- 更多网络例句与细胞损伤毒素相关的网络例句 [注：此内容来源于网络，仅供参考]
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The book comprehensively reviews the molecular and cellular biology of the most important microbial toxins with a focus on recent advances in the field, covering topics such as toxin ADP-ribosylation of proteins, toxin inhibition of protein synthesis, modulation of actin cytoskeleton, cytolethal distending toxins, cell-damaging/pore-forming toxins, clostridial neurotoxins, toxins of the immune system, streptococcal and staphylococcal toxins, anthrax toxins, Shiga toxin, Yersinia toxin, mycotoxins, and the regulation of toxin expression, as well as the potential of microbial toxins in bioterrorism.
该书全面评论了最重要的微生物毒素的分子与细胞生物学，重点是这一领域中的最新进展，主题包括蛋白质毒素ADP核糖基化，毒素抑制蛋白质合成，肌动蛋白细胞骨架的调控，细胞致死增加毒素，细胞损伤/孔状毒素，梭菌属的神经毒素，免疫系统的毒素，链球菌和葡萄球菌毒素，炭疽毒素，Shiga毒素，Yersinia毒素，毒枝菌素，和毒素表达调节，以及生物恐怖中微生物毒素的潜在可能性。
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In these prevalent areas of HBV infection and risky regions of HCC,HCV has not become the main and sole etiogenic,neither is it a rare phenomenon.
HCV感染虽非HCC的主要发病因素，HCV引起肝细胞损伤，增加肝细胞对HBV 及其它致癌因素如黄曲霉毒素的敏感性，加快肝癌发生进程。
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In the past twelve years there have been hundreds of studies showing these nonthermal effects--- such as DNA damage and nonrepair, opening of the blood-brain barrier (allowing toxins and pathogens to reach the brain), lowered immunity, decreased melatonin levels, effects on stress proteins, formation of micronuclei (aberrations in cell nuclei which are often markers for cancer), changes in calcium metabolism affecting communication between cells, changes in brainwave patterns as seen on EEG's, plus effects observed on many different systems of the body.
在过去十二个月里，已有数以百计的研究表明这些非热效应---如DNA损伤和nonrepair ，开放血脑屏障（允许毒素和病原体到达大脑），降低免疫力，降低褪黑激素水平，对应激蛋白，形成的微（畸变细胞核往往是癌症的标志），改变钙代谢的影响细胞之间的沟通，改变脑电波模式脑电图上看到的，加上效果观察许多不同的系统的机构。
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The production of Shiga toxins and the induction of attaching and effacing lesions on intestinal epitheliums are the two most crucial elements of EHEC to trigger infectious diseases.
EHEC O157：H7菌感染宿主后，主要定植在宿主肠道黏膜上皮细胞表面，通过分泌志贺毒素和引发宿主细胞黏附及擦拭性损伤(attaching and effacing lesions，A/E lesions)两种主要方式而引发宿主严重疾病。
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It is now known that seere hypoxia-ischemia may not cause immediate cell death, but may precipitate a complex biochemical cascade leading to the delayed deelopment of neuronal loss. These phases include a latent phase after reperfusion, with initial recoery of cerebral energy metabolism but EEG suppression, followed by a secondary phase characterized by accumulation of cytotoxins, seizures, cytotoxic edema, and failure of cerebral oxidatie metabolism from 6 to 15 h post insult.
现在认识到严重的局部缺氧有可能不立即引起细胞坏死，但是可能促成一种复杂的生化级联反应，这可能导致神经元丢失，进而导致延缓发育，这些时期包括再灌注后的潜伏期，该期出现早期的脑能量代谢复苏但脑电图却呈抑制状态，紧接着第二期表现为损伤后6－15个小时细胞毒素，癫痫，细胞毒性水肿，脑氧化功能衰竭的累积。
- 更多网络解释与细胞损伤毒素相关的网络解释 [注：此内容来源于网络，仅供参考]
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monolayer：单层
模孔增大,原具有屏障作用的细胞脂层被剥脱,其通透性增加,当内皮毒素等有害物质进入血液循环后,首先损伤肺泡毛细血管内皮细胞(pulmonary cpillary endothelial cells,PCEC),使内皮细胞单层(monolayer)渗透性增加,收缩,死亡,
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Bordetella pertussis：百日咳博德特氏菌
呼吸麻痹白喉杆菌细胞毒素 白喉棒杆(Corynebacterium diphtheriae) 白喉 抑制蛋白质合成 杀死咽喉细胞,损伤脏器百日咳毒素 百日咳博德特氏菌(Bordetella pertussis) 百日咳 抑制细胞
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cytost：细胞损伤毒素
cytospongium细胞海绵质 | cytost细胞损伤毒素 | cytostainapparatus细胞染色机