查询词典 decomposition group

Results l.Serum concentrations of VCAM-1 in PIH (93.39 + 57.3)ug/ml were higher than that in normal pregnant group. Serum concentrations of VCAM-1 in moderate(97.89 + 34.07)ug/ml and severe PIH(132.24 + 60.97)ug/ml were significantly higher than that in normal group.(P.05, P.01). There was no difference between serum levels of VCAM-1 mild PIH and normal pregnant group. 2.Serum levels of IL-6 in PIH group(102.17 ?48. 31)pg/ml were significantly higher than that in normal group(49.16 + 12.9)pg/ml.Serum levels of IL-6 in moderate(95.79+31.19)pg/ml and severe PIH( 127.27+11.3 8)pg,ml were significantly higher than that in normal group. There was no difference between serum levels of PIH in mild PIH (52.13 + 12.90)pg/ml and normal pregnant group. 3. In PIH group, serum concentrations of VCAM-1 correlate with the levels of IL-6,r=0.63. 4.The expression of VCAM-1 in cytotrophoblast of spiral arteries in normal pregnant group(100%) were significantly higher than that in PIH group.Theexpression of VCAM-1 in moderate PIH(37.50%) and sever PIH were lower than that in normal group.There was no difference between the mild PIH and normal group.Conclusions The increased levels of serum of VCAM-1 may participate in the process of vascular endothelium damages in PIH.

结果 1、妊高征组血清VCAM-1浓度为(93.39±57.3)μg／ml明显高于正常妊娠组(44.87±15.60)μg／ml，差别有显著性(P＜0.05)；中、重度妊高征组VCAM-1浓度分别为(97.89±34.07)μg／ml和(132.24±60.97)μg／ml，与正常妊娠组比较，差异有显著性(p＜0.05)和非常显著性(P＜0.01)；轻度妊高征组VCAH-1为(48.46±15.60)μg／ml与正常妊娠组比较，差异无显著性(P＞0.05)。2、妊高征组血清IL-6含量为(102.17±48.31)pg／ml，明显高于正常妊娠组(49.16+12.9)pg／ml，差异有非常显著性(P＜0.01)；中、重度妊高征组IL-6含量分别(95.79±31.19)pg／ml和(127.27±11.38)pg／ml，与正常妊娠组比较，差异有显著性(P＜0.05)和非常显著性(P＜0.01)；轻度妊高征组IL-6含量为(52.13±12.90)pg／ml与正常妊娠组比较，差异无显著性(P＞0.05)。3、VCAM-1与IL-6水平呈明显正相关，r=0.63(P＜0.01)。4、子宫胎盘床螺旋动脉滋养细胞VCAM-1表达，正常妊娠组都存在阳性表达(阳性表达率100％)，妊高征组有10例阳性表达阳性表达率为叩们，差别有非常显著性河＜0.01L 中、重度妊高征组阳性表达率分别为37、50％和0，与正常妊娠组比较，差别有显著性河＜0.05）和非常显著性汀＜0.01太轻度妊高征组阳性表达率为83.33凡与正常妊娠组差别无显著性问＞0.05）。

All the indexes were tested in 2 -. 4 .. 6 weeks of operative intervals respectively. Results:(1) The general observation in the experimental group ,such as spirit, hair, body weight and activity ,was superior to that in the control group.(2) The recovery rate of sciatic nerve functional index (SF1) was statistically significantly higher in the experimental group than in the control group .(3) electrophysciological indexes: m the experimental group ,the tardy rate of induced motorius action potential ,recovery rates of the amplitude of compound muscle action potential and twitch tension and tetanic tension of triceps surae muscle ,were significantly higher than in the control group.(4) Compared with the control group ,the triceps surae muscle wet weight was significantly greater in the experimental group.(5) histomorphological indexes: the count of myelinated nerve fibers ,diameter of regenerated axon, thickness of nerve myelin sheath and area of capillary were much more in the experimental group than in the control group.(6) observation of ultrastructure with the transmission election microscope: more abundant organell a and maturer myelin sheath lay in the injured peripheral nerve of the experimental group, while not lay in the control group.

