airway smooth muscle content begins to elevate. At two and four weeks, expression of phenotype mark protein begins to low down, except for sm α-actin at two weeks.

结果发现，模型组大鼠激发二周后出现管壁增厚，四周后出现平滑肌层增厚，二周时即开始出现气道平滑肌Brdu掺入增多；smα-actin于二周时表达增强，而于四周时减弱，sm-MHC、calponin在二周、四周时表达均出现减少；电镜发现平滑肌细胞肌丝稍减少，而高尔基体、粗面内质网等细胞器增多。

The transform of phenotype of VSMC: The mRNA expressions of SM22α-a notate gene of contractile VSM-were all detected on normal aortas,but decreased normally when the aortas were destroyed and cultured in vitro for 10 days,especially in the serum groups,whose SM22αmRNA could not be detected by RT-PCR.Here,BQ123 can increased their expressions.This demonstrated that serum culture and the secretion of ET-1 can produce the transform of phenotype from contractile to synthesize type.

血管平滑肌表型的转化：RT-PCR检测发现，血管平滑肌收缩表型标志基因SM22α在正常血管平滑肌中都有表达，但在内皮损伤后体外培养10天的血管表达明显减弱，其中血清培养组血管基本不表达，加内皮素受体阻断剂BQ123后，各组SM22α的表达均有所上调，这表明内皮素分泌和血清培养促进了血管平滑肌的表型由收缩型向分泌型的转化。

Wenxin capsule can promote endolethial cells releasing CO and synthesing 〓, enhance activity of HO, induce angiectasis through activating guanylic cyclase and adenglate cyclate of intracellar of vessel smooth muscle cell to enhance content of intracellar cGMP and AMP. 3. Wenxin capsule can inhibit releasing of platelet activiting factor 〓 which inhibit platelet aggregation and have antithrombosis function. 4. Wenxin capsule can promote activity of t-PA, reduce activity of PAI-1, strengthen fibrinolytic function, therefore inhibit fibrin disposition. 5. Wenxin capsule can inhibit synthesis of smooth muscle cellular DNA. Its function don't kill it directly but prevent transformation from period Go to peried S. 6. Wenxin capsule is an effective prescription of anti-coronary atherosclerosis atherosis. The results of image pattern analysis tastifies that area of lipid plaque in treating group reduce 54% and 58% in contrast with that in control group. The results of light, electro- microscopy show that structure of endolethial cell and smooth muscle cell in treating group is more integral than that in control group.

其作用机制之一是由于该药具有良好的调脂降脂作用，其不仅能明显降低血清LDL-c及升高HDL-c含量，且能有效阻止TC沉积于血管壁，从而保护内皮细胞结构完整；此外，温心胶囊尚具有明显的抗氧化作用，该药不仅能明显降低血浆LPO含量，且能提高SOD活性，同时其对血红素在细胞内代谢的调节作用也参与了抗氧化过程。2 温心胶囊能明显促进内皮细胞释放CO及合成〓，提高HO活性，通过激活血管平滑肌细胞内鸟苷酸环化酶及腺苷酸环化酶引起细胞内cGMP、AMP浓度增加而引起血管舒张。3 温心胶囊能明显抑制血小板激活因子〓的释放，从而抑制血小板聚集，抗血栓形成。4 温心胶囊能明显提高t-PA活性，降低PAI-1活性，增强纤溶功能，从而抑制纤维蛋白沉积。5 温心胶囊能明显抑制平滑肌细胞DNA合成，其抑制作用不是直接杀伤抑制，而是阻止细胞由Go期向S期转化。6 温心胶囊是抗冠状动脉粥样硬化的有效制剂，图象分析结果证实，该药治疗组脂斑面积比对照组缩小了54％和58％，光电镜结果显示内皮细胞及平滑肌细胞结构较为完整。

SMA staining was positive in basophilous small blocks, spindle or irregular cell cluster. Of the 218 cases, 56 cases had smooth muscle. S-100 staining demonstrated that neurofilament protein and adipocytes were positive. Of the 218 cases, 44 cases had adipose tissue.

