查询词典 modulation

Because the powerline is a complicated medium, selecting appropriate modulation means is a pivotal question in order to guara- ntee the quality of signals.

因为电力线是一种复杂的通信媒体，要保证通信质量，选择合适的调制方式是一个关键问题。

The intensity modulation method is the oldest and simplest method for fiber-optic sensor, in which a homochromatic laser is used as system light source.

强度法是干涉型传感器采用的最古老、最简单的方法，它采用单色激光光源作为系统的光源。

Fiber optic sensor ; phase modulation ; demodulator ; passive homodyne method ; Labview

光纤传感器；相位调制；解调；无源零差法； Labview

Dynorphin may act as a pronociceptive, not an antinociceptive, factor in the modulation of the spinal hyperalgesic state.

在脊髓痛过敏状态的调制中，强啡肽是作为一种致痛因子而不是抗痛因子起作用。

OT is involved in the modulation of the transmision of nociceptive information in the spinal cord of rats with sciatic nerve ligation Intrathecal administration of OT induced a significant analgesic effect during 1 to 10 day after sciatic nerve ligation and a significant hyperalgesic effect from 11 day after sciatic nerve ligation .

催产素参与神经损伤痛敏大鼠脊髓水平痛觉信息传递的调节在坐骨神经损伤性痛大鼠鞘内注射催产素对痛觉的影响呈现先镇痛后痛敏二相性，转折点是结扎后第11天。

GABA-ergic neuron may involve in the modulation of acetylcholine releasing in spinal cord and in the thermoregulation in preoptic area and anterior hypothalamus,which may lead to the TPM related hypohidrosis.

4GABA能神经元可能参与节前胆碱能神经元调节、GABA能神经元参与丘脑下部对高温的反应。这种参与活动可能与TPM泌汗障碍相关。

Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.

结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。

In the application of the OFDM system, combined with the adaptive modulation, adaptive coding and the receive diversity based on the accurate estimation of the characteristic of the subchannels, we can increase the spectrum usage, reduce the transmission power, the intercarrier interference and the peak-to-average power rate. So the system performance is optimized.

在OFDM系统的应用中，在对各子信道特性的准确快速估计的基础上，通过对各子载波进行自适应调制，自适应信道编码，以及和接收端的分集接收相结合，能够提高频谱利用率，降低发射功率，抑制子载波间干扰和降低功率峰均比，从而能够使系统的性能达到最优。

Single-phase Sinusoidal Pulse Width Modulation Inverter Power Supply in this paper belongs to AC power supply (AC-DC-AC convert). Control mode adopts feedback of voltage control. The methods of intermitting power flux and changing duty-cycle can change pulse width of drive voltage that adjust and rectify output voltage ultimately.

本文设计的单相脉宽调制逆变电源属于交流电源，采用电压反馈控制，通过中断功率通量和调节占空比的方法来改变驱动电压脉冲宽度来调整和稳定输出电压。

Of them 25 cases of oral cancer (G1-2) were randomly divided into trail and control group . The remained 11 cases of nasopharyngeal and maxillary sinus cancer (NPC-MSC, mainly G3-4) were attributed to a third group (1st course: sBRM, then 2nd course: IAC). The order of drug-use of sBRM is: pre-immunomodulation of the reactivity of patient low-dose CTX and PolyI∶C, then in- juring tumor and thereby modulation of its immunogeneity (DDP-PYM regimen , thereafter, reaction enhancement modu- lation [low-dose rhuIL-2, Cimetidine and Indomethacin ] . All drugs, except for CTX, PolyI∶C and Indo, were fractionally delivered in a manner of continously infution in 8-10 hours via a retaining intra-external carotid artery catheter CAVAFIX 335 SPLITTOCAN G14, B.

将其中25例晚期口腔高、中分化鳞癌病人随机分为实验治疗（12例）和对照治疗（13例）两组，实验组采用sBRM疗法，具体用药为：先用小剂量环磷酰胺和聚肌胞对机体反应性作预调变，然后用低剂量DDP-PYM化疗杀伤&致创&癌细胞，使其免疫原性增强并借之诱导肿瘤浸润淋巴细胞，化疗结束后续用小剂量rhuIL-2激活和扩增TIL，特别是其中的T细胞，并借助小剂量西咪替丁、消炎痛调节机体以原发灶为中心的癌受创相关反应。

When this condition occurs, inbound replication with the source partner is stopped on the destination domain controller and event ID 2042 is logged in the Directory Services event log.

计算机密码学是研究计算机信息加密、解密及其变换的科学，是数学和计算机的交义学科，也是一门新兴的学科。

Instructions: click on the thumbnails to see a larger image, then use the left-right arrow keys to scroll through the slideshow.

使用说明：滑鼠点在小图上即可放大观赏。开启后键盘左右键可用来换照片。