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All patients were randomly divided into observation group(lidocaine creosote group,37 cases) and control group(lidocaine hydrochloride group,37 cases).then compared the onset time, completion time, lasting time, block range, first total volume of novocaine and its lasting time of two kinds of novocaine.

随机分为观察组(碳酸利多卡因组,37例)和对照组(盐酸利多卡因组,37例)。比较两种局麻药的起效时间、阻滞完全时间、麻醉持续时间、阻滞节段、局麻药首剂总量及首剂量维持时间。

Then compared the onset time, completion time, lasting time, block range, first total volume of novocaine and its lasting time of two kinds of novocaine.

比较两种局麻药的起效时间、阻滞完全时间、麻醉持续时间、阻滞节段、局麻药首剂总量及首剂量维持时间。

Only one (5%) patient with femoralblock developed obturator motor block.

仅有一例股神经阻滞患者出现闭孔神经阻滞

Regarding to different successful ablation monitoring indicatives, patients were divided into ablation group in traditional methodand ablation group with atria pacemaking showing slow pathway conduction block as successful ablation indicative.86of groupⅰ presented junction rhythm in dischargeing 15s or early pacemade then consecutively discharged 60~90 seconds; in groupⅱ 67 patients after dischargeing 15s showed junction rhythm or after early pacemaking delayed dischargeing to 20s then stopped dischargeing.

按不同的有效消融判断指标分为传统方法消融组和以心房起搏显示慢径前传导阻滞作为有效消融指标消融组。i组86例在放电后15 s内以出现交界心律或早搏后继续放电60~90 s;ii组67例放电15 s内出现交界心律或早搏后延迟放电至20 s停止放电,以术前av1﹕1最短间期心房刺激(s1s1)显示慢径前传阻滞后停止起搏继续放电至60 s。

Using nerve stimulator suppelementary fixed position to reach brachial plexus block is more accurate and successful than using paresthesia.

应用神经刺激器辅助定位实施臂丛神经阻滞,比异感定位准确,阻滞成功率高。

Results: The aneshesia methods included general anesthesia (1 pateint), ketamine anaesthesia with midazolam premedication (16 patients), epidural anesthesia (59 patients), and brachial plexus block (13 patients).

结果:气管插管全麻1例,氯胺酮咪达唑仑静脉麻醉复合局部麻醉16例,硬膜外阻滞59例,臂丛神经阻滞13例。

Methods]By observing the cytoplasmic Ca2+ concentration change of single cardiocyte in I/R status and the effect of Na+/H+ permutoid retarder to Ca2+ concentration in different phases (diabetes pure oxygen-lack deficit group called DM group,the oxygen deficit/reaeration on entire journey for medicine group called DM-EIPA group, the first group giringmedicine before reaeration on called DM-EIPA 1 group, and the second group giringmedicine before reaeration on called DM-EIPA 2 group) to detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration injury.

方法] 通过观察糖尿病鼠单个心肌细胞在缺氧/复氧时细胞胞浆Ca2+浓度的动态变化以及Na+/H+交换体阻滞剂在不同时相(糖尿病单纯缺氧组即DM组,缺氧/复氧全程给药组即DM-EIPA组,复氧前给药1组即DM-EIPA 1组,复氧前给药2组即DM-EIPA 2组)对Ca2+浓度的影响来研究Na+/H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧/复氧损伤的保护机制。

Results The anesthesia range was narrowed and anesthesia duration was shortened with the prolongation of epidural catheter placement,Epidural roentgenography showed that the medium diffused normally within epidural space,biopsy of epidural tissues showed that there were inflammative cells infiltration and fiber proliferation with longer epidural catheter placement(more than 4 weeks),without significant differences between both groups.

结果 随着硬膜外腔置管时间延长,麻醉阻滞范围变窄,麻醉阻滞时间也明显缩短,两组之间没有明显区别;硬膜外腔造影显示:造影剂在硬膜外腔扩散良好,无明显受阻;组织学检查显示:置管时间长(4周以上)的动物,硬膜外组织炎细胞浸润、纤维组织增生、硬膜增厚,而且随置管时间延长,变化加显著,但两组之间没有明显区别。

Results The anesthesia range was narrowed and anesthesia duration was shortened with the prolongation of epidural catheter placement,Epidural roentgenography showed that the medium diffused normally within epidural space,biopsy of epidural tissues showed that there were inflammative cells infiltration and fiber proliferation with longer epidural catheter placement(more than 4 weeks),without significant differences between both groups.Conclusion Epidural corticosteroids can not prevent epidural adhesion from occurrance induced with long-term epidural catheter placement.

结果 随着硬膜外腔置管时间延长,麻醉阻滞范围变窄,麻醉阻滞时间也明显缩短,两组之间没有明显区别;硬膜外腔造影显示:造影剂在硬膜外腔扩散良好,无明显受阻;组织学检查显示:置管时间长(4周以上)的动物,硬膜外组织炎细胞浸润、纤维组织增生、硬膜增厚,而且随置管时间延长,这"种变化更"加显著,但两组之间没有明显区别。

In acute experiments, APO-induced stereotypy and locomotion could be blocked by both selective 〓 antagonists SCH 23390 and 〓 antagonist sulpiride, and they had a synergistic effect when used in combination.

急性实验中,选择性〓阻滞剂SCH 23390和〓阻滞剂舒必利能有效地对抗APO诱发的定型活动和肢体运动,两药相加有协同作用。

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