平滑肌肌动蛋白染色显示嗜碱性的小团状、梭形或不规则形的细胞束呈阳性反应，证明为平滑肌，218例中56例显示了平滑肌的存在。S-100染色神经纤维丝及脂肪细胞均呈阳性反应，218例中44例有脂肪组织。

In the normal uterus, Cytokeratins immunolabelling were detected in glandular cell, luminal epithelial cell, Vimentin immunolabelling were detected in stromal cell and endoblastic cell; CK7 immunolabelling were not detected in any tissue of the yak utenus.

研究结果显示：未妊娠时，泛角蛋白在子宫内膜腺上皮细胞、腔上皮细胞内表达，波形蛋白在子宫内膜基质细胞内表达，平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达，牦牛子宫任何部位均不表达角蛋白7；妊娠30天左右时，泛角蛋白在子宫内膜腺上皮细胞、子宫内膜腔上皮细胞、滋养层细胞、内胚层细胞和尿囊细胞内表达，波形蛋白在子宫内膜基质细胞和内胚层细胞内表达，平滑肌肌动蛋白在子宫平滑肌和血管平滑肌内表达，角蛋白7在尿囊细胞内表达，偶尔在腔上皮细胞的细胞核边缘表达；消化法进行原代培养时，组织经胶原酶消化并通过100目和400目筛网组合可以有效地分离原代子宫内膜基质细胞和子宫内膜腺上皮细胞；分离得到的子宫内膜基质细胞活率达90%以上，并可在体外传代7次以上；分离得到的子宫内膜腺上皮细胞活率可达85%以上，并可在体外传代5次以上；RPMI1640培养基最适合子宫内膜基质细胞和子宫内膜腺上皮细胞的生长，维持子宫内膜基质细胞正常生长的FBS添加量为20%，维持子宫内膜腺上皮细胞正常生长的胎牛血清添加量为30%。

Result: Moderate to strong immunostainning was observed at the circularmuscle,submucosal region in normal bowel and in ganglionic bowel from patients with HD.

电镜下在环形肌近粘膜缘及环形肌外层见肠间质细胞与平滑肌细胞之间、平滑肌细胞与平滑肌细胞之间有缝隙连接存在，在无神经节细胞肠段未见此结构。

Chloride channels have been identified in vasucular smooth muscle cells. These channels have been shown to be involved in myogenic tone regulation and neuromuscular transmission in various vascular beds. However, whether the chloride channels are responsible for the formation of excitatory junction potentials of the smooth muscle cells in the spiral modiolar artery remains elucidated.

许多研究已证实，血管平滑肌细胞膜上存在氯离子通道，进一步研究发现氯离子通道不仅参与调节平滑肌细胞的肌原性紧张，而且参与了许多种类血管床的神经平滑肌细胞之间的信息传递，但Cl-通道及氯离子通道阻断剂对耳蜗螺旋动脉细胞兴奋性接头电位(excitatory junction potential，EJP)是否有影响，目前还不清楚。

Under the same concentration, tadalafil induce higher ratio of relation responses in the smooth muscles of CRF rabbit cavernosal strips, compared with papaverine and sodium nitroprussiate, with most significant difference occurring under the concentration indicators of 10-5 mol/L and 10-4 mol/L (P.01). Conclusions The effects of tadalafil on relaxation responses in CRF rabbit cavernosal strips highly depend on its concentration. The effect of tadalafil on relaxation responses in CRF rabbit cavernosal strips is better than that of papaverine and sodium nitroprussiate. Tadalafil may be an effective drug for treating CRF reduced erection dysfunction.

同一浓度下，他达拉非对慢性肾功能衰竭兔阴茎海绵体平滑肌舒张百分比与罂粟碱、硝普钠相比较，舒张百分比均大于罂粟碱和硝普钠，且在10~(-5)mol/L、10~(-4)mol/L浓度，他达拉非对慢性肾功能衰竭兔阴茎海绵体平滑肌舒张百分比，与罂粟碱、硝普钠相比较有显著性差异(P＜0.01)结论他达拉非对慢性肾功能衰竭兔阴茎海绵体平滑肌舒张反应的影响具有浓度依赖性，他达拉非对慢性肾功能衰竭兔阴茎海绵体平滑肌舒张反应的影响优于罂粟碱及硝普钠，他达拉非可能是治疗慢性肾功能衰竭性阴茎勃起功能障碍的理想药物。

Motilin and gastrin induced contraction of isolated gastric smooth muscle cells in a dose-dependent manner.(2) Motilin- and gastrin-induced antral smooth muscle cell contraction was inhibited by antibodies against Gαi-3. Motilin and gastrin caused an increase of Guanosine-5′-(3-［35S］thio)triphosphate(［35S］GTPγS) binding of Gαi-3.(3) Contraction of antral muscle cells induced by motilin and gastrin was inhibited by the phospholipase C inhibitor U-73122 and the IP3 receptor antagonist heparin.

