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附着胞

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Utricles convex abaxially, plane or slightly concave adaxially; rostra 1/8-1/5 length of kernel; seed adherent to pericarp; leaves and bracts acute but not acicular at apex.

胞果凸背面,正面的平面或者稍凹;喙1/8-1/5段珠心;种子附着于果皮;叶和苞片锐尖但不是针状的在先端。 16 Corispermum 虫实属

Most conidia penetrate host cuticle with germ tubes in acanthoid surface topography and spinous surface topography of host while a few conidia does so with appressorium.

在嵴状突起结构区和刺状突起结构区,多数孢子萌发后产生芽管,直接以芽管侵入幼虫体壁,少数形成附着胞侵染寄主。

They were identified by 80 characteristics and clustered by numerical taxonomy. The isolates were clustered to Pseudomonas, Aeromonas, Vibrio, Bacillus and Alcaligenes.

鉴定结果表明,海湾扇贝附着基上异养菌属于假单胞菌、气单胞菌、弧菌、芽孢杆菌和产碱杆菌。

Banana fruit were inoculated by conidia suspension, conidia germinated within 6hr, produced dilutus appressoria within 12hr,produced dematiaceous appressoria within 24hr.

分生孢子在叶部、假茎与在果实上的发育过程相似,但假茎上暗色附着胞形成部位不象叶部和果实上那样局限于细胞间隙。

Lagenarium was higher 60%(for example, Glycyrrhiza yralensis, Lycium barberum and Desmodium styracifolium) as the concentration 0.01g/ml. Especially, the G. yralensis showed the strongest inhibitory effect on it with inhibitory rate of 99.6%. The inhibitory rate of 12 PE against mycelium growth and 15 PE against appressorium formation of C.

供试提取物在质量浓度为0.01 g/ml条件下,甘草、枸杞、金钱草等11种植物的80%乙醇提取物对黄瓜炭疽病菌分生孢子萌发的抑制率大于60%,其中甘草的抑制作用最强,达99.6%;12种提取物对黄瓜炭疽病菌菌丝生长的抑制率大于50%;15种提取物对黄瓜炭疽病菌附着胞形成抑制率达50%。

Results Microscopically, different degrees of hepatocytic degenerative changes and hyperplasic fibrous tissue and typical false lobule formation could be seen. Timm's staining result showed uneven distributed black granular deposits in the hepatocytes. No specific PAS staining was observed. Utrastructurally, the mitochondria were increased in volume and dramatically different in shape. The number of lysomes were increased.

结果 光镜下肝细胞表现为不同程度的退行性变,胶原纤维增生以及典型假小叶形成;Timm's染色阳性,发现不均匀分布黑色颗粒或团块样物质沉积;PAS染色则普遍缺乏特异性染色;超微结构显示线粒体形态多样,体积增大,溶酶体增多,粗面内质网管腔扩张,附着核糖体的脱颗粒,以及胞质水肿,质膜溶解。

The condium diluted with water or potato dextrose liquid medium could produce melanic appressoria in glass slide in 12 hours or in corn leaf surfaces in 24 hours.

在一定湿度条件下,影响玉米大斑病菌附着胞生长和发育的决定因素是温度。

While the tissue spaces surrounding a few blood vessels wasAl and Fg positive,no Al or Fg positive cells were observed.In antemortem injurygroup,diffuse subarachnoid hemorrhage,cerebral edema,swelling or pyknotic neu-rons could be observed.The axons showed irregular swelling and disconnection at1~3h,marked swelling and disconnection at 6h,and retraction ball at 15h whichwas more remarkable at 24h after injury.The space between myelin sheaths andaxons was increased at 3~6h after injury.Tortuous and wavelike myelin sheathswhich adhered on axons incompletely,or even peeled off could be found from 15hto 24h after injury.Perinuclear lysis of Nissl bodies began at 24h after injury.Thenumber of GFAP positive cells in cerebrum and brain-stem increased significantlyfollowed by decrease,and then increased again,but the time courses of the changesin different areas of brain were not same.Al and Fg positive neural cells,mainlysurrounded blood vessels,with diffuse or peripherally distributed positive matter incytoplasm could be observed at 0.5h after injury.The number of Al or Fg positivecells and the intensity of immunoreaction increased with the time of injury.The areaof SYN positivity in medulla oblongata and pons decreased notably 3~6h afterinjury,then return to normal levels and continued to 24h after injury.

生前损伤组,可见广泛蛛网膜下腔出血,脑组织水肿,神经细胞肿胀,晚期神经元固缩;伤后1~3h见部分神经轴突不规则增粗、断裂,伤后6h断端膨大,伤后15h可见收缩球,至伤后24h更为明显;伤后3~6h可见部分神经髓鞘与轴突之间的间隙增宽,伤后15h髓鞘明显曲折,不完全附着在轴突两侧,甚至剥脱,持续到伤后24h;核周尼氏体减少在伤后24h才开始出现;同一部位的GFAP阳性细胞数目随损伤时间发生改变,先增多(最早在伤后0.5h),达到高峰后减少,其后又有增多趋势,但不同部位的GFAP阳性细胞数目增减的时间过程不尽相同,同时,大脑中的GFAP阳性细胞数目也有改变;伤后0.5h,可在脑干组织中见到Al和Fg阳性神经细胞,主要位于血管周围,阳性物在胞浆中呈弥散性分布,但部分细胞的阳性物仅分布于靠近胞膜的胞浆中而呈环状,随损伤时间延长,阳性细胞数目增多,反应强度增加;伤后3~6h,延髓及桥脑中的SYN阳性物面积减少,其后恢复到正常水平,并持续到伤后24h。

Turgor pressure builds up inside the appressorium,forcing a penetration peg into the plant tissues;this is then followed by invasive growth and eventurally conidiation of the fungus.

该菌侵染循环过程经历了分生孢子与植物表面接触、萌发、附着胞分化、侵入栓的形成以及侵染生长,并最终产生新的分生孢子导致新一轮的病害循环。

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