分别在木后第2、4、6周时进行各项指标的检测，结果发现：（1）实验组SD大鼠在整个实验过程中，其精神、毛发、体重，运动灵活性等一般情况优于对照组；（2）实验组的坐骨神经功能指数恢复率显著优于对照组，P＜0.01；（3）神经电生理指标：实验组运动神经诱发电位潜伏期的延迟率显著优于对照组，P＜0.05；复合肌肉动作电位振幅的恢复率显著高于对照组，两组比较第2周时P＜0.01，第4、6周时，P＜0.05；小腿三头肌单收缩力和强直收缩力的恢复率实验组显著高于对照组，P＜0.01；（4）小腿三头肌湿重的恢复率，第2、4周时实验组高于对照组，差异非常显著，P＜0.01；第6周时，差异仍显著，P＜0.05；（5）组织形态学指标：有髓种经轴突计数、再生轴突直径的恢复率，毛细血管面积，实验组均显著大于对照组，P＜0.01、P＜0.05、P＜0.001；实验组髓鞘厚度的恢复率也显著高于对照组，第2周时P＜0.01，第4、6周时，P＜0.05；（6）透射电镜超微结构观察：实验组再生轴突中细胞器丰富，髓鞘结构成熟，神经再生情况优于对照纽。

Result The inhibitory effect on pulmonary metastasis of transplanted tumor were 46.36%, 32.73%, 18.2% in the Runing granule preparation group, warming kidney group, dispersing liver and promoting blood circulation group respectively. There wasn＇t significant difference between Runing granule preparation group and CTX group （50.91%）. The expression of VEGF was dropped in Runing grannie preparation group and dispersing liver and promoting blood circulation group. The expression of TIMP-1mRNA was up-regulated in Runing Granule group and warming kidney group. The expression of MMP-9 was dropped in Runing granule preparation group and warming kidney group.

乳宁冲剂组及拆方温肾组、疏肝活血组对裸鼠乳腺癌移植瘤的肺转移抑制作用分别为46.36％、32.73％、18.2％，乳宁冲剂组作用与CTX组（50.91％）比较无显著差异；而乳宁冲剂组、疏肝活血组均可下调肿瘤VEGF蛋白表达；乳宁冲剂全方、温肾方可明显使TIMP-1mRNA的表达升高，MMP-9表达下降；对MMP-9／TIMP-1蛋白表达比值影响以温肾组下降最明显。

24H treatment group compared with 24h model group: the density of light of HSP70 of the group increased and which of TGF-β〓, is not different with the model group; the density of the light the HSP70 of the Baicalin group increased and which of it the TGF-β〓, increased with the model group; the density of light of the Concha Margatitifera Usta is not different and which of TGF-β〓, increased with the model group; the density of light of the Cholic Acid group is not different and the density of light of it of TGF-β〓 increased with the model group; the density of light of the Hefang Group is not different and which of TGF-β〓 is not different than the model group.

合方治疗脑缺血12小时组缺血脑组织TGF-β1光密度值较缺血12小时模型组无显著性差异（p＞0.05）。本实验结果发现，与单纯缺血12小时、24小时大鼠相比，药物干预组缺血侧大脑皮层TGF-β1免疫反应明显增强，计算机辅助图像半定量分析两组间缺血侧大脑皮层TGF-β1免疫阳性细胞数差异非常显著。说明药物对局灶性脑缺血损伤可能有一定保护作用，其机制可能与诱导脑细胞TGF-β1合成增加有关。然而，药物调控TGF-β1表达，其促TGF-β1表达增强的机制可能是这样的：脑缺血时TGF-β1mRNA表达增加可能与缺氧诱导神经和胶质细胞刺激生长因子表达有关。

METHODS: 40 rats were randomly divided into four groups:normal group, asthenic spleen group, high-dosage group and low-dosage group with 10 rats in each group, the asthenic spleen rat models were established in group B, C group as well as group D. Rats in group B, group C, group D were fed with normal distilled water, high-dosage of Guizhi decoction and low-dosage of Guizhi decoction respectively, NFAT mRNA, IL-4 mRNA, IFN-γmRNA were determined by PCR.

40只大鼠随机分为空白对照组、脾虚组、桂枝汤大剂量组和桂枝汤小剂量组等4组，每组10只；用规范的脾气虚证大鼠模型造模方法将B组、C组、D组大鼠造模，B组、C组、D组分别用蒸馏水、桂枝汤大剂量、桂枝汤小剂量灌胃，标本采集后用RT-PCR检测NFAT mRNA，IL-4 mRNA和IFN-γ mRNA表达水平。

Adult rats are divided into control group, intrapericardial injection group and negative group, sublingual vein injection group and negative group. 6ds after transfection, issues of heart, liver, lung and kidney are stained with X-gal to observe transfection to myocardium and non-target transfection. 3. CaN Aβ gene silencing in vivo. Adult rats are divided into Control group, hypertrophy model group, intrapericardial injection group and negative group, sublingual vein injection group and negative group.