结果显示：(1)胃动素和胃泌素对胃窦平滑肌细胞均有收缩作用；(2) Gαi-3抗体可抑制胃动素和胃泌素加强胃窦平滑肌细胞的收缩，胃动素、胃泌素明显增加Gαi-3抗体与［35S］GTPγS的结合；(3)磷脂酶C抑制剂U-73122、三磷酸肌醇受体拮抗剂肝素可抑制胃动素和胃泌素引起的胃窦平滑肌细胞的收缩。

与平滑肌相关的网络解释 [注：此内容来源于网络，仅供参考]

smooth muscle：平滑肌

心肌纤维的连接处称三、平滑肌 平滑肌(smooth muscle)广泛分布于血管壁和许多内脏器官,又称内脏肌. 平滑肌的收缩较为缓慢和持久. (一)平滑肌纤维的光镜结构 平滑肌纤维呈长梭形,无横纹. 细胞核一个,呈长椭圆形或杆状,

smooth muscle：血管平滑肌细胞

血管内皮细胞:Vascular smooth muscle cell | 血管平滑肌细胞:smooth muscle | 子宫平滑肌:uterine smooth muscle

jejunal smooth muscle cell：空肠平滑肌细胞

氧化低密度脂蛋白:vascular smooth muscle cell | 空肠平滑肌细胞:jejunal smooth muscle cell | 平滑肌细胞:Smooth muscle cell interstitiall cells of Cajal

leiomyosarcoma：平滑肌肉瘤

平滑肌肉瘤(leiomyosarcoma)是一种由平滑肌细胞或向平滑肌分化的间充质细胞所组成的恶性肿瘤,常见于子宫、胃肠道、腹膜后腔[1~7]. 发生于口腔者极为少见. 本文报道一例发生于下颌牙龈的平滑肌肉瘤,结合文献对其发病及诊治特点、治疗与预后进行讨论.

leiomyosarcoma：平滑肌: 平滑肌肉瘤

Invasive carcinoma 侵袭癌 | leiomyosarcoma 平滑肌: 平滑肌肉瘤 | leukemia 造血细胞: 白血病

clitoral corporal smooth muscle cell：阴蒂海绵体平滑肌细胞

血管平滑肌细胞:vessel smooth muscle cell | 阴蒂海绵体平滑肌细胞:clitoral corporal smooth muscle cell | 肺动脉平滑肌细胞:pulmonary artery smooth muscle cell

smooth muscle cell：平滑肌细胞

研究组说,由脂肪干细胞转化而成的细胞能像平滑肌细胞(smooth muscle cell)那样收缩和舒张. 平滑肌能帮助心脏跳动、血液输送、消化系统和膀胱运作. 洛杉矶的加利福尼亚州大学医学院助理教授罗德里格斯说:"我们能用平滑肌细胞来再生和修复受损的器官.

smooth muscle cell：血管平滑肌细胞

血管反应性:arterial smooth muscle cell | 血管平滑肌细胞:smooth muscle cell | 胃平滑肌细胞:Gastric smooth muscle cell

Smooth muscle cells：平滑肌细胞

而动脉硬化症的形成与血管内皮细胞(endothelial cells)、平滑肌细胞(smooth muscle cells)及白血球,尤其是单核白血球(monocytes)的交互作用有关. 此外,在发炎过程中,平滑肌细胞由正常的收缩型(contractile phenotype)转变为合成型(synthetic phenotype),

tracheal smooth muscle：气管平滑肌

平滑肌细胞:arteriolar smooth muscle cell | 气管平滑肌:tracheal smooth muscle | 肺动脉平滑肌:Pulmonary Artery Smooth Muscle