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toxication相关的网络例句

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与 toxication 相关的网络例句 [注:此内容来源于网络,仅供参考]

The animal models of subchronic cad-mium toxication and antagonism of selenium were established.

结果显示,亚慢性镉中毒可引起鸡肝组织细胞凋亡,加硒后可有效减少肝细胞凋亡数量及损伤程度。

Then, all of the rats in toxication group received a single intragastric administration of ANIT (200 mg/kg) in order to induce acute intrahepatic cholestasis.

中毒组按200 mg/kg一次性灌服ANIT诱发大鼠急性肝内胆汁淤积病变,观察灌服ANIT后中毒组在48, 96, 144, 192 h胆汁流量、血中TB和ALT值及胃肠MMC的变化。

RESULTS: Forty-eight hours after ANIT was given, the biliary flow of rats in toxication group was almost ceased completely while the levels of ALT and TB were increased remarkably.

结果: 中毒组灌服ANIT后, 48 h时胆汁流量中断,血清TB和ALT明显升高; 48 h后其胆汁流量逐渐增加,血清TB和ALT逐渐下降,于192 h基本恢复。

Acute diffuse lung injury were identified in toxication control group, intra-pul

结果 中毒组大鼠肺的病理学改变为急性弥漫性肺损伤,表现为肺泡腔内出血、渗出、炎性细胞浸润,肺泡隔炎性细胞浸润及纤维组织增生,⌒秃廷⑿蜕掀は赴坏死脱落。

Mechanism of liver injury in cadmium toxication and protection of metallothionein Jin Huiying*,Hu Huimin,Zhou Young,et al.*Institute of Military Medicine of Nanjing Command,Nanjin 210002

目的 研究脂质过氧化和自由基在急性镉中毒性肝脏损伤中的作用及金属硫蛋白的保护作用。

Toxication Department,Institute of Occupational Disease of Guangxi Autonomous RegionNanning 530021,China

作者:苏素花,廖瑞庆,农康,葛宪民,蒋东方,韦国铭作者单位:广西壮族自治区职业病防治研究院中毒科,南宁 530021

Results There were 17, 228 victims from 2006 to 2007, and most victims were slight injuries (59.66%), male (65.78%) and 25~44 years old (43.34%). Injury incidents occurred in Autumn (27.65%), and lots of incidence were happened in highway/street (37.26%) and home (20.70%). Tumble/falling was the most major types of injury (28.18%), followed by vehicle accident (26.89%). Ratio of gender was 1.92:1 except toxication and apnoea/hanging. 82.67% belonged to misadventure.

结果 三家哨点医院共接诊伤害病例17 228人,多为轻伤(59.66%)且男性为主(65.78%),以25~44岁年龄组最多(43.34%);伤害时间主要集中在秋季(27.65%);地点主要为公路/街道(37.26%)和家中(20.70%);跌倒/坠落(28.18%)是首要原因,其次是机动车车祸(26.89%);除中毒和窒息/上吊主要为女性外,其他伤害事件中均以男性居多(男女性别比为1.92:1);82.67%的伤害罹难者为意外所致。

Acute diffuse lung injury were identified in toxication control group, intra-pulmonary alveoli hemorrage, effusion, inflammotory cell infiltration and inter-pulmonary alveoli, fibroplasia were seen; necrosis and abbscission were seen in of typeⅠand Ⅱ epithelial cells.

结果 中毒组大鼠肺的病理学改变为急性弥漫性肺损伤,表现为肺泡腔内出血、渗出、炎性细胞浸润,肺泡隔炎性细胞浸润及纤维组织增生,Ⅰ型和Ⅱ型上皮细胞坏死脱落。

Conclusions1 Capsule FuYuanYuGan shows definite protective and therapeutic effect on experimental hepatic fibrosis model rat.2 Capsule FuYuanYuGan had no liver toxication.

湖北中医学院肝病专业硕士研究生毕业论文结论:1复元愈肝胶囊具有优于秋水仙碱的抗肝纤维化的作用。2复元愈肝胶囊无明显肝毒副作用。

Sweet in toxication.

甜蜜地陶醉吧。

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