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cardiac fissure相关的网络例句

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与 cardiac fissure 相关的网络例句 [注:此内容来源于网络,仅供参考]

[Objective]To detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration on injury.

[目的]探讨Na+/H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧/复氧损伤的保护机制。

Methods]By observing the cytoplasmic Ca2+ concentration change of single cardiocyte in I/R status and the effect of Na+/H+ permutoid retarder to Ca2+ concentration in different phases (diabetes pure oxygen-lack deficit group called DM group,the oxygen deficit/reaeration on entire journey for medicine group called DM-EIPA group, the first group giringmedicine before reaeration on called DM-EIPA 1 group, and the second group giringmedicine before reaeration on called DM-EIPA 2 group) to detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration injury.

方法] 通过观察糖尿病鼠单个心肌细胞在缺氧/复氧时细胞胞浆Ca2+浓度的动态变化以及Na+/H+交换体阻滞剂在不同时相(糖尿病单纯缺氧组即DM组,缺氧/复氧全程给药组即DM-EIPA组,复氧前给药1组即DM-EIPA 1组,复氧前给药2组即DM-EIPA 2组)对Ca2+浓度的影响来研究Na+/H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧/复氧损伤的保护机制。

Furthermore, the limiter not only improved the recording quality but also protected the cardiac electronic equipment because overdue stimulation signal was limited at a given electronic level during the recording of transesophageal pacing.

此外,在经食管心电生理的记录中,过大的刺激脉冲也因实施了限幅技术而被限制在特定的电平内,不仅明显提高了记录质量,而且也保护了心电记录设备。

Conclutions: Under ischemic condition, the decrease of Iks could prolong the mid-late repolarization phase of APD. It could cause EAD and then arouse TdP. Because the decrease of M cell was significantly greater than that of sub-endocardial myocytes and sub-epicardial myocytes, it could increase TDR. Cardiac arrhythmias could last due to reentrance that secondary the increase of TDR.

缺血时,心肌细胞的Iks电流减弱,使得细胞动作电位复极中后期延长,容易出现EAD并引发TdP;由于M细胞的Iks电流减弱程度明显大于心内、外膜细胞,造成心室跨壁复极离散度增加,有利于折返的发生,使得心律失常得以持续。

Objective To study ischemic effects on reentrant activities and cardiac arrhythmias using a computational approach.

目的 利用计算机仿真手段,研究缺血对折返波活动状态的影响及其与心律失常的关系。

The effect of DM on 30-day cardiac outcome was assessed by multivariate logistic regression and the effect on long-term survival was assessed by Cox regression analysis.

通过多元回归分析围术期30天内 DM 对心输出量的影响,通过 Cox 回归分析评估 DM 对其术后长期生存的影响。

Logistic regression was used to estimate the association between excessive bleeding at the first and second cardiac procedures.

使用 Logist 回归分析第一次和第二次心脏手术大量出血之间的关系。

Multiplicity showed that regular exercises were the independent factor of death and rehospitalization within 1 year of chronic heart failure patients with the exception of sex, age, cardiac functional grading, ejection fraction, complication and treatment.

多因素分析显示,排除性别、年龄、心功能分级、射血分数、有无合并症及治疗情况等干扰因素后,规律运动锻炼是预测慢性心力衰竭患者1年内死亡和再住院的独立因素。

Cardiac enlargement was detected in 60% patients with moderate and 100% patients with severe aortic regurgitation on echocardiography.

在21例轻至中度主动脉瓣返流的患者中,胸片提示1例患者(5%)的心脏扩大,而心脏超声则提示7例患者(35%)左室扩大。

At 30mg/kg there is the best effective and induced the heart rate to be reduced by12.8%, systolic pressure and diastolic pressure by 12.5% and 16.3%.At the same time the cardiac max contractility and relaxtion have also been reduced by 12.5% and 16.3%.

在30mg/kg剂量时达到最佳作用效果,可使心率降低12.8%,动脉收缩压和舒张压各降低12.5%和16.3%,同时心肌最大收缩和舒张速率也分别降低了34.3%和39.4%,明显降低了高血压对心脏的压力。2、Rb1能够剂量依赖的降低L-NNA导致的慢性高血压模型的血压,并且对高血压心肌舒张功能障碍起到保护作用。

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