门静脉的

基本解释 (translations)

pylic

与门静脉的相关的网络例句 [注：此内容来源于网络，仅供参考]

Oeeki Y reported a method of partial arterialization of portal vein with ileo-colon arteriovenous anastomosis in 1997, he thought this can prevent theliver failure caused by embolism of hepatic artery after liver operation.

但过量动脉血(门静脉内动脉血灌流量达到门静脉正常血流量的一倍以上时)灌流肝内门静脉则对肝脏有害，如导致肝内门静脉分支增宽，内膜纤维变性，以及肝细胞微粒体酶活力的改变。

Objective To assess the ability of CTA for displaying the portal vein.

目的 评价螺旋CT门静脉成像对门静脉的显示能力。

Results Along with the formation of liver cirrhosis and PHG, the gastric mucosa became thicker and more congestive. The portal pressure elevated gradually, followed by the decrease of endothelin-1 levels in both the peripheral and the portal blood. ETA mRNA expression was significantly elevated in the postcapillary venule and the precapillary arteriole, and predominantly in the latter.

结果：Wistar大鼠在肝硬化门静脉高压形成过程中，其胃黏膜厚度增加，黏膜淤血；门静脉压力呈梯度性升高；外周及门静脉血浆ET水平随门静脉压力升高而降低；胃黏膜ETA mRNA主要在黏膜基底部的微循环前微动脉和后微静脉血管壁表达，其表达水平随ET浓度的降低和门静脉压力的升高而增加，且其在小动脉表达明显强于小静脉。

Radiography of veins or a vein after injection of a radiopaque substance.

门静脉门静脉的，门静脉系统的，与门静脉系统有关的

MAIN OUTCOME MEASURES: Occurrence of branches of hepatic portal vein; clearness and satiation of the vessel wall.

主要观察指标：门静脉的各级分支的出现以及管壁清晰度、饱满度。

And There was no serious complication;②In the course ofinterventional therapy, Direct portal vein angiography demonstrated vena coronaria ventriculi(100%)andgastricveins(65.26%)andvenagastricaposterior38.43%, Angiography demonstrated venacoronariaventriculi communicate esophagus varicose veins, gastric veins and vena gastrica posterior communicategastric varicose veins. vena coronaria ventriculi had only a small percentage of double vein, about30.57%. The sites of vena coronaria ventriculi arising from the portal vein, splenic vein, portosplenic junction, were found in 52.06%、27.39%、20.55% respectively.③12 extrahepaticprotosystemic shunts were found in these patients. Include gastro-nephrosshuntof 3 cases, 7 caseswere splenetic- nephros shunt and 2 cases shown recanalization of umbilical vein .④The averageportal pressure before and after the procedure were 3.87±1.82kPa and 3.64±1.14kPa in 73patients, but to the time of rebleeding, it was 3.96±0.23kPa in the 11 cases.⑤There werethree kinds of variceal outcome: disappearance (54,low degree (19).⑥Spearman logisticanalyse and ANOVAtest shown liver function class, variceal degree of the splenic necrosis area,the blood direction in portal vein before operation and remain small collateral routes were thesignificant factors concerning outcome of varices.⑦The bleeding volume and portalhypertensive gastropathy are main risk factors of rebleeding.⑧The course of livercirrhosis is the risk factor of survival and extrahepatic portosystemic shunt , fine varices are thebeneficial factors to survival.⑨During all cases'followed-up data, the 1, 2, 3, 4, 5 yearcumulative survival rates and rebleeding rates were 17.81%, 28.77%, 38.36%, 43.84%, 47.95%and93.15%,91.78%,86.30%,83.56%,80.82%respectively. Conclusion The interventional disconnection treatment for liver cirrhosis and portalhypertension was designed suitability. It rapidlycontrol bleeding,butpressure of portal vein was notobvious high, perfusion was not low .it was compared with surgery therapeutic that interventionaldisconnection treatment was safe and had a significant clinical effect to hemorrhage and preventfrom rebleeding.

