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Pyemia is caused by making a sneak attack the body-wide inflammation response syndrome which the infection causes. And further development may cause multi-organ function barrier syndrome. How to prevent and reduce pyemia is important.

脓毒症是由于侵袭性感染引起的全身性炎症反应综合征，进步发展可导致器官功能障碍综合征怎样预防和降低脓毒症具有重要意义。

From occurrence and development mechanism, this article considers that the prevention and treatment for pyemia after severe empyrosis includes full body fluid anabiosis, early gastro-intestinal tract feeds, preventing intestinal tract endotoxin shifting, cutting the scab as soon as possible, eliminating necrosis, applying the antibiotic reasonably, immunity recuperation, maintaining the cellular immunity function, enhancing organism nutrition support, multi-organ function protection and improving organism prognosis.

本文通过对脓毒症的发生发展机制的阐述，认预防和治疗严重烧伤脓毒症全面的临床措施包括：快速、充分的体液复苏治疗和预防烧伤休克；早期胃肠道营养，以防止肠道内毒素移位；尽早切痂，清除坏死组织，封闭创面，合理应用抗生素，预防感染；免疫调理提高机体免疫能力，维护细胞免疫功能；加强机体营养支持治疗和多器官功能的保护，改善机体预后。

Accumulation of serous fluid in one or both pleural cavities.

积脓体内积有脓液，尤指在胸膜腔内

Some specifical genes or the variation of gene expression will directly influence the susceptivity and the prognosis of sepsis.

基因多态性与脓毒症易感性的关系的研究对于从基因水平上揭示脓毒症的发病机制和患者临床表现多样性的物质基础具有重要意义。

And then patients in treatment group (n=80) were treated by combination of high pressure oxygen and applying of qufushengji paste to raw surface or putting in vomica. patients in control group (n=76) were treated by applying of 0.5% iodophors gauze to raw surface or putting in vomica. changes of wound area were observed in two groups after nine days.

治疗组80例予祛腐生肌膏敷料敷于创面或填塞入脓腔直至底部并配合高压氧舱治疗，对照组76例以0.5%碘伏纱块或纱条湿敷创面或填塞脓腔。2组均治疗9日后观察伤口面积的变化，2周后观察临床疗效及愈合时间。

The disease, also known as serpiginous dermatitis with aseptic's acrodermatitis continua characterized by pustules, it was felt that with pustular psoriasis of the same disease.

本病又名匐行性皮炎连续性肢端皮炎以无菌性的脓疱为其特征，因此有人认为与脓疱型牛皮癣为同一疾病。

The disease, also known as serpiginous dermatitis with aseptic's acrodermatitis continua characterized by pustules, it was felt that with pustular psoriasis of the same disease.

本病又名匐行型皮炎连续型肢端皮炎以无菌型的脓疱为其特征，因此有人认为与脓疱型牛皮癣为同一疾病。

The inflammation reaction hyperplactic stage and lymphatic nodular stage are of nonspecific morphological changes and should be diagnosed with the help of positive tuberculin test and obvious increase of adenosine deaninase ; tuberculous nodular stage has a great number of epithelioid cells,and Langhans cells, caseous necrosis stage is characterized by a great deal of necrotic tissue and debris,a small number of fragmented epithelioid cells, and mainly by antiacid bacteria fibrinous hyperplastic stage is featured by a few fibrous tissue, cells and mucous oweing to hardness to get puncture, neans tuberculous restoration,and scar formation.

结核初期-炎性增殖期60例，占5.5%；结核早期-淋巴结节期130例，占11.9%；结核中期-结核性结节期有590例，占54.1%；结核晚期-干酪样脓样坏死期有280例，占25.7%；结核恢复期-纤维素增殖期30例，占2.8%。炎性增殖反应期非特异性形态学变化，需要结合结核抗体阳性和腺苷酸脱氨酶明显增高有助于诊断，结核结节期主要可有较多类上皮样细胞及郎罕氏细胞；而干酪样脓样坏死主要见大量坏死组织及碎屑、少数残碎不全类上皮样细胞，此期主要能查到抗酸菌为特点；纤维增殖期，抽出物难取，仅见少数纤维组织、纤维细胞和黏液间质为其特征，提示结核恢复、瘢痕形成所致。

Statistic indicates that half of the sepsis is caused by Gram-negative bacteria,and that lipopolysaccharide,which release from the cytoderm of Gram-negative bacteria,is considered to be the major molecule which is responsible for the endotoxin sepsis.

统计表明，临床半数的脓毒症是由于革兰氏阴性菌(Gram-negative bacteria)感染引起的，革兰氏阴性菌的细胞壁上的脂多糖(lipopolysaccharide，LPS)，或称为内毒素，被认为是革兰氏阴性菌引起脓毒症的主要作用分子。

Endplate potentials, acetylcholine potentials, and electrotonic potentials were recorded from the motor endplates of isolated diaphragms from acute late septic and nonseptic rats.

记录急性迟发性脓毒血症和非脓毒血症大鼠的离体横膈运动终板上的终板电位、酰胆碱电位和电紧张电位。

Since historical times,England ,where the early inhabitants were Celts, has been conquered three times .

从有历史以来，英国，在此地早期居住的是凯尔特人，已经被征服了三次。

Bluetooth OBEX File Transfer Enables the sending and receiving of files on your phone via Bluetooth.

蓝牙OBEX文件移动允许经过蓝牙传送和接受文件。。。。