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peripheral sensory neuron相关的网络例句

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与 peripheral sensory neuron 相关的网络例句 [注:此内容来源于网络,仅供参考]

Objective To summarize the clinical symptom and the medicative feature of motor neuron disease. And to analyze internal relationship between motor neuron disease and the theory of the correlation of spleen and kidney.

目的总结归纳运动神经元疾病的临床症候、治疗用药的特点及其与中医脾肾相关理论的内在关系。

In pure cultured neuron, the inhibitory effect of 0.5mmol/L L-NNA on theincrease of LDH leaking after exposure of H/R and SIN-1 had also no statistical significance, but it could inhibit significantly the above index in mixedly cultured cerebral cortex neuron and glia.

在单纯培养神经细胞中0.5mmol/LL-NNA对H/R+SIN-1导致的LDH漏出率增加仍无显著抑制作用,但可显著抑制H/R引起的混合培养的神经细胞和神经胶质细胞的LDH漏出率。

recovery of occlusion support facilitated increase of neuron numbers, or improvement of neuron functions, and decrease of neuroglial cell numbers in correlated brain sections of the patients, which may help improve the functions of nervous system.

研究表明,咬合支持的丧失可以导致相关脑区神经组织和神经代谢物的改变,并且这些变化与以学习记忆等功能衰退为特征的神经系统疾病如老年性痴呆的研究结果部分趋于一致,提示我们要对咬合支持丧失的危害性进行更深入地研究。

Nerve grows factor and brain-derived neurotropic factor, as one of the important active molecules, belong to neurotrophic family, which can affect the development of neuron and prevent neuron from damage.

神经生长因子(Nerve grows factor,NGF)和脑源性神经生长因子(Brain-derived neurotropic factor,BDNF)属于神经营养素家族,是神经系统重要的活性分子之一,是中枢神经系统胆碱能神经元、周围神经系统交感神经元及感觉神经元发育、分化、功能活动必不可少的营养分子。

Objective To investigate the interrelationship between the expression of VEGF and the oncosis of neuron after ICH by establishing the rat ICH model through the expression of oncosis of neuron after ICH and the cell location of VEGF-positive cells in perihematoma and the expression of VEGF. Methods The rat ICH model was induced by two-step.

目的 通过建立大鼠脑出血动物模型观察血管内皮生长因子在出血灶周的细胞定位和动态表达变化和脑出血后神经细胞胀亡时程表达以探讨两者之间的相关性。

Get the neonate Sprague-Dawley rats medulla spinalis, through the conventional primary cell culture procedure, we use the method of difference adherence time and get the motor neurons of anterior spinal cord. When the neuron is in maturity, we make the mechanical injury model in vitro. All the models were divided into four groups: group A is control group; group B is 100μM ATP group; group C is 100μM ATP+20μg/ml suramin group and group D is 100μM ATP+10μM ouabain+10μg/ml Thapsigargin group. Culture the four groups neurons, after one day, we count the motor neuron, observe the survival and activity of neurons through MTT shade selection experiment, use flow cytometry to analyze the percentage of apoptosis of motor neurons of anterior spinal cord and detect the expression of protein p-GSK-3β(ser9) through Western-Blot technology.

方法取新生大鼠脊髓,通过常规的原代细胞培养程序,采用差速贴壁法分离出大鼠脊髓前角运动神经元,培养成熟后制作神经元机械损伤体外模型,分为A组:对照组、B组:100μMATP组、C组:100gMATP+20μg/ml Suramin组和D组:100μM ATP+IOμM Ouabain+10μg/ml Thapsigargin组,对各组机械损伤的运动神经元进行培养,1天后分别进行运动神经元计数、MTT比色实验观察运动神经元的存活及活性、流式细胞仪分析机械损伤的脊髓前角运动神经元凋亡百分率和Werstern Blot技术检测p-GSK-3β(ser9)的表达。

H reflex recovery curve examimation shows that electric acupuncture can obviously suppress neuron excitability of motor neuron pool in cornu anterius medullae spinalis, decrease accentuatal tension of muscle and Liddell and Sherrington reflex.Conclusions: electric acupuncture can relieve spasm of limbs after cerebral apoplexy.

实验结果说明:针刺阳陵泉缓解脑卒中后肢体偏瘫痉挛状态,可能与针刺刺激提高了中枢神经系统中抑制性氨基酸递质受体的表达,使受体数量增加、敏感性提高,从而提高了抑制性神经递质的效率,使下位中枢运动神经元兴奋性受到抑制有关。

The amount of neuron at 2st week for EG and CC were 36.50±1.78/EF and 19.70±3.71/EF respectively, with the percentage of (43.60±4.83)% and (39.04±3.87)% for EG and CG, which had significant difference (P.05) Conclusion: Self-assembled IKVAV peptide nanofibers, which can degrade the neuron mortality as well as promote neurite sprouting and growth, is a bioactivity scaffold, it can serve as a superordinary nerve tissue engineering scaffold.

自组装IKVAV多肽纳米支架能降低神经元的死亡率,并诱导轴突的发生和生长,具有支架及生物活性双重作用,可作为神经组织工程支架材料。

ResultsThere was no expression of Caspase-3 in group A. The expression of Caspase-3 in group C was decreased compared with that in group B(P<0.01. In groups B and C, expansion and vacuolization of mitochondria, swollen neurons and apoptotic bodies appeared, which suggested that WBH could induce neuron injury, and the neuron injury in group C was more severe than that in group B.

结果A组大鼠海马区神经元无Caspase-3表达,B、C两组大鼠海马区神经元有Caspase-3表达,B组较C组Caspase-3表达平均灰度值减小。B、C两组大鼠海马神经元超微结构发生改变、细胞器水肿,部分线粒体空化,内质网稀疏,突触前膜、后膜结构不完整,可见自噬体结构;B组大鼠的神经元超微结构损害较C组轻。

To explain relationship between the effect of CLD on senile Demention and neuron in hippocampal area. The change of natural aging mice's neuron in hippocampal area were studied by applying microscope and transilluminating electric microscope. Results: Comparing with the young control, the old control mice's hippocampus had less small pyramid cells, the nerve cells denaturalizing and their branches reducing in the microscope. Hippocampal nerve cells were degenemiceing, with nucleus membrance crinkling like wave shape were not clear, and chromatin condensing with high electron density, heterochromatin accruing, part cell organs inclosing nucleus, nucleus condensing in the EM.

为了阐明菖龙丹抗老年性痴呆作用与海马区神经元结构的关系,运用光镜和透射电镜观察了菖龙丹对自然衰老小鼠海马区形态的影响,结果显示老年对照组小鼠光镜下海马锥体细胞较青年对照组数量减少,细胞体积变小,神经元变性,分枝减少;电镜下海马神经细胞发生退化改变,核膜皱缩,呈波浪状,膜结构不清晰,核染色质浓缩,电子密度高,异染色质较多,部分胞浆细胞器向细胞核聚集,有的可见细胞核固缩。

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