结果：①术后一过性发热62例(84.9%)，腹痛腹胀48例(65.8%)是介入断流术常见的并发症，未发生严重并发症；②门静脉造影显示胃冠状静脉、胃短静脉和胃后静脉的曲张分流的出现率是100%、65.26%和38.34%；显示食管静脉曲张主要由胃冠状静脉供血，胃静脉曲张主要由胃短静脉和胃后静脉供血；胃冠状静脉大多数为单支，少数为双支，其双支的出现率分别为30.57%；胃冠状静脉开口于门静脉主干的为52.06%，开口于脾静脉主干的为27.39%和开口于门脾静脉交汇处的为20.55%；③发现胃肾分流3例，脾肾分流7例、腹膜后门腔静脉分流2例，以及CTA检查发现脐静脉开放者2例；④73例患者介入断流术前和术后平均自由门静脉压力分别为3.87±1.82kpa和3.64±1.14kpa，前后比较存在显著性差异；11例再次介入手术患者的术前、术后和复发后的自由门静脉压力分别为4.02±0.24kpa、3.82±0.25kpa和3.93±0.23kpa ，前后比较发现首次术前与术后存在显著性差异，首次术前和复发出血术前门静脉压力比较无显著性差异；⑤介入术后复查曲张静脉转归基本消失54例，轻度19例；⑥Spearman相关分析和Logistic多因素回归分析，肝功能分级、静脉曲张程度、门脉血流方向和残存小侧支四个因素对曲张静脉转归有影响；Spearman相关分析和Logistic多因素回归分析门脉高压性胃病和出血量等因素对复发出血时间有影响；⑦COX回归分析，门体分流和曲张静脉转归两个因素对术后生存有影响；⑧术后随访6-70月，1、2、3、4、5年的累计复发出血率和累计生存率分别为17.81%、28.77%、38.36%、43.84%、47.95%和93.15%、91.78%、86.30%、83.56%、80.82%；结论：介入断流术治疗门脉高压食管胃底静脉曲张有独特的优点，可以快速直接控制曲张静脉出血而门静脉压力无显著增高，保证了肝脏灌注；与外科分流术相比适应证广、损伤轻、术后恢复快，不易遗漏曲张静脉；肝功能分级、曲张静脉程度、门脉血流方向和残存侧支血管对食管胃曲张静脉转归有影响；门脉高压性胃病对复发出血时间有影响；门体分流和曲张静脉转归对生存时间有影响。

The first to tertiary ramification of portal vein and their significance. The first order branch of portal vein had four patterns: namely, normal branching pattern, 90 cases (90%); trifurcation, 5 cases (5%); independent branching of right posterior branch from main portal vein, 4 cases (4%); and complicated branching pattern, 1 case (1%).

门静脉第一到第三级分支的解剖和变异门静脉第一级分支有四种类型，即正常分支型，90例（90％）；右前支、右后支和左门静脉同时分支型，5例（5％）；右后支独立分支型，4例（4％）；及复杂分支型，1例（1％）。15例（15％）右门静脉不形成单独右后支，而是右后下支P6和右后上支P7分别从右门静脉分出，这种变异并非少见而文献中却很少描述。

Methods A retrospective analysis was carried out in 245 cases with portal hypertension treated with PCDV or PSRS plus PCDV. The diameter, blood flow velocity and blood flow of portal vein, splenic vein and superior mesenteric vein were measured by magnetic resonance angiograph at 1 week preoperatively and at 2 week postoperatively.

回顾性总结近8年采用脾肾分流加门奇断流联合手术和门奇断流术治疗门静脉高压症245例，于术前一周和术后2周应用核磁共振血管造影测量门静脉、脾静脉和肠系膜上静脉的直径，流速和流量，并于术中动态测量门静脉压力。

Methods: For 56 cases of arterial bleeding, performed angiography in the artery which was suspected to be the bleeding one, and then infused the embolus into the bleeding after super-selection to embolize it. For 17 cases of esophageal veins and fundic veins bleeding caused by the higher blood pressure in portal vein, we performed angiography in portal vein stem through puncturing the liver, found the bleeding spot, then infuse the embolus into the bleeding veins through super-selection to stop bleeding.