二整体动物水平上探讨心肌有效转染途径：以PLacZ为报告基因，将成年大鼠分为空白对照纽(生理盐水心包腔内注射后超声导入)；转染质粒心包腔内注射组及其阴性对照组：质粒+微泡+酶的混合物心包腔内注射，超声导入；转染质粒舌下静脉注射组及其阴性对照组：质粒+微泡混合液经舌下静脉注射，超声导入。

Methods: Wistar rats were divided into normal group, model group, TCM group Ⅰ, TCM group Ⅱ and western medicine group. In addition to the normal group, the other four groups were reproduced animal model of pulmonic fibrosis. The model group were given normal saline gavage, The TCM group Ⅰ given Feifltkang Mistura (1g/ml) gavage, The TCM group Ⅱ were given Fe4fukang Mistura (0.5g/ml) gavage, WM group were given prednisone gavage. Half of each group were taken blood to test in 28th, 56th day.

选择Wistar大白鼠随机分为正常组、模型组、中药Ⅰ组、中药Ⅱ组、西药组，除正常组外，其余四组均复制肺纤维化动物模型；模型组用生理盐水灌胃，中药Ⅰ、Ⅱ组分别用高、低剂量肺复康合剂灌胃，西药组用强的松灌胃；各组分别检测血清相关指标。

Based on a formal analysis of lifting decomposition, the hierarchical structure concept of the decomposition set, as well as the lifting decomposition technique of the polyphase matrix' greatest mth-order common divisor, is proposed to conquer the difficulties in the practical computation of and the optimal search in the huge decomposition set.

本文在形式化分析Lifting分解原理的基础上，提出了分解集合的分级结构概念以及多相矩阵的m阶最大公因子Lifling分解技术，克服了分解集合求解问题的高复杂性所带来的实际计算与搜索困难；进一步地，从数值稳定性及计算代价两方面研究了分解的评价问题，设计出两种实用的分解稳定性准则；最后，结合分级结构技术提出一种最优分解算法。

The results show that the decalescence of catalyst bed affects the process of hydrogen peroxide catalytic decomposition greatly. If the catalyst bed is preheated or made up of materials with low thermal conductivity, that influence can be depressed. At the same time, the response characteristic of catalyst bed and the efficiency of catalytic decomposition can be improved. The low frequency pressure instability of catalyst bed occurs because of the coupling of catalytic decomposition and supplying. The decomposition plane pushes all the way through the catalyst bed during pressure oscillations, causing unreacted liquid hydrogen peroxide to be exhausted from the bed and leading to the appearance of liquid hydrogen peroxide channeling. Consequently, how to prevent or at least limit this channeling is the key to eliminate the pressure instability of catalyst bed. It was found that adding clapboard to the catalyst bed can depress the pressure instability efficiently.

结果表明催化剂床及催化剂的吸热对过氧化氢分解过程影响很大，对催化剂床进行预热或采用热导率低的材料制造催化剂床等措施，可以降低催化剂床壁面吸热对催化分解过程的影响，提高催化剂床的响应特性和催化分解效率；催化剂床的低频不稳定是催化分解过程与供应系统耦合产生的，催化剂床气体界面的波动过程中，液体过氧化氢穿透催化剂床形成过氧化氢液体通道，过氧化氢液体通道的产生和扩大是催化剂床不稳定的主要原因，也是消除催化剂床不稳定的关键，试验中在催化剂床内加入分流板成功地抑制了催化剂床的低频不稳定。

By applying these two tools,we mainly discuss the problems as follows:1 Perturbation identities for the generalized polar decomposition:By applying singular value decomposition, we study the perturbation bounds of the generalized polar decomposition under unitary invariant norms when A is perturbed by addition or multiplication,respectively and give our bounds in identities.2 A class of new unitary invariant metrics: This paper generalized a class of new unitarily invariant metrics and give the upper-bounds and lower-bounds by C-S decomposition.

利用这两个强有力的工具，本文主要讨论了如下问题：1 矩阵广义极分解的扰动等式：利用奇异值分解，本文分别讨论了加法扰动和乘法扰动下矩阵广义极分解在任意酉不变范数下的扰动界，并首次以恒等式的形式给出了酉极因子和半正定因子的扰动界。2 一类新的酉不变度量：本文推广了文[22] 中的结论，定义了一类新的酉不变度量。

Yet, Edith says, there was always money in the bank.

但是Edith说，她的银行户口里面总是有钱。

We have created a series of files that resolve this problem--one for each day of the 40 days of purpose.

我们制作一系列的档案去解决这问题---一个为标竿40天中的每一天。