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superfusion相关的网络例句

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与 superfusion 相关的网络例句 [注:此内容来源于网络,仅供参考]

The increase in calcium current occurred within 1 minute of superfusion and the sustained inward holding current was slowly activated after prominent cell swelling.

钙电流的增加发生在灌流后1 min内,而内向性钳制电流在细胞明显膨胀之后缓慢激活。

By superfusion of ET-1, the hypoxiainduced decrease in RPF was remarkably potentiated (from 136±9 to 48±27 bpm) and the occurrence of pacemaker arrest was shifted to 20±6 min.

在缺氧的条件下持续灌流ET-1(1 nmol/L)20 min时,可显著加重缺氧所致的RPF降低,使起搏细胞的RPF由对照的136±9 bpm降至48±27 bpm(p.01),起搏细胞的停跳时间前移至20±6 min。

Intracellular pH change did not influence the actions of ketamine while superfusion of NMDA .

在pH 6.81的人工脑脊液中,ketamine抑制放电频率增快的药效减弱了35 %,在pH 8.1的人工脑脊液中,ketamine抑制去极化的药效减少了29 %,而胞内酸化或碱化则不影响ketamine拮抗NMDA的药效。

We compared the volume change of the plasmid control (C1), Bcl-2 wild-type (B6), and Bcl-2 mutant cells following superfusion with hypotonic solutions.

我们在低张溶液下比较了控制株C1、野生型B6 与突变型mI-2.4, mI-2.7 细胞的体积调控能力。

Hypotonic superfusion induced cell swelling resulted in the appearance of active channel.

采用膜片钳单通道(细胞贴附式和内面向外式)方法,记录急性分离的小鼠心肌细胞暴露于低渗溶液时出现的肿胀激活氯通道电流。

Results In hypoxia control group, the time for OPS to disappear was (186±16) seconds; HIP occurred in all slices within about 10 minutes of hypoxia and lasted for (113±16) seconds. When normal ACSF superfusion was restored, the rates of OPS recovery and OPS amplitude were 25% and (30±23)% respectively.

结果 缺氧组OPS消失时间为(186±26)s,在缺氧10min左右,均出现HIP,持续时间为(113±16)s;恢复供氧后,OPS恢复率和恢复幅度分别为25%和(30±23)%。

Then superfusion of celiac ganglia with Flavonoids (10~100ug/ml) would converted subthreshold PSP to action potentials, The effects

推测EGb和银杏黄酮对交感神经节细胞的作用可能与细胞当时所处的机能状态有关。

BACKGROUND:-Fish oil reduces sudden death in patients with prior myocardial infarction. Sudden death in heart failure may be due to triggered activity based on disturbed calcium handling. We hypothesized that superfusion with omega3-polyunsaturated fatty acids (omega3-PUFAs) from fish inhibits triggered activity in heart failure.

摘要翻译:背景:鱼油能够减少心肌梗死患者的猝死,心衰患者的猝死可能是由于钙调节机制的紊乱,我们假设以鱼来源的omega-3多不饱和脂肪酸(omega3-PUFAs)表面灌注能激活心衰患者的心肌细胞。

The whole cell-patch clamp recording technique was used to record I in specific pipette solution and superfusion according to specific holding potential and command potential program.

采用经典的酶分离心肌细胞方法,应用全细胞膜片钳技术记录I,观察MI后I、的变化,予以β2受体激动剂salbutamol、β受体激动剂异丙肾上腺素及不同选择性β受体阻滞剂后I的变化。

The process of cerebral hypoxia was simulated by giving superfusion with ACSFOGD for 14 minutes in the control and test groups. The propofol test group was subdivided into P1, P5 and P15 groups to undergo superinfusion with ACSF containing 1, 5 and 15μmolL^(-1) propofol respectively.

缺氧组:脑片用缺氧无糖的人工脑脊液灌流14min,实施缺氧过程;药物组分为P1、P5和P15组:在ACSFOGD灌流液中分别含1,5和15μmolL^(-1)的丙泊酚。

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