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While the β-catenin homologus Aardvark of Dictyostelium is absent, adherent junctions are lost. Aar also has a function in cell signaling and regulates expression of pre-spore gene.

盘基网柄菌β-连环蛋白同源物Aardvark的缺乏使细胞间失去粘著连接，Apr也有信号转导功能，调控了前抱子细胞基因的表达。

As a phytohormone, abscisic acid regulates many aspects of plant growth and development, such as seed dormancy, germination, vegetative growth, and environment stresses.

植物激素脱落酸对植物的生长发育具有多方面的调节作用，比如种子休眠、萌发，营养生长，环境胁迫反应等。

The New Law regulates that leasing expenses incurred for fixed assets under operating leases can be deducted evenly over the leasing period; where leasing expenses incur for fixed assets under finance leases, depreciation expenses shall be accrued on the portion of such lease which constitutes the value of fixed assets in accordance with relevant provisions, and shall be depreciated and deducted in installment.

新法规定，以经营租赁方式租入固定资产发生的租赁费，按照租赁期限均匀扣除；以融资租赁方式租入固定资产发生的租赁费，按照规定构成融资租入固定资产价值的部分应当提取折旧费用，分期扣除。

TLRs activation not only initiates innate immunity, but also regulates enhance antigen-specific acquired immunity, and thus associates innate and adaptive immunity.

TLRs能特异地识别病原相关的分子模式，不仅在激活天然免疫中发挥重要的作用，而且还调节获得性免疫，是连接天然免疫和获得性免疫的桥梁。

According to these findings, we suggest that PT-262 is a potential anticancer drug, which regulates the actomyosin contractility and migration inhibition mediating by Rho/ROCK/MLC pathway in the human lung cancer cells.

综合以上结果，我们推测PT-262是一种有潜力的抗癌药物，经由抑制Rho/ROCK/MLC路径，调控人类肺癌细胞肌动球蛋白的收缩和抑制癌细胞移行的作用。

Therefore, we suggest that it is mainly the reducing sugar, which sensitively regulates the amylase activity.

因此，认为对淀粉酶活性起灵敏调节作用的主要是还原糖。

Germany Medicine Law regulates: herb finished product must have the same quality, safety and standard of curative effect. The guide line of heavy metals, remain of pesticide and animalcule are determined in the import material. Chinese Traditional Medicine preparation belongs to the health foodstuffs in America and the harmful elements, such as mercury, arsenic, lead, and cadmium, are controlled severely. The status of Chinese Traditional Medicine is established in Japan, Korea and Singapore, and so on, but the import herbs must be determined.

德国药品法规定：草药成品药物必须符合与其它成品药物相同的质量、安全和疗效标准，进口原料药材需要检测重金属、农残、微生物等指标；中药制剂在美国被列为健康食品范畴，对有害元素砷、汞、铅、镉严格控制；日本、韩国、新加坡等亚洲国家，虽确定了中药的药品地位，但进口药材时法律要求进行等同于欧洲的有关重金属、农残、微生物等指标的法检。

One set projects to the basal forebrain and regulates EEG components of REM sleep, whereas the other projects to the medulla and spinal cord and regulates atonia during REM sleep.

一方面刺激基底前脑并调控快动眼睡眠的脑电波，另一方面刺激髓鞘和脊髓使其在快动眼睡眠时处于松弛状态。

The bodies have much greater potential to regulate spermatogonial stem cell proliferation and differentiation. The dosage of GFRα1 mRNA produced by undifferential spermatogonial cells regulates the signal transduction of glial cell-derived neurotrophic factor so as to regulates the direction of spermato gonial stem cells: at a low level, spermatogonoial stem cell favors differentiation; at a high level, spermatogonial stem cell favors self-proliferation.

GFRαl在小鼠生精恢复过程中以剂量相关性调控著胶质细胞源性神经营养因数资讯传人，从而调控著精原干细胞的去向：高剂量时诱导其增殖，低剂量时诱导其分化。

Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.

结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。

I use an example quoted by Hu Jiaqi:"It is reported that America separates the DNA of a kind of virus by making use of genetic technology and combines it with another kind of DNA. Finally, they get a kind of virulent biological agent called a "pyrotoxin". Someone discloses in private that 20 grams of such a kind of biological agent could result in the global death of 6 billion people due to infection."

我在这里例举胡家奇所引用过的例子："据报道，美国利用转基因技术，将一种病毒的DNA分离出来，与另外一种DNA进行结合，拼结成一种剧毒的"热毒素"生物战剂，且私下有人透露，这种生物战剂只需20克，就可以导致全球60亿人全部感染死亡。"

Waiting, for the queers and the coons and the Reds and the Jews.

等待着疯子和黑人还有红色共产主义者还有犹太人