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dox相关的网络例句
与 dox 相关的网络例句 [注:此内容来源于网络,仅供参考]

Variability of encapsulation efficiency of DOX and VER was small over 14 days, and particle sizes of DARSLs were slightly increased.

该制剂在血浆介质中的体外泄漏试验结果表明同时包载的维拉帕米可能会影响脂质体稳定性。

The results showed that the encapsulated DOX had better efficacy than that of the free drug in terms of tumor inhibition in a 4 week in vivo culture period.

经4周体内培养,结果显示包埋的DOX(抑制率40.3%)比游离的DOX(抑制率30.6%)具有更好的治疗效果。

The expression of MST1 was identified under Dox induction.

在强力霉素诱导条件下,肝癌细胞系Huh-7可以表达MST1。

Objective To investigate the inhibitive effect of metallothionein on DOX-induced cardiac apoptosis and the involved possible mechanisms.

目的 研究锌诱导的金属硫蛋白对阿霉素所致心肌细胞凋亡的的抑制作用并探讨其作用机制。

Methods:Doxorubincin-resistant human hepatoma cell subline (HepG2/DOX) was induced by doxorubincin-gradually increasing concentration.

以浓度梯度递增方法,用DOX诱导HepG2产生耐药性。

The endocellular Fura-2 increased to 2.39,3.14,and 3.24 fold respectively by addition of AA,bullatacin and atemoyacin-A and the accumulation of Dox in MCF-7/ADR cells was similar to that of Fura-2,increased to 2.98,4.10 and 4.24 fold respectively by AA,bullatacin,and atemoyacin-A while no in MCF-7 cells.

细胞内阿霉素的积累与Fura-2 相似,加入AA,bullatacin和atemoyacin-A均分别增加 MCF-7/ADR 细胞内 ADR 的积累至2.98,4.10和4.24倍,而未见显著增加敏感株内 ADR 的积累。bullatacin和atemoyacin-A均能诱导MCF-7与MCF-7/ADR细胞的凋亡,且相同浓度下,两细胞株的凋亡百分数无显著差异。

Preinduction of MT by zinc protects the heart from DOX-induced cardiotoxicity, and this effect is possibly correlated with the property of MT on scavenging free radicals in vivo.

Zn诱导MT表达对DOX心脏毒性具有明显的保护作用,其机制可能与MT体内清除自由基功能有关。

So our work is further to study the characteristics of DOX-induced cardiotoxicity with proteomics and metabonomics, combined with conventional toxical technologies, so as to exploring the unknown toxicity targets and selecting biomarkers to detecting the adverse effects.

为了深入了解DOX心脏毒性的作用机制、寻找新的毒性作用靶标及合理选用生物标志物以监测其不良反应,本研究拟将代谢组学技术和蛋白质组学技术与传统毒性评价技术相结合,研究DOX心脏毒性的特征。

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