- 更多网络例句与乙酰化作用相关的网络例句 [注：此内容来源于网络，仅供参考]
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The immunoregulation activity of SPG-1 mainly existed in the part of sugar chain, but keeping its molecular structure intact had some effects on its immunoregulation activity; SPG sulphation had reducing effects on the immunoregulation of SPG-1, whereas, after medium and low acetification of SPG, the immunoregulation of SPG was enhanced. Conversely after high acetification, the immunoregulation of SPG-1 decreased, that is, after introducing sulfuric and acetic acid base in SPG-1, its immunoregulation changed. All of these indicated the structure of SPG-1 was closely connected with its immune activity.⑤.
甘薯糖蛋白SPG-1的免疫调节活性主要在其糖链部分，但保持分子结构的完整性对其免疫调节的活性有一定的作用；甘薯糖蛋白硫酸化对甘薯糖蛋白SPG-1的免疫调节有降低作用，而中、低度乙酰化后，可提高甘薯糖蛋白的免疫调节作用，高度乙酰化后则降低其免疫调节作用，即甘薯糖蛋白引入乙酰基和硫酸基团后，其免疫调节活性发生了改变，这些均表明了甘薯糖蛋白的结构与其免疫调节活性有密切的关系。
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This is further supported by the demonstration that Elp3 promotes acetylation and counteracts HDAC6-mediated deacetylation of this substrate invitro.
Elp3对乙酰化的促进作用，和对HDAC6介导体外基质乙酰化的阻碍作用，更进一步的证实了上述观点。
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BMI-1(B-cell-specific Moloney murine leukemia virus integration site 1), a Polycomb group repressor, is always present as a PcG complex combined with RING1, HPC2, Mph2, et al. BMI-1 prolongs cells'capacity to proliferate and represses their senescence by repressing the transcription of CDKN2A through acetylhydrolasing or deacetylhydrolasing the Polycomb response elements in chromosome.
BMI-1 (B-cell-specific Moloney murine leukemia virus integration site 1)属于PcG家族，它与该家族其他成员如RING1，HPC2，Mph2等结合形成蛋白复合体，作用于染色质PREs位点，通过对组蛋白进行乙酰化和去乙酰化修饰，从而稳定抑制CDKN2A转录，最终促进细胞的增殖并抑制细胞凋亡。
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Significant processing steps can include: a phase changes involving either the desired molecule or the solvent, inert carrier or vehicle, e.g., dissolution, crystallization, evaporation, sublimation, distillation or absorption; b a phase separation such as filtration or centrifugation; c any chemical change involving the desired molecule, e.g., removal or addition of water of hydration, acetylization, formation of the salt; d an adjustment of the solution containing the molecule such as adjustment of pH or pO2; e a precision measurement of contained or added BPC components, in-process solutions, recycled materials is performed, i.e., weighing, volumetric measuring, optical rotation, spectrophotometric determinations, etc.; and f changes occur in surface area, particle size, or lot uniformity, e.g., milling, agglomeration, blending.
意义较大的步骤可能会包括：a相变化，包括指定分子或溶剂，惰性载体或溶媒，如溶出作用，结晶，蒸发，升华，蒸馏或吸附；b相分离，如过滤或离心；c涉及指定分子的任一化学变化，如结晶水的除去或添加，乙酰化作用，成盐；d包含分子的溶解的调节，如调节pH或pO2；e 含有或添加原料药组分，过程内溶液，回收物料的精密测量，如，称重，体积测量，旋光度，分光光度测定，等和f 表面，粒度，或批一致性的变化，如磨碎，凝块，混合。
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Histone acetyltransferases and deacetylases function antagonistically to control histone acetylation.
组蛋白的乙酰化在调节染色体结构和功能中起着重要的作用，而这种调节受组蛋白乙酰化酶和去乙酰化酶的共同调节。
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In this review, we discussed the consequences of inhibiting histone deacetylases in cycling versus non-cycling cells, in light of the dynamics of histone acetylation patterns with a specific emphasis on heterochromatic regions of the genome.
本综述通过讨论HDACIs对周期和非周期细胞中组蛋白去乙酰化酶的抑制结果，来阐明组蛋白乙酰化模式的动力学特征，特别是对基因组异染色质的作用。
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To elucidate the possible mechanism of differentiation and /or apoptosis in NB4, K562 cells induced by tributyrin, a histone deacetylase inhibitor, the level of acetylated histone H3 was detected by Western blot, p21~ WAF1expression was detected by semi-quantitative RT-PCR.
