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Cherry angiomas natural treatments are available, however, you must first learn the cause of cherry angioma.

樱桃血管瘤的自然疗法,可但是,你必须先了解的原因樱桃状血管瘤。

We reported 5 cases with intra-articular synovial hemangioma of the knee in order to improve diagnosis and treatment of intra-arfieular synovial hemangioma of the knee.

目的报告5例膝关节内滑膜血管瘤病例,以提高对膝关节内滑膜血管瘤的诊断、治疗水平。

The cervico-facial trunk of facial nerve in one case during the stripping operation was surgically injured and sutured immediately by the end to end anastomosis. Facial paralysis disappeared half year after the operation. These cases were followed up from 8 months to 4 years. Only one case had local recurrence.

在行耳廓血管瘤剥脱手术的8例中有1例在分离结扎腮腺区血管时误伤面神经颈面干,术中行神经断端缝合,半年后面瘫基本消失。8例行剥脱术治疗耳廓血管瘤的患者经8个月至4年随访,除1例可见局限性血管隆起外,未见复发。

Hemangioma model established in nude mice by hypodermically injection with human hemangioma endothelial cells retains all the biological characteristics of the human hemangioma and can be a good model for research on hemangioma.

皮下植入人血管瘤内皮细胞的裸鼠动物模型建模方法可靠,能较真实地反映人血管瘤的发展过程,是研究血管瘤的较理想的平台。

To sdudy the clinical and radiological features and the therapeutic efficacy of cavernous angiomas in central nervous system.

中枢神经系统海绵状血管瘤鲍伟民周良辅姜观富摘要目的:分析总结中枢神经系统海绵状血管瘤的临床和放射学特点及治疗效果。

EchT to treat hemangiomas of liver is safe and efficacy, with less traumatic and a quick recovery.

电化学治疗肝血管瘤具有安全有效、创伤小、恢复快等优点,可视为治疗肝血管瘤的新方法。

Our study shows that Bax ,the protein of accelerating apoptosis, may participate in the proliferation and regression of hemangioma. 2 The expression of TGFβ1 in the involuting phase was higher than in the proliferative phase, the difference was statistically significant, p<0.01. 3 This shows that Bax and TGFβ1 may be the important factors which cause the apoptosis of hemangioma and accelerate its regression as well.

1 Bax在血管瘤增生晚期开始表达,至退化期Bax的表达达到高峰,提示Bax这种促进细胞凋亡的蛋白参与血管瘤的增生与退化过程。2 TGFβ1在血管瘤退化期的表达高于增生期,差异有统计学意义,p<0.01.3 提示Bax与TGFβ1可能是引起血管瘤凋亡,进而促进其退化的重要因素。

It is suggested that CDK2 might progress the proliferation of endothelial cells in human proliferative hernangiomas; while p27(superscript Kip1) might inhibit the proliferation of endothelial cells in involuting hemangiomas.

p27(上标 kip1)能抑制血管瘤内皮细胞的增殖,在血管瘤的退化过程中起了重要作用;而CDK2能促进血管瘤内皮细胞增殖,在血管瘤的增生过程中起了重要作用。

The positive expression rates of Tissue Inhibitors of Metalloproteinase-2 protein in the cases of involuting groups were significantly higher than those in proliferating group and vascular malformation groups and normal skin group (P.05). Its difference was significant. Conclusions: Matrix MetallProtinase-2 and Tissue Inhibitors of Metalloproteinase-2 may play an important role in the pathogenesis of hemangiomas through angiogenesis. They may have no effect on vascular malformation and normal skin

血管瘤退化期,TIMP-2表达明显升高,说明 TIMP-2与血管瘤纤维化、消退有关,其机制可能是一方面通过抑制MMP-2介导的基底膜降解而在内皮细胞迁移、聚集中起作用,另一方面直接抑制内皮细胞的增殖及迁移,使内皮细胞高频率调亡,细胞外基质增多,组织出现纤维化,最终导致血管瘤的退化。2、血管畸形和正常皮肤小血管中MMP-2和TIMP-2不表达或表达较弱,说明血管畸形和正常皮肤小血管中并不存在异常的血管生成,据此可以对血管性疾病进行分型、分期,并指导临床治疗。

objective:to investigate the expression and significance of rb in the occurrence, development and regression of infantile hemangiomas.methods:the expression of rb was examined in the proliferative stage and catagen of human hemangiomas and normal skin tissues by using immunohistochemical technique.immunohistochemical technique for factorⅷ-related antigen was used to prove that the cells which expressed rb were endothelium.image analysis system was applied to measure the expression level of rb at different stages of hemangiomas and in normal skin tissues.results:the expression of rb was significantly lower in proliferating hemangiomas than that in involuting hemangomas(p.05).conclusion:rb might play an important role in the regression of human hemangioma endothelial cells and anti-angiogenesis.

目的:探讨rb蛋白在血管瘤发生、发展及退化过程中的表达状况及其意义。方法:采用免疫组织化学方法检测人皮肤血管瘤增生期、退化期及正常皮肤组织中rb的表达水平,并结合第ⅷ因子相关抗原的免疫组织化学染色证实表达rb的细胞是血管内皮细胞。利用计算机图像分析技术测量不同时期血管瘤组织和正常皮肤组织rb表达的积分光密度和面积。结果:增生期血管瘤内皮细胞rb表达水平低于退化期,差异有显著性(p.05),退化期血管瘤内皮细胞rb表达水平与正常皮肤组织相比,差异有显著性(p.05)。结论:rb通过抑制血管瘤内皮细胞增殖和血管生成而在血管瘤的退化过程中起重要作用。

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