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glucagon相关的网络例句

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与 glucagon 相关的网络例句 [注:此内容来源于网络,仅供参考]

PACAP is a pleiotropic neuropeptide that belongs to the secretin/glucagon/vasoactive intestinal peptide family.

垂体腺苷酸环化酶激活肽是多效性的神经肽,属于肠促胰液素/胰高血糖素/肠血管活性肽家族。

This self-generated effect the liver, liver glucagon is the target organ.

此效产生自肝脏,肝脏是胰高血糖素的靶器官。

Through binding GLP-1 receptor , it can arouse insulin secretion, increase sensitivity of insulin, restrain glucagon secretion and slower the velocity of stomachic emptying. More recent studies showed that GLP-1 is effective for both type 1 and type 2 diabetes mellitus. Treatment of type 2 diabetes patients with GLP-1 does not lead to hypoglycemia.

此外,GLP-1通过激发其受体产生生理功能,能明显刺激胰岛素分泌、增加胰岛素敏感性、抑制胰升血糖素水平和减慢胃的排空,对2型糖尿病和1型糖尿病均有效,而且将其用于治疗不会产生低血糖。

Objective To study the effect of amylin on the secretion of glucagon and insulin in isolated rat islets.

目的 研究胰淀素对大鼠胰岛素和胰高血糖素分泌的影响。

Therapies based on glucagon-like peptide 1 and amylin have now been introduced into clinical practice.

基于高血糖样肽1和淀粉不溶素的治疗方法现已进入临床实践。

Recently, the gastrointestinal peptide hormones glucagon-like peptide 1 and amylin have been shown to significantly contribute to this complex physiology.

近来发现,胃肠肽激素,高血糖样肽1和淀粉不溶素也在这一复杂的生理过程中具有重要作用。

Methods The models of isolated rat islets were established by collagenase digestion and dextran gradient centrifugation. The influence of various concentrations of amylin on both glucagon and insulin secretion was studied.

应用胶原酶消化和不连续密度梯度离心技术建立离体大鼠胰岛模型,研究不同浓度胰淀素对胰岛素和胰高血糖素分泌的影响。

The experiment two: enzyme preparation significantly improved average daily gainand feed conversion ratio (P<0.05). Enzyme preparation significantly increased energymetabolizability and digestibility of crude fiber, crude protein and neutral detergent fiber,but had no remarkable effect on digestibility of dry matter, crude fat and acid detergentfiber. Enzyme preparation significantly decreased the relative viscosity of duodenal andjejunal digesta. The pH of intestine had no noticed difference in all groups. Enzymepreparation significantly decreased relative weight of gizzard, proventficulus, duodenum,jejunum and ileum. Enzyme preparation significantly increased villus size of duodenumand jejunum, and villus to crypt ratio of duodenum and ileum significantly increased too.Enzyme preparation considerably decreased ileal crypt height (P<0.05), and didn"t affectthickness of intestinal wall. Supplementing enzyme preparation, the serum glucose, totalprotein and alanine aminotransferase, but enzyme preparation hadn"t noticed influenceupon uric acid, total cholesterol, triglyceride and high-density lipoproteins. Enzymepreparation significantly increased insulin, triiodothyronine and insulin-like growthfactor-Ⅰ. Adding enzyme preparation, the percentage of thyroid stimulating hormone andgrowth hormone in the serum increased 16.44%, 19.18% and 18.84%, 21.74%respectively, and the percentage of glucagon and thyroxine decreased 12.07%, 14.36% and 13.79%, 15.40%, but failed to reach statistical significance (P>0.05). Enzymepreparation significantly increased (P<0.05) the trypsin and amylase activity of duodenaland jejunal digesta, but enzyme preparation didnt affect significantly (P>0.05) theintestinal lipase activity and pancreatic digestive enzyme. Enzyme preparation had nosignificant effect on caecal microbial population.

试验二:酶制剂显著提高平均日增重和饲料转化率(P<0.05);酶制剂显著提高能量代谢率及粗纤维、粗蛋白、中性洗涤纤维消化率(P<0.05),而对干物质、粗脂肪、酸性洗涤纤维消化率影响不显著;酶制剂显著降低十二指肠和空肠食糜相对粘度(P<0.05);添加酶制剂对肠道pH影响不显著;酶制剂显著降低肌胃、腺胃、十二指肠、空肠、回肠相对重(P<0.05),显著提高十二指肠和空肠绒毛高度,显著增加十二指肠和回肠绒毛高度/隐窝深度,降低回肠隐窝深度(P<0.05),对肠壁厚度影响不显著;酶制剂显著提高血清葡萄糖、总蛋白和谷丙转氨酶浓度(P<0.05),对尿酸、总胆固醇、甘油三酯及高密度脂蛋白浓度影响不显著,显著提高胰岛素、T_3、IGF-Ⅰ水平,添加酶制剂后,促甲状腺激素、生长激素分别提高16.44%、19.18%和18.84%、21.74%,胰高血糖素和T_4分别降低12.07%、14.36%和13.79%、15.40%,但差异不显著;酶制剂对胰腺消化酶活性影响不显著,显著增加十二指肠和空肠胰蛋白酶、淀粉酶活性,对小肠脂肪酶活性影响不显著;酶制剂对盲肠微生物菌落数影响不显著。

C-peptide and insulin/glucagon ratios of patients decreased significantly,which suggested that both a and βcell functions ofpancreas have damage, and that damagement of βcell were more severe.

细胞及β细胞功能受损害,且以β细胞功能受损明显;胰岛素/胰高血糖素比值下降提示慢性呼吸衰竭患者以分解代谢占优势。

To investigate the change of the portal venous pressure, conjugated glycocholic acid and pancreatic glucagon concentration in rats peripheral and portal venous blood in the course of experimental liver cirrhosis induced with carbon tetrachloride and to investigate the mentioned changes after portal-azygous devascularization.

袁晖应用四氯化碳诱发建立大鼠肝硬变模型,以观察大鼠肝硬变形成过程中和行门奇静脉断流术后门静脉压力、门脉及外周静脉血中甘氨胆酸和胰高糖素的变化。

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