为了探讨组蛋白去乙酰化酶抑制剂三丁酸甘油酯(tributyrin，TB)诱导NB4、K562白血病细胞分化和凋亡的作用机制，利用Westernblot方法及逆转录聚合酶链反应检测TB作用前后NB4、K562细胞组蛋白H3乙酰化水平以及p21WAF1表达量的改变。
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To elucidate the possible mechanism of differentiation and/or apoptosis in NB4, K562 cells induced by tributyrin, a histone deacetylase inhibitor , the level of acetylated histone H3 was detected by Western blot, p21(superscript WAF1) expression was detected by semi-quantitative RT-PCR.
为了探讨组蛋白去乙酰化酶抑制剂三丁酸甘油酯(tributyrin， TB)诱导NB4、K562白血病细胞分化和凋亡的作用机制，利用Western blot方法及逆转录聚合酶链反应检测TB作用前后NB4、K562细胞组蛋白H3乙酰化水平以及p21(上标 WAF1)表达量的改变。
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Virtually every enzyme in glycolysis, gluconeogenesis, the tricarboxylic acid cycle, the urea cycle, fatty acid metabolism, and glycogen metabolism was found to be acetylated in human liver tissue.
这一命题中的关键之处在于乙酰化在生物调控中的重要作用。目前蛋白质组学已经发现了上千的被乙酰化的哺乳动物蛋白，中国科学家的这些发现证明了代谢酶。
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Methods After treated with a specific demethylating agent,Aza and acetylating agent, TSA, the status of 5'CpC island methylation of ING1b gene in HT29 human colon cancer cell line was analyzed using methylation specific polymerase chain reaction,and the level of histone acetylation was analyzed by chromatin immunoprecipitation,and reverse transcription polymerase chain reactionwas used to examine ING1b mRNA expression.
应用特异性DNA甲基转移酶抑制剂5-氮-2'-脱氧胞苷（5-Aza-2'-dc，以下简称Aza）及组蛋白去乙酰化酶抑制剂曲古抑菌素A作用人结肠癌细胞株HT29后，用甲基化特异性PCR检测该ING1b基因核心启动子区域CpG岛甲基化情况，用染色质免疫沉淀检测其乙酰化组蛋白绑定的DNA情况，并用逆转录聚合酶链反应检测ING1bmRNA表达。
- 更多网络解释与乙酰化作用相关的网络解释 [注：此内容来源于网络，仅供参考]
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acetification：乙酸化作用,醋化作用
acetic anhydride乙酸酐 | acetification乙酸化作用,醋化作用 | acetin乙酸甘油酯,三乙酰甘油酯
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Acetyl：乙酰基
聚酮是源自乙酰基(acetyl)与丙酰基(propionyl)的聚合. 可能的作用有抗生素、抗真菌素、细胞稳定(cytostatic)或天然杀虫剂等. 具有商业价值. 聚酮树脂(又称醛酮树脂、酮醛树脂)是一种具有高亮度、耐光性的中性、非皂化型环已酮--甲醛树脂.
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acetylation：乙酰化
3.1.4 乙酰化(acetylation)学说:Farr(1970)指出,本病可能是由于阿司匹林乙酰化作用最终产物的积蓄所致,但这一作用没有被证实. 阿司匹林耐受不良的患者中,嗜酸粒细胞活化的事实,已经为鼻息肉透射电镜检查见到的嗜酸粒细胞脱颗粒现象所证实.
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acetylation：乙酰化作用
论文摘要: 表遗传修饰和染色质结构重组(chromatin remodelling)参与了胚胎形成及肿瘤发生,乙酰化作用(acetylation)是调控基因转录的关键修饰之一,组蛋白去乙酰基酶抑制剂被视为肿瘤治疗中最有前景的药物开发目标.
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acetylation value：乙酰化值
acetylating agent | 乙酰化剂 | acetylation value | 乙酰(化)值 | acetylation | 乙酰化作用
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phosphorylase：磷酸化酶
如胆碱乙酰化酶(ChAC)、磷酸化酶(Phosphorylase)等. 蛋白酶(Protease) 水解蛋白质转化为蛋白练、氨基酸,从促进蛋白消化与吸收. 有些种类的蛋白酶具有消炎溶解血栓以及抗肿瘤的作用. 脂肪酶(Lipase) 迅速消除体内囤积的多于脂肪、有减肥作用.
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acetin：乙酸甘油酯,三乙酰甘油酯
acetification乙酸化作用,醋化作用 | acetin乙酸甘油酯,三乙酰甘油酯 | acetoacetic acid乙酰乙酸
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acetylate：乙酰化,乙酰化产物
acetylvanilloyl乙酰基香草酰 | acetylate乙酰化,乙酰化产物 | acetylation乙酰化作用
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acetylization：乙醯化<作用>
acetylith | 电石 | acetylization | 乙酰化作用 | acetylizer | 乙酰化器
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ethanoyl iodide：乙酰碘
ethanoyl chloride乙酰氯 | ethanoyl iodide乙酰碘 | ethanoylation乙酰化<作用>