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The direct signs of Pulmonary embolism were luminal constriction,emphraxis,branched decreased,intraluminal lowD filling defect of blood vessel;while the indirect signs,formation of pulmonary infarction focus,pleural effusion,pneumonia,atelectasis,pulmonary dilatation and hydropericardium,etc.

肺栓塞的直接征象为管腔狭窄、阻塞、分支减少及血管腔内的低密度充盈缺损,间接征象为肺梗死灶形成、胸腔积液、肺炎、肺不张、肺动脉增宽、心包积液等。

The direct signs of Pulmonary embolism were luminal constriction,emphraxis,branched decreased,intraluminal low??D filling defect of blood vessel;while the indirect signs,formation of pulmonary infarction focus,pleural effusion,pneumonia,atelectasis,pulmonary dilatation and hydropericardium,etc.

肺栓塞的直接征象为管腔狭窄、阻塞、分支减少及血管腔内的低密度充盈缺损,间接征象为肺梗死灶形成、胸腔积液、肺炎、肺不张、肺动脉增宽、心包积液等。

Studies of empyema incidence conducted after the introduction of PCV7 showed contradictory results: Empyema incidence was reported to have increased by 88% in Utah and by 400% in California but was shown to have decreased by 55% in Texas and remained unchanged in Quebec, Canada.

引进PCV7后进行的肺脓疡发生率研究显示不一致的结果:据报导,肺脓疡发生率在犹他州增加了88%,在加州增加了400%,但在德州却下降了55%,在加拿大魁北克则是没有变化。

Thunderous is empyreal: Call is empyreal the target inside big range of the whole body of oneself of thunder electrical shock, cause successive harm for many times, can make condition of the many corona that be immersed in by the target of attack, floating rate is decreased greatly.

雷动九天:召唤九天雷电击自身周身大范围内的目标,造成连续的多次伤害,并可以使得多个被攻击的目标陷入电晕状态,移动速度大减。

S Grace of the Empyrean Lord recharge decreased from 5 to 3 min.

主神的恩宠CD时间由5分下调到3分。

Cleric□s Grace of the Empyrean Lord recharge decreased from 5 to 3 min.

主神的恩宠CD时间由5分下调到3分。

Meanwhile the function of antioxidant defense system increased to enhance the ability for clearing free radical and antagonized the damage to organism from free radical. So the activity of GSH-Px and T-AOC enhanced. But the compensative ability was limited and the activity of GSH-Px decreased abruptly and the T-AOC was not increased further in cerebellum of chicken with encephalomalacia. This showed that it existed the exhaustion of GSH-Px in cerebellum of chicken with encephalomalacia.

在体内自由基蓄积的同时,机体抗氧化系统功能出现代偿性增强,提高自由基的清除能力,以对抗自由基对机体的损伤,表现为GSH-Px活性和T-AOC的增强,但机体的代偿能力是有限的,发病雏鸡小脑中GSH-Px活性的突然降低和T-AOC的不再增高,表明GSH-Px在发病雏鸡小脑中存在耗竭现象。

The content of Fe and Cu increased and the content of Zn decreased, the change of the content of Se was consistent with the change of activity of GSH-Px and the content of Ca and Co did not changed in chicken with encephalomalacia The change of Ca and trace element was all relative to the mechanism of free radical The change of Fe ion status showed that Fe acted as medium in the progress of synthesizing free radical.

脑软化雏鸡体内铁、铜含量增高,锌含量降低,硒含量的变化与GSG-Px活性的变化相一致,而钙、钴、锰的含量没有变化。

ARID (AT-rich interaction domain) protein is a transcription factor family in higher eukaryotes that regulates cell proliferation, development, and differentiation. Specificity of DNA binding ability in this family prefers AT-rich sequences, but some ARID family proteins are not sequence-specific DNA-binding proteins or they do not bind AT-rich sequences. We found two genes that encode ARID in Giardia lamblia genome database, garid1 and garid2. We analyzed the function of garid1 first. AU1-tagged gARID1 was found to localize to nuclei. During encystation, gARID1 mRNA level decreased emphatically, but protein level increased. We also found that gARID1 can bind AT-rich initiator of the cwp1 promoter by EMSA. Mutation analysis revealed gARID1 binding sequence was AGATC and AATAAAATA. We used ChIP to demonstrate that gARID1 can bind cwp1 gene promoter in vivo.

ARID(AT-rich interaction domain)蛋白质家族是真核生物的一种转因子,在许多同种的真核生物有它的同源基因,这个家族的蛋白质通常与调控细胞的生长、发育和分化的作用有关,而这个家族的蛋白质和DNA的结合能,各种ARID蛋白质的专一性尽相同,过大致上偏好於和AT-rich的序结合;我们已经在形鞭毛虫的基因组中找到个含有ARID 的基因,分别是garid1和garid2,我们首先对於garid1做分析;将AU1标记接到gARID1转染形鞭毛虫,用免疫萤光染色可发现gARID1存在於细胞核中。gARID1的讯息RNA在囊体化后会明显下,过其阳性染色和蛋白质表现有明显增加;EMSA实验中也发现gARID1会明显的与cwp1基因启动子之AT-rich initiator结合,经由突变序分析,也显示gARID1的结合序为AGATC和AATAAAATA,随后我们也用ChIP证明gARID1在细胞内也的确会和cwp1基因的启动子结合。

In August 2002, endocrinologist Stephen Bloom and colleagues at Imperial College London showed that when they injected PYY3-36 into rodents and humans, it decreased hunger for 12 hours or more.

2002年8月,伦敦帝国学院的内分泌学家Stephen Bloom和同事们展示了他们的研究结果。他们将PYY3-36注入啮齿动物和人体内,发现它能够在12个小时甚至更长时间内抑制饥饿的感觉。

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