豆状核

基本解释 (translations)

lenticula  ·  putamina

词组短语

lenticular nucleus

与豆状核相关的网络例句 [注：此内容来源于网络，仅供参考]

Results: In the 13 cases, the CT diagnosis show that five cases of low-density lesions of Bilateral hepatolenticular, five cases of globus pallidus, one case of Bilateral hepatolenticular with corpus callosum, one cases of External capsule putamen, one case of Putamen with the caudate nucleus, three cases of low-density of Bilateral lobi frontalis and lobi parietalis, one case of unilateral putamen hemorrhage, three cases of cerebral atrophy.

结果 13例中，CT示双侧豆状核低密度5例、苍白球5例、双侧豆状核伴胼胝体1例、壳核伴外囊1例、壳核伴尾状核1例；双额顶叶低密度3例、单侧壳核出血1例、大脑萎缩3例。

At 6h after ischemia both MRI were found to indicate definite infarct foci in the caudate nucleus and the lentiform nucleus,mass effect was seen too.Histopathologic findings:At 2h after ischemia,electro microscope revealed the changes of ischemia and edema.At 4h after ischemia,the photomicroscope demonstrated the changes of ischemia edema and the electromicroscope indicated the damage to blood brain barrier.

结果 梗塞2h MRI T2加权可见尾状核头部、豆状核信号增高；6h MRI可见尾状核、豆状核形成明确梗塞，并出现占位征象。2h电镜下已有缺血水肿改变，4h光镜下出现脑缺血水肿改变，电镜下见到血脑屏障受损。

Objective To discuss the VEEG changes in hepatolenticular degeneration patients.Methods VEEG was performed in 28 patients with hepatolenticular degeneration and 20 control subjects.

目的 探讨肝豆状核变性患者视频脑电图改变方法对28例肝豆状核变性患者及20例健康对照组视频脑电图监护检查。

In the original studies,[4,5] all the acute stroke patients with angiographically proven internal carotid artery or M1 occlusion had ALN, presumably because the lentiform nucleus is fed by lenticulostriate arteries from M1 without collateral flow from cortical anastomoses.

先前的研究报道：所有经脑血管造影证实存在颈内动脉闭塞或大脑中动脉M1段闭塞的急性卒中患者，都会出现豆状核低信号征；可能系豆状核是由大脑中动脉M1段发出的豆纹动脉供血，缺乏来源于皮层吻合支的侧枝循环供血所致。

On the other hand, if the medial lenticulostriate arteries escape from ischemia, the medial part of the lentiform nucleus may be spared ischemic injury, and early CT signs may be restricted to the lateral or posterior part of the lentiform nucleus.

另一方面，如果内侧豆纹动脉群（medial lenticulostriate arteries）没有受累发生缺血，那么豆状核内侧区可能就不会发生缺血损伤，早期CT征象可能就仅限于豆状核的后部或外侧部

The 8 cases of 12 patients examined by cranial CT showed abnormal low density lesion in basal ganglia,lentiform nucleus,and so on.The 5 cases of 8 patients examined by cranial MRI showed abnormal signs in basal ganglia,lentiform nucleus,and so on.

12例患者行头颅CT检查，8例发现基底节，豆状核等异常低密度灶；8例行头颅MRI检查，5例显示基底节，豆状核等处异常信号。

The results of in situ hybridization and fluorescence quantitative PCR showed that the gene expression of dopamine receptors(including D1 and D2) in the prefrontal cortex and DRD1 in lentiform nucleus of SHR from the Kongsheng Zhenzhong Pill group is higher than that in the methylphenidate hydrochloride group and both groups are lower than that in the control group:the gene expression of dopamine receptors D2 in the lentiform nucleus from the methylphenidate hydrochloride group is higher than that in the Kongsheng Zhenzhong Pill group while both groups are lower than that from the control group.

原位杂交和荧光定量PCR表明，SHR大鼠前额叶皮质的多巴胺受体(包括D1和D2)和豆状核受体D1的基因表达，孔圣枕中丹组比盐酸哌甲酯组要高，且两者均低于对照组；豆状核多巴胺受体D2的基因表达，盐酸哌甲酯组要比孔圣枕中丹组高，且均低于对照组。

Results:Sensitivity of dense artery sign,lentiform nucleus sign,insular ribbon sign,space occupying effect,hypodensity lesion and cortical sign were 20.8%,23.6%,8.8%,11.6%,56.8%and 14.8%respectivelyP＜0.05 as compared with normal control group.

