粘附

4DRG was co-culture with sciatic nerve segment in 10鸖 DMEM;the axons were longer and surround the sciatic nerve segment which was regard as anew evidence for chemotropism.

结果：(1)在无血清条件下单独培养的DRG，背根神经节的轴突数目众多，外形纤细弯曲，不成束，并且施万细胞和成纤维细胞稀少，所以可以排除两者对轴突生长的影响，为观察来源于变性坐骨神经段的可溶性因子对轴突生长的作用提供了有利条件；(2)在无血清条件下DRG和变性坐骨神经段联合培养，①先单独培养DRG，4天后待神经元轴突长出，再与坐骨神经段联合培养，观察到神经元的轴突数目减少，外形挺直，部分轴突之间相互粘附成束；②变性坐骨神经段和DRG同时联合培养，神经元的轴突数目明显减少，外形粗壮，轴突之间相互粘附成束；(3)有血清条件下单独培养DRG，轴突数目较多，外形挺直，长短不一，部分神经元的轴突之间相互粘附，施万细胞和成纤维细胞数目众多，观察到的轴突生长情况受到施万细胞和成纤维细胞的直接或者间接的影响。

The method uses radiative immunity law to determine CV group, healthy group and content of the SIgA in taking secretion of bacterium group vagina, method of the conglutinate outside using system determines white beads bacterium is epithelial to the vagina (the conglutinate ability of VEC), the influence of VEC of conglutinate of white beads bacterium when observing SIgA standard.

方法采用放射免疫法测定CV组、健康组及带菌组阴道分泌物中sIgA含量，用体外粘附方法测定白念珠菌对阴道上皮细胞的粘附能力，观察sIgA水平时白念珠菌粘附VEC的影响。

So the Scripps Research team built a dendron structure that can bind to the DC-SIGN protein, preventing HIV from doing so, and which also mimics the sugar clusters that 2G12 binds to, prompting the immune system to produce destructive antibodies to the viral coat.

因此，Scripps的研究小组建立了一个能够粘附DC-SIGN蛋白的树突状机构，来阻止HIV与DC-SIGN蛋白粘附，这样也模拟了富糖类区域2G12的粘附，从而促使免疫系统产生对病毒蛋白外壳有破坏作用的抗体。

Methods Proliferation,cytotoxicity in vitro and expression of CD11 c of the cells cultured in the two different media were compared.To observe the tumor target cells killed by adˉherent natural killer cellsthrough electroscope.

分别用AIMV及完全培养基培养粘附NK细胞和非粘附NK细胞，比较细胞的增殖能力、杀伤肿瘤细胞的能力、表达粘附分子CD11 c 的能力，并通过电镜对A-NK细胞所杀伤的肿瘤细胞进行形态学观察。

Next, I detected the expression of E-cadherin, an important adhesion molecular mediating the adhesion among epithelia cells.

细胞间的粘附是由粘附分子介导的，E-cadherin是介导上皮细胞间粘附的重要粘附分子，其表达与功能的丧失与多种肿瘤的转移呈负相关。

The results show that the PSD fractal dimension of Light chestnut soil, Yellow cultivated loessial soil, Orthic dark locssial soil, Orthic cinnamon soil (stratified old manured loessial soil), Fluvo-aquic soil, Orthic gray desert soil, Orthic black soil, Orthic yellow-brown earth, Orthic yellow earth, Orthic ted earth and Orthic lateritic red...

结合上壤颗粒表面分形特性及土壤粘附的基本原理，探讨了土壤PSD分维对土壤—固体表面粘附行为的影响。PSD分维高的土壤与固体表面粘附力大，与固体表面发生明显粘附的起始含水量提高，发生明显粘附的含水量区间增宽。

Results:Berberine inhibited HUVEC adhesion for lymphocyte when pretreated HUVEC and antagonised IL-1 and TNF-induced upregulation of HUVEC adhesion for lymphocyte.Meanwhile,IL-1-stimulated lymphocyte adhesion to HUVEC was diminished in the presence of berberine.The main molecular mechanism of berberines function may be downregulating the expression of ICAM-1 on the HUVEC .

结果：小檗碱能抑制静息的及IL-1、TNF激活的内皮细胞与淋巴细胞间的粘附，其主要分子机制为下调内皮细胞表面粘附分子ICAM-1的表达；小檗碱能抑制IL-1激活的淋巴细胞与内皮细胞的粘附，对淋巴细胞表面粘附分子CD18的表达无影响。

Results:Berberine inhibited HUVEC adhesion for lymphocyte when pretreated HUVEC and antagonised IL-1 and TNF-induced upregulation of HUVEC adhesion for lymphocyte.Meanwhile,IL-1-stimulated lymphocyte adhesion to HUVEC was diminished in the presence of berberine.The main molecular mechanism of berberine's function may be downregulating the expression of ICAM-1 on the HUVEC .

结果：小檗碱能抑制静息的及IL-1、TNF激活的内皮细胞与淋巴细胞间的粘附，其主要分子机制为下调内皮细胞表面粘附分子ICAM-1的表达；小檗碱能抑制IL-1激活的淋巴细胞与内皮细胞的粘附，对淋巴细胞表面粘附分子CD18的表达无影响。

RESULTS:Berberine inhibited IL-1, TNF-induced HUVEC adhesion for PMN when pretreated HUVEC and antagonised IL-1, TNF-induced upregulation of ICAM-1 on HUVEC. Meanwhile, TNF-stimulated PMN adhesion for HUVEC and CD18 upexpression on PMN was diminished in the presence of berberine.

结果：小檗碱与IL-1或TNF共同处理HUVEC，能抑制IL-1、TNF诱导的PMN与HUVEC间的粘附增强，且能下调由IL－1、TNF诱导的HUVEC表面粘附分子ICAM-1表达的增高；小檗碱与TNF共同处理PMN，能抑制TNF诱导的PMN与HUVEC的粘附增强，亦能降低TNF诱导的PMN表面粘附分子CD18的表达。

Cellular adhesion molecule is a kind of glucoprotein expressed on the membrane surface of various cells,which mediate the conjunction and adhesion of cells and extracellular matrix.Vascular cellular adhesion molecule-1(VCAM-1) and intercellular adhesion molecule-1 (1CAM-1) are members of CAM.

细胞粘附分子(cellular adhesion molecule，CAM)是一类由多种细胞产生的，调节细胞和细胞间以及细胞和基质间相互结合并起粘附作用的膜表面糖蛋白，主要包括血管细胞粘附分子-1(vascular cellular adhesion molecule-1，VCAM-1)和细胞间粘附分子-1(intercellular adhesion molecule-1，ICAM-1)等。

I use an example quoted by Hu Jiaqi:"It is reported that America separates the DNA of a kind of virus by making use of genetic technology and combines it with another kind of DNA. Finally, they get a kind of virulent biological agent called a "pyrotoxin". Someone discloses in private that 20 grams of such a kind of biological agent could result in the global death of 6 billion people due to infection."

我在这里例举胡家奇所引用过的例子："据报道，美国利用转基因技术，将一种病毒的DNA分离出来，与另外一种DNA进行结合，拼结成一种剧毒的"热毒素"生物战剂，且私下有人透露，这种生物战剂只需20克，就可以导致全球60亿人全部感染死亡。"

Waiting, for the queers and the coons and the Reds and the Jews.

等待着疯子和黑人还有红色共产主义者还有犹太人