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The activation of NRM neurons by electroaeupuncture was eliminated after decerebration at superior colliculus level, indicating that this effect of EA depends mainly on the supra-midbrain structure. Furthermore, all treatments including AR isolation, disconnection of #beta#-endorphinergic tract and micrpinjection of naloxone or anti-#beta#-endorphin serum into PAG, would make the effect of EA disappear. Thus, it is the #beta#-endorphinergic pathway between AR and PAG-NRM system that mediates the major excitatory effect of EA on NRM.

检验AR至 PAG-NRM的神经通路在电针效应中的作用：实验显示上丘水平去大脑后，电针兴奋NRM的效应完全消失，表明此效应主要依赖中脑以上结构实现，进一步分别用Halfisz氏刀游离AR、切断AR至PAG的β-End能束、PAG内注射纳洛酮或抗β-End血清后，也使电针效应基本上完全消失，说明电针使NRM神经元兴奋的效应，主要通过AR与PAG-NRM间的β-End能神经通路实现。

The levels of asparate in 6 brain regions including striatum cerebral cortex,hippocampus,midbrain,cerebellum,diencephalon and olfactory bulb at 5,30 and 90 min after seizure were significantly decreased as compared with the control.Conclusion:The excitatory amino acid transmitter activity may play an important role in soman-induced seizures.

梭曼中毒后脑内谷氨酸和天门冬氨酸水平显著降低，递质耗竭是递质活动增加的征象，因此，谷氨酸和天门冬氨酸递质活动增加可能在梭曼中毒性惊厥的发生和维持中起重要作用。

Hyperkinetic dysarthria is the result of damage to inhibitory proceses of the extraphraidal system, whereas hypokinetic dysarthria results from lesion to excitatory mechanisms.

运动减退的构音困脑是在锥体外神经系统中抑制的过程所损害的结果，然而运动减退的发音不良却由於对刺激机制的损害而产生的。

Such excitatory neurotransmitters are necessary for normal alertness, memory, muscle tone and co-ordination, emotional responses, endocrine gland secretions, heart rate and blood pressure control and a host of other functions, all of which may be impaired by benzodiazepines.

这些兴奋神经传导质对正常的警觉，记忆，肌肉的紧张性和和谐性，情感反应，内分泌，心率和血压控制以及许多其他功能是必需的，所有这些都会被苯二氮卓类药物损坏。

Methods: The conventional intracellular recordings were conducted in MNs of spinal cord slices isolated from neonatal rats (8~14 days old). Observed in MNs were the effects of TRH on excitatory postsynaptic potentials evoked by contralateral ventrolateral funiculus stimulation i.e.

应用新生大鼠（8~14d龄）脊髓切片MN细胞内记录技术，TRH灌流给药，观察其对cVLF电刺激诱发的兴奋性突触后电位的影响。

Results Muscone increased SOD content, lowered MDA content, reduced the increase of EAA content caused by ischemic and hypoxia, and inhibited the excitatory neurotoxicity caused by EAA.

结果 麝香酮组可明显增加大鼠脑组织SOD含量，降低MDA含量，减轻缺血、缺氧造成的EAA含量增加，抑制EAA所导致的兴奋性神经毒性。

Methods The global cerebral ischemia/reperfusion injury model was established by the four-vessel occlusion (4VO) method, and then the model animals sere given high-middle-and low-dose muscone. Meanwhile, sham-operation group, model group, and positive control group were also set up. The rats neuroethology behavior, and the contents of superoxide dismutase, malondialdehyde and excitatory amino acid in is-chemic brain tissue were examined.

取Wistar大鼠，采用四血管阻断(4VO)法制备全脑缺血再灌流损伤模型，将成模大鼠随机分为麝香酮高、中、低剂量组(3.6， 1.8， 0.9 mg/kg)，并设假手术组，模型组，阳性对照药尼莫地平组(10.8 mg/kg)，共6组，每组12只对大鼠神经行为学，缺血区脑组织超氧化物歧化酶，丙二醛含量，脑组织兴奋性氨基酸含量进行检测。

However, a growing number of studies suggest that excessive extracellular glutamate and resultant overstimulation of its receptors cause excitotoxic roles and lead to neuronal damage in several pathological conditions such as cerebral ischemia. Normally, the excitatory postsynaptic roles of glutamate are terminated by the activities of high-affinity sodium dependent glutamate transporters that locate on neuroglial cells and neurons near the synaptic cleft, which are thought to play important roles in inactivating released glutamate and keep it below toxic levels in the extracellular space.

在生理状态下，突触间隙中谷氨酸的清除取决于邻近神经胶质细胞及神经元上高亲和力、钠依赖性谷氨酸载体的活动，通过该载体的再摄取机制保持细胞外谷氨酸浓度于正常范围，这在预防谷氨酸的神经毒中起着重要的作用。

Thus, in the electrophysiological aspect of CNS, the excitatory model was set up and the effect of JuA on the bioelectrical activity of the CNS was tested. The results were comparable with those obtained in the experiments of western medicine, eg. Phenobarbital sodium.

实验中比较了两种不同的Ca〓荧光探针Fluo-3和Calcium Orange的染色条件及荧光发射特点，分析了F1uo-3在检测Ca〓快速反应变化中的优点，而Calcium Orang比较能够体现Ca〓的空间分布差异，国内尚无有关类似的结果的研究报道。

JuA with the concentrations 0.05g/L and 0.10g/L were tested based on the excitatory effect of 1000KIU/L penicillin and it was found that JuA could inhibit the burst induced by penicillin in CA1 pyramidal cells. Phenobarbital sodium was also applied as apositive control, and the results were in consistent with those reported.

说明青霉素主要通过升高神经组织内的谷氨酸水平破坏神经系统的兴奋和抑制的平衡，而JuA则可以逆转青霉素引起的谷氨酸的升高，在递质的水平起到神经抑制和保护作用。

His female bullfighter Lydia gradually Health love, but she was in a vegetative state after the Bulls Zhuangcheng away from her, just because Lydia's ex-boyfriend showed more affectionate than he is like love.

他对女斗牛士莉迪亚渐生爱意，却在她被公牛撞成植物人后离她而去，仅仅因为莉迪亚的前男友表现出好似比他更深情的爱。

I know of none which are not easy to answer, but what I am trying to do now is only by a series of hints to throw some light on the narrow path of legal doctrine, and upon two pitfalls which, as it seems to me, lie perilously near to it.

我不认为有什么难以回答的问题，但我现在要做的仅仅是通过一系列线索，来给法律理论的狭窄路径以及在我看来离它近得危险的两个陷阱提供一些启示。