结果：正常组：致密动脉征占11％，豆状核征占5％，脑岛带征占1％，其它征象均为阴性；病变组：致密动脉征占20.8％，豆状核征占23.6％，脑岛带征占8.8％，低密度灶占56.8％，占位征占11.6％，皮质征占14.8％(与正常组相比P均＜0.05)。

Methods:102 whole brains were serially transected with each section 2mm in thickness.82 red nuclei,substantia nigra and subthalamic nuclei,110 amydaloid nuclei,122 lenticular nuclei and thalami,and 114 caputs nuclei naudati were located in three dimension.

对102个整脑作三维切面的2mm厚连续切片，分别对82个红核、黑质、丘脑底核，110个杏仁核、122个豆状核、丘脑和114个尾状核头等脑内主要神经核团进行定位研究。

Results: In the 13 cases, the CT diagnosis show that five cases of low-density lesions of Bilateral hepatolenticular, five cases of globus pallidus, one case of Bilateral hepatolenticular with corpus callosum, one cases of External capsule putamen, one case of Putamen with the caudate nucleus, three cases of low-density of Bilateral lobi frontalis and lobi parietalis, one case of unilateral putamen hemorrhage, three cases of cerebral atrophy.

目的 探讨双侧基底节区对称性低密度病变的CT诊断价值。方法回顾性分析肝豆状核变性5例、中毒性病变5例、Wernicke脑病2例、Huntington氏大舞蹈病1例的CT平扫表现及临床意义。结果 13例中，CT示双侧豆状核低密度5例、苍白球5例、双侧豆状核伴胼胝体1例、壳核伴外囊1例、壳核伴尾状核1例；双额顶叶低密度3例、单侧壳核出血1例、大脑萎缩3例。

与豆状核相关的网络解释 [注：此内容来源于网络，仅供参考]

putamen：豆状核

抗体接合(antibodiesantibodies)利用有相对之激子与受体接合)利用有相对之激子与受体接合由静脉注射18F-FDOPA后,其会分布於相关基底核(basal ganglia)部位,尤其是尾状核(caudate nucleus),豆状核(putamen)等(上图)自体产物

WD：肝豆状核变性

肝豆状核变性基因第3~20号外显子突变及多态的DNA测序研究 中华神经科杂志1998年第6期第31卷研究快讯 作者:吴志英 王柠 慕容慎行 林珉婷 单位:350005福州,福建医科大学附属第一医院神经内科 肝豆状核变性(WD)是与铜代谢障碍有关

hepatolenticular degeneration：肝豆状核变性

(四)铜代谢障碍疾病-肝豆状核变性 肝豆状核变性(hepatolenticular degeneration)又称威尔逊病(wilson's disease). 本病为位于第13染色体的隐性基因传递的遗传性疾病,家族性多发. 患者多为儿童及青少年. 本病的特点是铜代谢障碍,不能正常排出而蓄积于各器官.

hepatolenticular degeneration, HLD：肝豆状核变性

1921年Hall定名肝豆状核变性(Hepatolenticular Degeneration HLD)或威尔逊氏(Wilson)假性硬化症 后人又称为威尔逊氏病(Wilson's disease WD). 本病已明确属常染色体隐性遗传性铜代谢障碍 造成铜在体内各脏器尤以大脑豆状核、肝脏、肾脏及角膜大量沉着 而由于铜离子在各脏器沉积的先后不同和数量不一 临床出现多种多样的

lenticula：豆状核

Lennox-Gastaut syndrome 小儿癫痫性脑病 | lenticula 豆状核 | lenticularis ansa 豆状袢

lentiform nucleus：豆状核

可见在侧脑室前角切面的后外侧,有一大致卵圆形的灰质团块切面,为尾状核(caudate nucleus)头的切面.在尾状核头切面的后外侧有一三角形的灰质切面为豆状核(lentiform nucleus)切面,此核中部由二纵走的白质分隔为三部,

lentiform nucleus：豆状核,透镜状核

豆状乳突 lenticular papilla | 豆状核,透镜状核 lentiform nucleus | 球形晶体 lentiglobus

lentiform nucleus , lenticular nucleus：豆状核

豆掌韧带|pisometacarpal ligament | 豆状核|lentiform nucleus , lenticular nucleus | 豆状核后部|retrolentiform part

thalamolenticular：丘脑豆状核的

thalamolaminotomy 丘脑髓板切开术 | thalamolenticular 丘脑豆状核的 | thalamolenticularpart 丘脑豆状核部

thalamolenticular part：丘脑豆状核部

thalamolenticular 丘脑豆状核的 | thalamolenticular part 丘脑豆状核部 | thalamomamillary bundle 乳头丘脑束