- 更多网络例句与主动脉内膜炎相关的网络例句 [注：此内容来源于网络，仅供参考]
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Results The body weight, visceral fat and body weight ration, as well as the levels of blood glucose, insulin, triglyceride, total cholesterol, LDL, and total FFA, were significantly lower in high fat-fed group than in control group (P.01). The aortic tunica intima was intact, the number of foam cells was decreased, and inflammatory cells infiltrated into the med-intima smooth muscle of the n-3 PUFA group. However, with no such changes were observed in control and n-3 PUFAs groups.
结果 n-3脂肪酸组大鼠体重第1~11周均明显降低，空腹血糖、血清胰岛素、TG、TC、LDL-C、总FFA和内脏脂肪占体重百分比均明显低于HF组，HDL-C则明显高于HF组，与对照组无明显差别；n-3脂肪酸组大鼠主动脉主动脉内膜完整无内皮细胞脱落现象，内膜下泡沫细胞及炎细胞浸润减少，内膜的NF-κB呈弱阳性表达，IκBα呈阳性表达，ICAM-1和MCP-1均呈阴性或弱阳性表达。
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The aortic tunica intima was intact, the number of foam cells was decreased, and inflammatory cells infiltrated into the med-intima smooth muscle of the n-3 PUFA group. However, with no such changes were observed in control and n-3 PUFAs groups. Little or no expression of NF-κB, ICAM-1 and MCP-1, and a high expression level of IκBα were detected in aortic tunica intima of the n-3 PUFA and control groups, while high expression levels of NF-κB, ICAM-1 and MCP-1, and little expression of IκBα were detected in aortic tunica intima of the high fat-fed group.
结果 n-3脂肪酸组大鼠体重第1~11周均明显降低，空腹血糖、血清胰岛素、TG、TC、LDL-C、总FFA和内脏脂肪占体重百分比均明显低于HF组，HDL-C则明显高于HF组，与对照组无明显差别；n-3脂肪酸组大鼠主动脉主动脉内膜完整无内皮细胞脱落现象，内膜下泡沫细胞及炎细胞浸润减少，内膜的NF-κB呈弱阳性表达，IκBα呈阳性表达，ICAM-1和MCP-1均呈阴性或弱阳性表达。
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And rheumatic valves also accounted for 70 cases (88.5%) of aortic stenosis plus insufficiency. The rheumatic valves was decreased dramatically as the cause of the aortic valve disease, aortic root dilation and degenerative valves increased significantly, while others had no significant changes.
随着社会进步和人口老龄化，风湿性病因所占比例大幅下降，主动脉根部扩张导致的主动脉关闭不全有较大幅度的上升，退行性主动脉瓣有所上升，二叶式主动脉瓣、感染性心内膜炎所致瓣膜损害变化不明显。
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The characteristics and changes of etiology were analyzed. All cases with aortic diseases were divided into single aortic stenosis, single aortic insufficiency, and combined aortic stenosis plus insufficiency group.
分别以主动脉瓣狭窄、主动脉瓣关闭不全、主动脉瓣狭窄合并关闭不全3组分析，具体病因包括风湿性、二叶式主动脉、退行性主动脉瓣、主动脉根部扩张、感染性心内膜炎、少见及未确定等6类病因。
- 更多网络解释与主动脉内膜炎相关的网络解释 [注：此内容来源于网络，仅供参考]
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Aortic dissection：主动脉夹层
主动脉夹层(aortic dissection)指主动脉内的循环血液通过内膜破裂口进入主动脉中层而形成的血肿,是一种极为严重的大动脉疾病. 其形成原因较多,有动脉硬化、高血压、动脉中层囊性坏死、马凡氏综合征、主动脉缩窄、大动脉炎、外伤及梅毒等. 尽管主动脉夹层...
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endaortitis：主动脉内膜炎
endangered 有危险的 | endaortitis 主动脉内膜炎 | endarch 内始式的
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endaortitis：主动脉内膜炎 (名)
endangered species 濒临绝种的动植物 | endaortitis 主动脉内膜炎 (名) | endarteritis 动脉内膜炎 (名)
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endarteritis：动脉内膜炎 (名)
endaortitis 主动脉内膜炎 (名) | endarteritis 动脉内膜炎 (名) | endarterium 动脉内膜; 动脉内层 (名)
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rheumatic endocarditis：风湿性心内膜炎
1.风湿性心内膜炎 风湿性心内膜炎(rheumatic endocarditis)常侵犯心瓣膜,其中二尖瓣最常被累及,其次为二尖瓣和主动脉瓣同时受累. 三尖瓣和肺动脉瓣一般不被累及. 腱索和左心房壁内膜有时也可被侵犯. 2.风湿性心肌炎 风湿性心肌炎(rheumatic myocarditis)主要累及心肌间质结缔组织.
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aortic insufficiency：主动脉瓣关闭不全
主动脉瓣关闭不全(aortic insufficiency)主要由主动脉瓣疾病引起,可以是风湿性主动脉瓣炎,也可以是感染性心内膜炎及主动脉粥样硬化和梅毒性主动脉炎累及主动脉瓣,病变致使瓣膜增厚、变硬、缩短、弹性减弱或消失,引起瓣膜环扩张,致使主动脉瓣关闭不全.
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Mitral stenosis：僧帽瓣狭窄
(A) 并非真正的发炎 (B) 最易发生在上肢静脉 (C) 其最可怕的并发症是栓塞 (D) 患部可见发红肿胀. ('97高考)(B) 12.后天性僧帽瓣狭窄(mitral stenosis)多半为哪一种病变所造成?(A) 梅毒性主动脉炎 (B) 慢性风湿性心内膜炎 (C) 亚急性传染性心内膜炎 (D) 瓣膜黏液变性.
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aortic regurgitation：主动脉瓣关闭不全
主动脉瓣关闭不全(aortic regurgitation)的病因除先天性主动脉瓣发育畸形和马凡综合征外,最常见的是风湿性瓣膜病,其次为细菌性心内膜炎和主动脉夹层动脉瘤等.
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endarch：内始式的
endaortitis 主动脉内膜炎 | endarch 内始式的 | endarteritis 动脉内膜炎
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rheumatic pancarditis：风湿性全心炎
风湿病时病变常累及心脏各层,故称为风湿性全心炎(rheumatic pancarditis). 1.风湿性心内膜炎 风湿性心内膜炎(rheumatic endocarditis)常侵犯心瓣膜,其中二尖瓣最常被累及,其次为二尖瓣和主动脉瓣同时受累. 三尖瓣和肺动脉瓣一般不被累及.