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inter nos相关的网络例句

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与 inter nos 相关的网络例句 [注:此内容来源于网络,仅供参考]

Mehods: After making the AD models by Aβ(subscript 1-42), draw the whole brain or hippocampus of rats in ordinary group, model group, traditional Chinese Medicine prevention group, traditional Chinese Medicine treatment group and positive control-group, and detect the biochemical marks of T-AOC, MDA, NO, NOS, chAT, TChE.

用凝聚态Aβ(下标 1-42)制备出AD模型后,取正常组、假损伤组、模型组、中药预防组、中药治疗组、阳性药物对照组各组大鼠的全脑或海马检测T-AOC、MDA、NO、NOS、chAT、TchE等指标。

Results: After torsion NO and NOS activity increased greatly in both side, and showed obviously more in the torsive side; The percentage of monoploid significantly decreased in the B group ,verapamil could reduce the extent of these changes.

结果:B组单倍体细胞占细胞总数明显下降,而且睾丸组织NO含量、NOS活性升高,维拉帕米可减轻上述变化。

(1)SCG is a sensitive, simple and economic method in evaluating the cytotoxicity of SM in toxication and the degree of intoxication, and with good prospects in early diagnasis;(2)The cytotoxicity caused by the elevation of NO induced by the NOS activity after intoxication might be one of the mechanisms of SM intoxication;(3)Apoptosis participates in the process of SM intoxication also might be one of the mechanisms of SM intoxication.

(1)SCG法敏感、经济、简便,可用于硫芥中毒细胞毒性、中毒程度的评价,在早期诊断上有良好的应用前景;(2)中毒后因NOS的活性升高导致NO的升高而产生细胞毒作用可能是硫芥的中毒机制之一;(3)细胞凋亡参与了硫芥的中毒过程,亦可能是其中毒机制之一。

Results NO and NOS were higher after taking the drug than before taking it, and there was a significant difference between them (P.01). Conclusion Sildenafil can reinforce the ability of anti-oxidation and have an important effect on the recovery of cellular function and inner environmental tranquilization.

结果 海拔3700m和5380m负荷运动,实验组较对照组NO和NOS增高,有非常显著性差异(P.01)结论西地那非可明显改善高原机体运动抗氧化能力,对细胞功能恢复、内环境的稳定有重要作用。

Kangdai Ⅰ has protective function to the damaged neurons and astrocytes: Main results:(1) It has direct protective function to the damaged neurons. It can increase the activity and survival rate, decrease the mortality and the transudation rate of LDH in cultured medium and the strong positive cell count of NOS expression of injured neurons.(2) It has also directly protective function to the damaged astrocytes. It can increase the activity and survival rate and protein content in conditioned medium.(3) It can strengthen the ability of BDNF, GDNF, bFGF, HSP and IL-6expression in damaged astrocytes.(4) It can also strengthen obviously the expressions of NSE, bFGF-receptor and bc1-2, lower the expression of bax and caspase-3.(5) It can indirectly protect and restore the damaged neurons by astrocytes. Because the effect of ACMK (ACM interfered by Kangdai Ⅰ) is stronger than ACM+K (ACM associated with Kangdai Ⅰ).

抗呆Ⅰ号对受损的神经元和星形胶质细胞均具有保护作用:主要表现为:(1)对受损神经元具有直接的保护作用,可提高受损神经元的活性和存活率,降低细胞培养液LDH的漏出率、细胞死亡率和NOS染色强阳性细胞的表达量;(2)对受损的星形胶质细胞也有直接的保护作用,可提高其活性、存活率以及培养液蛋白质的含量;(3)能增强受损星形胶质细胞分泌BDNF、GDNF、bFGF、HSP和IL-6的能力;(4)可明显增强受损神经元对NSE、bFGF的受体和bc1-2的表达,降低受损神经元对bax和caspase-3的表达;(5)抗呆Ⅰ号可通过星形胶质细胞间接地保护和修复受损的神经元,因为在多数实验组中经抗呆Ⅰ号作用的ACM的作用远大于ACM与抗呆Ⅰ号联合应用的作用,统计学上具有显著性差异。

In this study, we examined this point in mice deficient in all 3 NOS isoforms (triply n/i/eNOS-/- mice) that we have recently developed.

本试验中,我们使用最近培育出的缺少三种一氧化氮合酶亚型的小鼠(triply n/i/eNOS-/- mice)以阐明体内一氧化氮的作用。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

In Christ and extra nos lies "a fountain, open to us, from which we may draw what otherwise would lie unprofitably hidden" [6]; but, the fountain is profitable only for that sinner who receives it to himself by "drink his fill."

在基督里,在我们之外,&有一个源泉,已经向我们开启,我们可以从那里汲取那原本是隐藏的、无用处的&(注6.3.11.9);但是,那源泉是只有当罪人靠&喝到饱足&,领受到他里面,才会有益处的。

In summary, we identified firstly that existence and distribution of JA and NOS in guard cell; presumed that the direct transport of JA from unwounded tissue to wounded site could account for the increase of JA after leaf wounding, and involved stomatal closure, and proposed a possible pathway of JA involved in regulating stomatal movemnts as follows: The production of NO stimulated by JA triggers the increases of cytosolic Ca〓 concentration, subsequently activate other downstream reactions and eventually induces stomatal closure.

本研究中我们首次确认了气孔保卫细胞中JA和NOS的存在和分布;伤引起的JA运输和分配的变化可能参与对气孔运动的调控,并提出JA调控气孔运动信号转导的一条可能途径:JA引起NO含量的增加,经第二信使Ca〓的作用及其下游事件,最终诱导气孔关闭。

Results AE from CM had vasodilating effect on the contracti on of mesentery blood capillary in guinea pigs and the contraction of rat thoracic aortic ring induced by l0μmol/L phenylephrine, even under the condition of pre-treating with glibenclamide (1μmol/L) or propranolol (3μmol/L). When the endothelium of the rat thoracic aortic ring was removed, AE from CM had not vasodilating effect hut constrictive effect. AE from CM could also increase the contents of NO, NOS and cNOS in the aortic tissue and had no obvious effect on the NO content in plasma and iNOS in aortic tissue of rats.

结果 桑白皮丙酮提取物有抑制去甲肾上腺素收缩豚鼠肠系膜微血管的作用,抑制苯肾上腺素(10μmol/L)收缩离体大鼠胸主动脉环的作用,预加优降糖或普蔡洛尔(3μmol/L)后仍然有松弛作用;大鼠胸主动脉环去内皮后药物无松弛作用,反而起收缩作用;显著升高血管的NO和NOS、cNOS含量,对大鼠血浆的NO和血管iNOS的含量影响不大。

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