56例动脉性出血采用Seldinger氏法，经股动脉穿刺将导管选择插入可疑出血的动脉内先行DSA检查，明确诊断，再超选择插入出血动脉内注入栓塞材料，栓塞出血动脉。17例门静脉高压致食管胃底静脉曲张破裂出血，则采用经皮穿肝至门静脉主干行门静脉造影，明确出血部位，再超选择插管至出血静脉注入栓塞材料进行栓塞治疗。

This operation is suitable for the following portal hypertension: simple occlusion of hepatic vein (a pathological type of Budd-Chiari syndrome)thrombosis in portal vein or prehepatic portal hypertension due to cavernous transformation; intrahepatic portal hypertension with rebleeding after treatment of splenectomy or without operation, and the patients with liver function in Child A or B class.

本手术主要适应于以下原因引起的门静脉高压：①单纯化肝静脉闭塞(布-加综合征的一种病理类型)；②门静脉主干血栓形成成海绵样变性引起的肝前性门静脉高压；③肝内型门静脉高压行断流术后再发出血或未行手术，肝功能为Child A和B级者。

与门静脉的相关的网络解释 [注：此内容来源于网络，仅供参考]

portal hypertension：门静脉高压

门静脉高压(portal hypertension)是一组由门静脉压力持久增高引起的症候群. 绝大多数病人由肝硬化引起,少数病人继发于门静脉主干或肝静脉梗阻以及一些原因不明的因素. 当门静脉血不能顺利通过肝脏回流入下腔静脉就会引起门静脉压力增高.

portal hypertension：门静脉高压症

门静脉高压症(portal hypertension)是肝脏及其有关血管和胆管疾患所引起的综合征. 主要表现为充血性脾肿大和脾功能亢进、腹水、门体分流性肝性脑病,食管胃底静脉曲张和门静脉高压性胃炎以及两者引起的上消化道大出血. 后者作为临床急症,

portal vein：肝门静脉

在正常人中,食道及胃的静脉血管是经肝门静脉(Portal Vein)跑到肝脏的. 当肝脏因肝炎或其它因素导致肝硬化时,肝门静脉血管内的血压便会上升. 正常肝门静脉血压是3mmHg,当它上升到12mmHg时,食道及胃的静脉便慢慢地曲张,最后会破裂而出血,

hepatic portal vein：肝门静脉

从肠壁返回的血液由毛细血管网集合成肠下静脉,尾静脉的部分血液也注入其中;肠下静脉前行至肝盲囊处血管又形成毛细管网,由于这条静脉的两端在肝盲囊区都形成毛细血管,因此称作肝门静脉(hepatic portal vein).

renal portal vein：肾门静脉

尾和后肢的静脉在前行中分为两对,一对沿肾脏的外缘成肾门静脉(renal portal vein),进入肾脏,分成许多细小血管,再次汇集成数条肾静脉(renal vein),由两肾之间通出,与来自生殖腺的生殖腺静脉(genital vein)一起,将血液送入后大静脉;

periportal：门静脉周的

peripolesis /周边运动/ | periportal /门静脉周的/ | periposeidon /近海王点/

pylethrombosis：门静脉血栓形成

pylethrombophlebitis 门静脉血栓静脉炎 | pylethrombosis 门静脉血栓形成 | pylic 门静脉的

pylethrombosis：门静脉血栓形成无忧雅思网

pylethrombophlebitis 门静脉血栓静脉炎无忧雅思网,_|7O[@Z9J QW | pylethrombosis 门静脉血栓形成无忧雅思网R\\^(f&RZ;y;WU#p | pylic 门静脉的无忧雅思网mH]MNT

pylic：门静脉的

pylethrombosis 门静脉血栓形成 | pylic 门静脉的 | pylon 暂用假肢

pylic：门静脉的无忧雅思网

pylethrombosis 门静脉血栓形成无忧雅思网R\\^(f&RZ;y;WU#p | pylic 门静脉的无忧雅思网mH]MNT | pylon 暂用假肢